CXCR3-deficiency protects influenza-infected CCR5-deficient mice from mortality.
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CCR5 ameliorates Japanese encephalitis via dictating the equilibrium of regulatory CD4+Foxp3+ T and IL-17+CD4+ Th17 cellsInnate immune response of human alveolar macrophages during influenza A infectionA novel outbred mouse model of 2009 pandemic influenza and bacterial co-infection severity.A spatial model of the efficiency of T cell search in the influenza-infected lung.Host genetic background strongly influences the response to influenza a virus infections.Every breath you take: the impact of environment on resident memory CD8 T cells in the lung.Persistence in Temporary Lung Niches: A Survival Strategy of Lung-Resident Memory CD8+ T Cells.The chemokine receptor CCR5, a therapeutic target for HIV/AIDS antagonists, is critical for recovery in a mouse model of Japanese encephalitisCXCR3 and CCR5 are both required for T cell-mediated protection against C. trachomatis infection in the murine genital mucosa.Interleukin-7, but not thymic stromal lymphopoietin, plays a key role in the T cell response to influenza A virus.CXCR3 in T cell function.Protective immunity against Chlamydia trachomatis can engage both CD4+ and CD8+ T cells and bridge the respiratory and genital mucosaeInflammatory chemokine receptors regulate CD8(+) T cell contraction and memory generation following infectionThe magnitude of the T cell response to a clinically significant dose of influenza virus is regulated by TRAIL.Comparative Analysis of Liver Injury-Associated Cytokines in Acute Hepatitis A and B.In vivo Ebola virus infection leads to a strong innate response in circulating immune cells.CXCR3 directs antigen-specific effector CD4+ T cell migration to the lung during parainfluenza virus infectionCXCL10-CXCR3 enhances the development of neutrophil-mediated fulminant lung injury of viral and nonviral origin.CXCR3 ligands: redundant, collaborative and antagonistic functions.The Flavonoid Isoliquiritigenin Reduces Lung Inflammation and Mouse Morbidity during Influenza Virus Infection.Diverse roles for T-bet in the effector responses required for resistance to infection.Chemokine receptor CXCR3 facilitates CD8(+) T cell differentiation into short-lived effector cells leading to memory degeneration.Lymphoid follicle cells in chronic obstructive pulmonary disease overexpress the chemokine receptor CXCR3.TGF-β Controls the Formation of Kidney-Resident T Cells via Promoting Effector T Cell Extravasation.CCR5 deficiency increased susceptibility to lipopolysaccharide-induced acute renal injury.Natural Killer Cell Recruitment to the Lung During Influenza A Virus Infection Is Dependent on CXCR3, CCR5, and Virus Exposure Dose.
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P2860
CXCR3-deficiency protects influenza-infected CCR5-deficient mice from mortality.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on December 2008
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
CXCR3-deficiency protects influenza-infected CCR5-deficient mice from mortality.
@en
CXCR3-deficiency protects influenza-infected CCR5-deficient mice from mortality.
@nl
type
label
CXCR3-deficiency protects influenza-infected CCR5-deficient mice from mortality.
@en
CXCR3-deficiency protects influenza-infected CCR5-deficient mice from mortality.
@nl
prefLabel
CXCR3-deficiency protects influenza-infected CCR5-deficient mice from mortality.
@en
CXCR3-deficiency protects influenza-infected CCR5-deficient mice from mortality.
@nl
P2093
P2860
P356
P1476
CXCR3-deficiency protects influenza-infected CCR5-deficient mice from mortality.
@en
P2093
Andrew D Luster
Benjamin D Medoff
Shannon K Bromley
P2860
P304
P356
10.1002/EJI.200838628
P407
P577
2008-12-01T00:00:00Z