Much of late life cognitive decline is not due to common neurodegenerative pathologies. - Wikidata - wikimedia - TriplyDB

Much of late life cognitive decline is not due to common neurodegenerative pathologies.

Much of late life cognitive decline is not due to common neurodegenerative pathologies.

http://www.wikidata.org/entity/Q37354720

about

Cognitive Reserve as a Useful Concept for Early Intervention Research in Multiple Sclerosis How does it STAC up? Revisiting the scaffolding theory of aging and cognition Hippocampal sclerosis of aging, a prevalent and high-morbidity brain disease Genetic variants in Alzheimer disease - molecular and brain network approaches Identification of genes associated with dissociation of cognitive performance and neuropathological burden: Multistep analysis of genetic, epigenetic, and transcriptional data Cerebral amyloid angiopathy and cognitive outcomes in community-based older persons Building a pipeline to discover and validate novel therapeutic targets and lead compounds for Alzheimer's disease.Cognitive frailty.How Does Psychosocial Behavior Contribute to Cognitive Health in Old Age?Dietary fat composition and dementia risk The overlap between vascular disease and Alzheimer's disease--lessons from pathology.Epigenetic age of the pre-frontal cortex is associated with neuritic plaques, amyloid load, and Alzheimer's disease related cognitive functioning.Epigenomics of Alzheimer's disease.Three midlife strategies to prevent cognitive impairment due to Alzheimer's disease.Exercise improves cognitive function in aging patients.Prevalence of mixed pathologies in the aging brain B-lymphocyte-mediated delayed cognitive impairment following stroke.Clinical and imaging features of mixed Alzheimer and vascular pathologies.Conscientiousness, dementia related pathology, and trajectories of cognitive aging What we can learn from animal models about cerebral multi-morbidity.Residual decline in cognition after adjustment for common neuropathologic conditions.Odor identification and Alzheimer disease biomarkers in clinically normal elderly.Coupled changes in brain white matter microstructure and fluid intelligence in later life.Negative social interactions and risk of mild cognitive impairment in old age.Increased SNARE Protein-Protein Interactions in Orbitofrontal and Anterior Cingulate Cortices in Schizophrenia β-amyloid disrupts human NREM slow waves and related hippocampus-dependent memory consolidation.Association of DNA methylation in the brain with age in older persons is confounded by common neuropathologies.Post-mortem brain pathology is related to declining respiratory function in community-dwelling older adults.Mixed pathologies and neural reserve: Implications of complexity for Alzheimer disease drug discovery.Loss of Munc18-1 long splice variant in GABAergic terminals is associated with cognitive decline and increased risk of dementia in a community sample Neuropathologic comorbidity and cognitive impairment in the Nun and Honolulu-Asia Aging Studies The Impact of Age on Cognition Apolipoprotein E*4 (APOE*4) Genotype Is Associated with Altered Levels of Glutamate Signaling Proteins and Synaptic Coexpression Networks in the Prefrontal Cortex in Mild to Moderate Alzheimer Disease.Relation of neuropathology with cognitive decline among older persons without dementia.Temporal course of neurodegenerative effects on cognition in old age.Rescue of Early bace-1 and Global DNA Demethylation by S-Adenosylmethionine Reduces Amyloid Pathology and Improves Cognition in an Alzheimer's Model The Effect of Vascular Neuropathology on Late-life Cognition: Results from the SMART Project.TDP-43 pathology, cognitive decline, and dementia in old age Efficiency, capacity, compensation, maintenance, plasticity: emerging concepts in cognitive reserve.Predictors of ageing-related decline across multiple cognitive functions.

