Overexpression of glutamine:fructose-6-phosphate amidotransferase in transgenic mice leads to insulin resistance.
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Hexosamines, insulin resistance, and the complications of diabetes: current status.Glucose transport and sensing in the maintenance of glucose homeostasis and metabolic harmonyCaenorhabditis elegans ortholog of a diabetes susceptibility locus: oga-1 (O-GlcNAcase) knockout impacts O-GlcNAc cycling, metabolism, and dauerEffects of oral glucosamine sulphate on serum glucose and insulin during an oral glucose tolerance test of subjects with osteoarthritisGLUT2 is a high affinity glucosamine transporterThe hexosamine signaling pathway: deciphering the "O-GlcNAc code"Glutamine metabolism to glucosamine is necessary for glutamine inhibition of endothelial nitric oxide synthesisInsulin is a stronger inducer of insulin resistance than hyperglycemia in mice with type 1 diabetes mellitus (T1DM).Insulin control of glycogen metabolism in knockout mice lacking the muscle-specific protein phosphatase PP1G/RGL.Heme oxygenase-1 protects retinal endothelial cells against high glucose- and oxidative/nitrosative stress-induced toxicity.Simultaneous, quantitative analysis of UDP-N-acetylglucosamine, UDP-N-acetylgalactosamine, UDP-glucose and UDP-galactose in human peripheral blood cells, muscle biopsies and cultured mesangial cells by capillary zone electrophoresis.Protein-protein interaction in insulin signaling and the molecular mechanisms of insulin resistanceThe hexosamine signaling pathway: O-GlcNAc cycling in feast or famineAutocrine and paracrine mechanisms in the early stages of diabetic nephropathy.Optimization based tumor classification from microarray gene expression data.Regulation of glucose transporters--implications for insulin resistance states.Elevated nucleocytoplasmic glycosylation by O-GlcNAc results in insulin resistance associated with defects in Akt activation in 3T3-L1 adipocytesAltered glycan-dependent signaling induces insulin resistance and hyperleptinemiaProtein glycosylation: nature, distribution, enzymatic formation, and disease implications of glycopeptide bonds.Response gene to complement 32 (RGC-32) in endothelial cells is induced by glucose and helpful to maintain glucose homeostasis.Iron overload and diabetes risk: a shift from glucose to Fatty Acid oxidation and increased hepatic glucose production in a mouse model of hereditary hemochromatosis.O-GlcNAcylation: a novel post-translational mechanism to alter vascular cellular signaling in health and disease: focus on hypertension.Increased hexosamine pathway flux and high fat feeding are not additive in inducing insulin resistance: evidence for a shared pathwayO-GlcNAcylation: a novel pathway contributing to the effects of endothelin in the vasculature.Multiple tissue-specific roles for the O-GlcNAc post-translational modification in the induction of and complications arising from type II diabetes.Glucose metabolism and cardiac hypertrophyHexosamine biosynthesis is a possible mechanism underlying hypoxia's effects on lipid metabolism in human adipocytesPleiotropic and age-dependent effects of decreased protein modification by O-linked N-acetylglucosamine on pancreatic β-cell function and vascularization.Breast milk oligosaccharides: structure-function relationships in the neonate.Molecular docking studies of (4Z, 12Z)-cyclopentadeca-4, 12-dienone from Grewia hirsuta with some targets related to type 2 diabetesCross talk between O-GlcNAcylation and phosphorylation: roles in signaling, transcription, and chronic disease.Early mitochondrial adaptations in skeletal muscle to diet-induced obesity are strain dependent and determine oxidative stress and energy expenditure but not insulin sensitivityThe roles of O-linked β-N-acetylglucosamine in cardiovascular physiology and disease.Mechanisms linking diabetes mellitus to the development of atherosclerosis: a role for endoplasmic reticulum stress and glycogen synthase kinase-3.Role of protein O-linked N-acetyl-glucosamine in mediating cell function and survival in the cardiovascular system.Reduction of O-GlcNAc protein modification does not prevent insulin resistance in 3T3-L1 adipocytes.Glycan antagonists and inhibitors: a fount for drug discovery.Regulation of fatty acid metabolism by mTOR in adult murine hearts occurs independently of changes in PGC-1αIncreased vascular O-GlcNAcylation augments reactivity to constrictor stimuli - VASOACTIVE PEPTIDE SYMPOSIUM.Up-regulation of O-GlcNAc transferase with glucose deprivation in HepG2 cells is mediated by decreased hexosamine pathway flux.
P2860
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P2860
Overexpression of glutamine:fructose-6-phosphate amidotransferase in transgenic mice leads to insulin resistance.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on August 1996
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Overexpression of glutamine:fr ...... e leads to insulin resistance.
@en
Overexpression of glutamine:fr ...... e leads to insulin resistance.
@nl
type
label
Overexpression of glutamine:fr ...... e leads to insulin resistance.
@en
Overexpression of glutamine:fr ...... e leads to insulin resistance.
@nl
prefLabel
Overexpression of glutamine:fr ...... e leads to insulin resistance.
@en
Overexpression of glutamine:fr ...... e leads to insulin resistance.
@nl
P2093
P2860
P356
P1476
Overexpression of glutamine:fr ...... e leads to insulin resistance.
@en
P2093
Daniels MC
Hebert LF Jr
McClain DA
Neidigh JL
Simmons ST
P2860
P304
P356
10.1172/JCI118876
P407
P577
1996-08-01T00:00:00Z