Inflammatory and hyperproliferative skin disease in mice that express elevated levels of the IL-1 receptor (type I) on epidermal keratinocytes. Evidence that IL-1-inducible secondary cytokines produced by keratinocytes in vivo can cause skin disease
about
Transgenic expression of the human amphiregulin gene induces a psoriasis-like phenotypeRegulation of epithelial-mesenchymal IL-1 signaling by PPARbeta/delta is essential for skin homeostasis and wound healingKeratinocyte expression of the type 2 interleukin 1 receptor mediates local and specific inhibition of interleukin 1-mediated inflammationInflammation and skeletal metastasisTranscriptomic analysis of the temporal host response to skin infestation with the ectoparasitic mite Psoroptes ovisDepletion of antigen-presenting cells by clodronate liposomes reverses the psoriatic skin phenotype in KC-Tie2 mice.Topical application of delphinidin reduces psoriasiform lesions in the flaky skin mouse model by inducing epidermal differentiation and inhibiting inflammationGenetically modified mice as a tool to study inflammatory skin diseases.IL-22 is required for Th17 cell-mediated pathology in a mouse model of psoriasis-like skin inflammation.Interleukin-1 regulates keratinocyte expression of T cell targeting chemokines through interleukin-1 receptor associated kinase-1 (IRAK1) dependent and independent pathways.Anti-Inflammatory Effects of Aspalathus linearis and Cyclopia spp. Extracts in a UVB/Keratinocyte (HaCaT) Model Utilising Interleukin-1α Accumulation as Biomarker.Modulation of the pro-inflammatory cytokines and matrix metalloproteinases production in co-cultivated human keratinocytes and melanocytes.IL-1α modulates neutrophil recruitment in chronic inflammation induced by hydrocarbon oilOverexpression of IL-1alpha in skin differentially modulates the immune response to scarification with vaccinia virus.Sodium pyruvate modulates cell death pathways in HaCaT keratinocytes exposed to half-mustard gas.Role of intracellular interleukin-1 receptor antagonist in skin carcinogenesis.The loss of ATP2C1 impairs the DNA damage response and induces altered skin homeostasis: Consequences for epidermal biology in Hailey-Hailey disease.Purification and characterization of an allergy-induced melanogenic stimulating factor in brownish guinea pig skin.Protective effect of liposome-encapsulated glutathione in a human epidermal model exposed to a mustard gas analog.Diet and nutrition in psoriasis: analysis of the National Health and Nutrition Examination Survey (NHANES) in the United States.IMQ-induced skin inflammation in mice is dependent on IL-1R1 and MyD88 signaling but independent of the NLRP3 inflammasome.[Beyond immunopathogenesis. Insulin resistance and "epidermal dysfunction"].
P2860
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P2860
Inflammatory and hyperproliferative skin disease in mice that express elevated levels of the IL-1 receptor (type I) on epidermal keratinocytes. Evidence that IL-1-inducible secondary cytokines produced by keratinocytes in vivo can cause skin disease
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on July 1996
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Inflammatory and hyperprolifer ...... in vivo can cause skin disease
@en
Inflammatory and hyperprolifer ...... ed levels of the IL-1 receptor
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type
label
Inflammatory and hyperprolifer ...... in vivo can cause skin disease
@en
Inflammatory and hyperprolifer ...... ed levels of the IL-1 receptor
@nl
prefLabel
Inflammatory and hyperprolifer ...... in vivo can cause skin disease
@en
Inflammatory and hyperprolifer ...... ed levels of the IL-1 receptor
@nl
P2093
P2860
P356
P1476
Inflammatory and hyperprolifer ...... in vivo can cause skin disease
@en
P2093
I R Williams
K Nakamura
R W Groves
T Rauschmayr
T S Kupper
P2860
P304
P356
10.1172/JCI118797
P407
P577
1996-07-01T00:00:00Z