P2860

Q26782847-8D4A1B84-97A7-4432-A15D-252370D614A1 Q26827532-9471C907-D7DA-458C-B2D4-CE51C872DA0A Q26996741-BEA07FE2-B4BE-4709-9BF3-5A4BB2603A62 Q28596011-F02FC157-E5EB-4405-A1B2-C6743B248C51 Q30000273-B4357C83-2F6D-4EA9-AFB7-2984E52DFFC8 Q30683853-1F392EF7-BB0D-4F9E-A350-1666922ED618 Q33745617-3D20680C-9D83-4800-8618-30C06E56C385 Q33804945-A69D2DFE-382D-465C-BC93-4E04DC49AD57 Q33831749-CA8BE6D9-2B5A-49EC-B22D-218489C8AA8E Q33935512-928D5B02-A2CE-4979-9C17-92D16519C0AC Q34484708-E473E903-5821-4C2A-9E29-0A56C0AE2492 Q34505808-7B873ACA-C850-45A0-85EB-7DD6109ED48E Q34518584-AA1CE888-6E56-4AF2-8D89-2BA128C5BEF9 Q34536301-736AD4E6-C126-4B92-B439-B5C8DE705BEB Q34547975-0CF518C3-D26F-4C57-A30F-F3F1E3E58700 Q34551519-35F1040F-5B2C-402A-9E47-4A57150553F1 Q35044819-7C62BA82-BC9E-4209-A662-450CB4E2DA32 Q35125123-87F395AE-C863-45C2-AE66-B674F577328B Q35181136-B7F358FC-011D-40FA-8F9B-88C213E7FF81 Q35215656-8694038C-6E42-4217-A37E-6D0365C302E4 Q35572267-912E8DA1-7D14-4543-B4A9-FC3CF682CF2B Q35671009-2DB1CA97-4E28-49E5-8994-D922EDBAAE6A Q35675231-18D4313E-DF76-4621-83CD-3AE402483657 Q35744313-F92CFD15-97A5-44E3-8B28-09DCECAC0881 Q35761611-1E744F4E-998B-4760-B0AB-49070A1146A7 Q35788393-14C2D96D-9124-43D6-8C7B-AB96AA5E713A Q36042472-FF9C0AF3-89D0-4768-B046-BA1DC4D7F89E Q36182270-B1D87006-162E-4854-B269-988FCAB511D2 Q36307408-9D86B235-E1D1-49E7-9915-184A599FA53E Q36341076-D0084BB0-979A-4081-9769-DA5D7AB6A8FA Q36706031-4FEEAB84-0917-45B4-B037-EF991181D2AD Q37000400-63A8C7A6-11A9-498A-80BD-093B7D298140 Q37076865-9F3E1255-832B-438C-AA37-D2EF4483FF35 Q37155465-49D706F0-7A08-4569-AD1A-BF68CFD681B3 Q37183041-590DD6DC-09EA-4AD4-A088-513CAD5DEB1A Q37294108-D2160DB1-85FD-4094-98E7-384032F50047 Q37305212-4D60B814-DA25-401F-95B6-2167998E700A Q37315699-27BD2CDF-6300-4656-B0D2-68CCBBF1A1A4 Q37343322-0CF27F2C-760E-497D-BFD9-72EA6D43EACC Q37452795-959BBDEA-EDFE-489E-A158-E71353EC45B3

P2860

Much of late life cognitive decline is not due to common neurodegenerative pathologies.

http://www.wikidata.org/entity/Q37354720

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 10 July 2013

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Much of late life cognitive decline is not due to common neurodegenerative pathologies.

@en

Much of late life cognitive decline is not due to common neurodegenerative pathologies.

@nl

type

label

Much of late life cognitive decline is not due to common neurodegenerative pathologies.

@en

Much of late life cognitive decline is not due to common neurodegenerative pathologies.

@nl

prefLabel

Much of late life cognitive decline is not due to common neurodegenerative pathologies.

@en

Much of late life cognitive decline is not due to common neurodegenerative pathologies.

