Osteoclast-derived matrix metalloproteinase-7, but not matrix metalloproteinase-9, contributes to tumor-induced osteolysis.
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Predictive computational modeling to define effective treatment strategies for bone metastatic prostate cancer.An integrated computational model of the bone microenvironment in bone-metastatic prostate cancer.Matrix metalloproteinase-induced epithelial-mesenchymal transition in breast cancer.Shedding of RANKL by tumor-associated MT1-MMP activates Src-dependent prostate cancer cell migration.An osteoblast-derived proteinase controls tumor cell survival via TGF-beta activation in the bone microenvironment.An MMP13-selective inhibitor delays primary tumor growth and the onset of tumor-associated osteolytic lesions in experimental models of breast cancer.Osteoclast-derived matrix metalloproteinase-9 directly affects angiogenesis in the prostate tumor-bone microenvironment.Matrix metalloproteinases and genetic mouse models in cancer research: a mini-review.Mesenchymal stem cells promote mammary cancer cell migration in vitro via the CXCR2 receptorCXCL13 activation of c-Myc induces RANK ligand expression in stromal/preosteoblast cells in the oral squamous cell carcinoma tumor-bone microenvironment.Contributions of the host microenvironment to cancer-induced bone disease.A loss of host-derived MMP-7 promotes myeloma growth and osteolytic bone disease in vivo.Collagenous and non-collagenous biochemical markers of bone metastases from prostate cancer.Potential molecular targets for inhibiting bone invasion by oral squamous cell carcinoma: a review of mechanisms.Signaling between tumor cells and the host bone marrow microenvironment.Roles of matrix metalloproteinases and their natural inhibitors in prostate cancer progression.Bone-Seeking Matrix Metalloproteinase-2 Inhibitors Prevent Bone Metastatic Breast Cancer Growth.Regulation of Bone Metabolism.Tumor-stromal interactions of the bone microenvironment: in vitro findings and potential in vivo relevance in metastatic lung cancer models.Soluble RANKL Cleaved from Activated Lymphocytes by TNF-α-Converting Enzyme Contributes to Osteoclastogenesis in Periodontitis.Extracellular matrix degradation and tissue remodeling in periprosthetic loosening and osteolysis: focus on matrix metalloproteinases, their endogenous tissue inhibitors, and the proteasome.Liensinine and Nuciferine, Bioactive Components of Nelumbo nucifera, Inhibit the Growth of Breast Cancer Cells and Breast Cancer-Associated Bone Loss.
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P2860
Osteoclast-derived matrix metalloproteinase-7, but not matrix metalloproteinase-9, contributes to tumor-induced osteolysis.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
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artigo científico
@pt
bilimsel makale
@tr
scientific article published on August 2009
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vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
Osteoclast-derived matrix meta ...... s to tumor-induced osteolysis.
@en
Osteoclast-derived matrix meta ...... s to tumor-induced osteolysis.
@nl
type
label
Osteoclast-derived matrix meta ...... s to tumor-induced osteolysis.
@en
Osteoclast-derived matrix meta ...... s to tumor-induced osteolysis.
@nl
prefLabel
Osteoclast-derived matrix meta ...... s to tumor-induced osteolysis.
@en
Osteoclast-derived matrix meta ...... s to tumor-induced osteolysis.
@nl
P2093
P2860
P1433
P1476
Osteoclast-derived matrix meta ...... s to tumor-induced osteolysis.
@en
P2093
Conor C Lynch
Ginger E Holt
Gregory R Mundy
Herbert S Schwartz
Jennifer Halpern
Lynn M Matrisian
Sophie Thiolloy
P2860
P304
P356
10.1158/0008-5472.CAN-08-3949
P407
P577
2009-08-01T00:00:00Z