Analysis of ventricular hypertrabeculation and noncompaction using genetically engineered mouse models.
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4-Dimensional light-sheet microscopy to elucidate shear stress modulation of cardiac trabeculation.Loss of Function Mutations in NNT Are Associated With Left Ventricular NoncompactionNumb family proteins are essential for cardiac morphogenesis and progenitor differentiationCritical roles of miRNA-mediated regulation of TGFβ signalling during mouse cardiogenesis.Dishevelled-associated activator of morphogenesis 1 (Daam1) is required for heart morphogenesis.Bioenergetics, mitochondria, and cardiac myocyte differentiationTbx20 transcription factor is a downstream mediator for bone morphogenetic protein-10 in regulating cardiac ventricular wall development and functionPLEKHM2 mutation leads to abnormal localization of lysosomes, impaired autophagy flux and associates with recessive dilated cardiomyopathy and left ventricular noncompactionTafazzin knockdown in mice leads to a developmental cardiomyopathy with early diastolic dysfunction preceding myocardial noncompaction.Fibulin-1 is required during cardiac ventricular morphogenesis for versican cleavage, suppression of ErbB2 and Erk1/2 activation, and to attenuate trabecular cardiomyocyte proliferationEbstein's anomaly may be caused by mutations in the sarcomere protein gene MYH7.Fkbp1a controls ventricular myocardium trabeculation and compaction by regulating endocardial Notch1 activity.Molecular mechanism of ventricular trabeculation/compaction and the pathogenesis of the left ventricular noncompaction cardiomyopathy (LVNC)Cardiomyocyte proliferation in zebrafish and mammals: lessons for human disease.Mismatch between the origin of premature ventricular complexes and the noncompacted myocardium in patients with noncompaction cardiomyopathy patients: involvement of the conduction system?Sequential Notch activation regulates ventricular chamber development.Essential but partially redundant roles for POU4F1/Brn-3a and POU4F2/Brn-3b transcription factors in the developing heart.Naturally Occurring Biventricular Noncompaction in an Adult Domestic Cat.Non-compaction of the left ventricle and associated ventricular septal defect.Opposite effects of Activin type 2 receptor ligands on cardiomyocyte proliferation during development and repair.Getting the heart into shape by the influence of cell death machinery.
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P2860
Analysis of ventricular hypertrabeculation and noncompaction using genetically engineered mouse models.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 25 April 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Analysis of ventricular hypert ...... cally engineered mouse models.
@en
Analysis of ventricular hypert ...... cally engineered mouse models.
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type
label
Analysis of ventricular hypert ...... cally engineered mouse models.
@en
Analysis of ventricular hypert ...... cally engineered mouse models.
@nl
prefLabel
Analysis of ventricular hypert ...... cally engineered mouse models.
@en
Analysis of ventricular hypert ...... cally engineered mouse models.
@nl
P2093
P2860
P1433
P1476
Analysis of ventricular hypert ...... cally engineered mouse models.
@en
P2093
Deqiang Li
Hanying Chen
R Mark Payne
Tim M Cordes
Weinian Shou
Wenjun Zhang
P2860
P2888
P304
P356
10.1007/S00246-009-9406-5
P577
2009-04-25T00:00:00Z