@nl

P1433

Q37354720-45A2C1DA-F703-47B4-BCE0-7C388A150137

P1476

Q37354720-090C265C-EE6F-46E8-B2D8-48C78950FCDE

P2093

Q37354720-14262161-E13B-48FF-B86F-2BB407A4552E

Q37354720-15DF2AA8-9F6F-4A95-B785-277CBCF97334

Q37354720-74D583DB-C834-44DC-97FD-A943DC1BEF91

Q37354720-90CFBBCE-641F-4114-8620-C3653F240FB9

Q37354720-C19E693F-246E-40F6-A4A4-D8911AFC868A

P2860

Q37354720-08F1B0BD-69CC-4AA2-BF18-1674042EAC46

Q37354720-1046B21E-32CC-4B56-B874-E666D8CD6FAD

Q37354720-16E85CAF-3EAC-4CE8-A504-4A2DD25B9946

Q37354720-17CCD6A4-D0A5-49A9-AE82-EE9498CE6593

Q37354720-1AB4E3DB-EACC-4241-BD0D-2EA7BFF4CBC7

Q37354720-249FACAA-8B7F-4EAA-AE19-6BC01E2BD92A

Q37354720-2F8636F8-3110-429B-81CE-009AE17E7B4C

Q37354720-33973AFE-939A-4582-AC53-AB9772CBD42C

Q37354720-39D3E786-D306-4B52-8CD3-C8C44A02EBD2

Q37354720-58102235-F6A3-4C1B-BA32-A07B9968862C

P304

Q37354720-F04F354B-2725-4D4D-BB73-1B526E31732B

P31

Q37354720-22A8AF7D-D68C-4B67-9AED-0C4E677F8210

P356

Q37354720-98C34237-6AD6-45D6-AFD9-895ECB36D3C1

P433

Q37354720-B2B174A9-7D9C-4EC8-AA82-248106D988F0

P478

Q37354720-CB13702E-F8F1-4A32-AAF4-4B040A55BD58

P50

Q37354720-8D140CE0-E2A9-4B88-BD7E-D147F9DE56B6

Q37354720-D9049DBA-89DF-42A2-A395-FB39E9B62C4B

P577

Q37354720-D446CA0B-0058-4F46-9FC2-8BFAAE2B279E

P5875

Q37354720-0CAACD1D-AD98-4FE8-AEB6-8373CE14FEDF

P698

Q37354720-80CBCEE8-7A33-404E-B843-E768ED68B45A

P921

Q37354720-0C851E96-8E35-458D-ACDD-C11FD4D6CB53

Q37354720-0d5af404-2de9-4e27-8f1b-1f30f81386f8

P932

Q37354720-8F2A1D4E-9868-4D08-AC3F-A26B02B1A031

P356

P1433

Annals of Neurology

P1476

Much of late life cognitive decline is not due to common neurodegenerative pathologies

@en

P2093

David A Bennett

http://www.w3.org/2001/XMLSchema#string

Julie A Schneider

http://www.w3.org/2001/XMLSchema#string

Lei Yu

http://www.w3.org/2001/XMLSchema#string

Patricia A Boyle

http://www.w3.org/2001/XMLSchema#string

Robert S Wilson

http://www.w3.org/2001/XMLSchema#string

P2860

Accumulation of transactive response DNA binding protein 43 in mild cognitive impairment and Alzheimer disease

Random-effects models for longitudinal data

What is cognitive reserve? Theory and research application of the reserve concept

Clinical, pathological, and neurochemical changes in dementia: A subgroup with preserved mental status and numerous neocortical plaques

Neurodegenerative basis of age-related cognitive decline.

Poor decision making is a consequence of cognitive decline among older persons without Alzheimer's disease or mild cognitive impairment.

Microinfarct pathology, dementia, and cognitive systems.

Terminal decline and practice effects in older adults without dementia: the MoVIES project.

Cognitive decline in the elderly: an analysis of population heterogeneity

Amyloid mediates the association of apolipoprotein E e4 allele to cognitive function in older people

P304

478-489

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1002/ANA.23964

http://www.w3.org/2001/XMLSchema#string

P433

3

http://www.w3.org/2001/XMLSchema#string

P478

74

http://www.w3.org/2001/XMLSchema#string

P50

Alasdair M Barr

William G. Honer

P577

2013-07-10T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

242018588

http://www.w3.org/2001/XMLSchema#string

P698

23798485

http://www.w3.org/2001/XMLSchema#string

P921

neurodegeneration

P932

3845973

http://www.w3.org/2001/XMLSchema#string