ATL-derived exosomes modulate mesenchymal stem cells: potential role in leukemia progression. - Wikidata - wikimedia - TriplyDB

ATL-derived exosomes modulate mesenchymal stem cells: potential role in leukemia progression.

ATL-derived exosomes modulate mesenchymal stem cells: potential role in leukemia progression.

http://www.wikidata.org/entity/Q37350228

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Extracellular Vesicles in Hematological Malignancies: From Biology to Therapy.Adult T-cell leukemia: molecular basis for clonal expansion and transformation of HTLV-1-infected T cells.Extracellular vesicle-mediated transport of non-coding RNAs between stem cells and cancer cells: implications in tumor progression and therapeutic resistance.Multiple Myeloma-Derived Exosomes Regulate the Functions of Mesenchymal Stem Cells Partially via Modulating miR-21 and miR-146a.Exosomes in cancer: small vesicular transporters for cancer progression and metastasis, biomarkers in cancer therapeutics.Extracellular Vesicles: A New Prospective in Crosstalk between Microenvironment and Stem Cells in Hematological Malignancies.Reticuloendotheliosis virus and avian leukosis virus subgroup J synergistically increase the accumulation of exosomal miRNAs.

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ATL-derived exosomes modulate mesenchymal stem cells: potential role in leukemia progression.

http://www.wikidata.org/entity/Q37350228

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 19 October 2016

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

ATL-derived exosomes modulate ...... role in leukemia progression.

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ATL-derived exosomes modulate ...... role in leukemia progression.

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type

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ATL-derived exosomes modulate ...... role in leukemia progression.

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ATL-derived exosomes modulate ...... role in leukemia progression.

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ATL-derived exosomes modulate ...... role in leukemia progression.

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ATL-derived exosomes modulate ...... role in leukemia progression.

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ATL-derived exosomes modulate ...... role in leukemia progression.

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Farah Nassar

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Jamal El-Saghir

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Marwan El-Sabban

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Nadim Tawil

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P2860

Role of Tax protein in human T-cell leukemia virus type-I leukemogenicity

The new deal: a potential role for secreted vesicles in innate immunity and tumor progression

Vesicle formation during reticulocyte maturation. Association of plasma membrane activities with released vesicles (exosomes)

Cell surface tetraspanin Tspan8 contributes to molecular pathways of exosome-induced endothelial cell activation

Biogenesis, secretion, and intercellular interactions of exosomes and other extracellular vesicles

Exosomes released by K562 chronic myeloid leukemia cells promote angiogenesis in a Src-dependent fashion.

Stable expression of the tax gene of type I human T-cell leukemia virus in human T cells activates specific cellular genes involved in growth.

Thermal- and oxidative stress causes enhanced release of NKG2D ligand-bearing immunosuppressive exosomes in leukemia/lymphoma T and B cells

Exosome: from internal vesicle of the multivesicular body to intercellular signaling device.

Human T-lymphotropic virus type 1-infected cells secrete exosomes that contain Tax protein.

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