Two core promotor mutations identified in a hepatitis B virus strain associated with fulminant hepatitis result in enhanced viral replication.
about
Low frequency of mutations in the core promoter and precore regions of hepatitis B virus in anti-HBe positive Brazilian carriersHepatitis B Virus e Antigen VariantsGenetic variation of hepatitis B virus and its significance for pathogenesisGenomic Diversity of Hepatitis B Virus Infection Associated With Fulminant Hepatitis B DevelopmentHepatitis C virus structural proteins assemble into viruslike particles in insect cells.Binding of hepatitis C virus-like particles derived from infectious clone H77C to defined human cell lines.Primary hepatocytes of Tupaia belangeri as a potential model for hepatitis C virus infectionCell Culture Models for the Investigation of Hepatitis B and D Virus InfectionMechanism of suppression of hepatitis B virus precore RNA transcription by a frequent double mutation.Naturally occurring mutations define a novel function of the hepatitis B virus core promoter in core protein expression.Clinical implications of hepatitis B virus mutations: recent advances.The core promoter of hepatitis B virus.Chimeric constructs between two hepatitis B virus genomes confirm transcriptional impact of core promoter mutations and reveal multiple effects of core gene mutations.Coexistence of hepatitis B virus quasispecies enhances viral replication and the ability to induce host antibody and cellular immune responses.Clinical significance of hepatitis B virus (HBV) genotypes and precore and core promoter mutations affecting HBV e antigen expression in TaiwanPoint mutations upstream of hepatitis B virus core gene affect DNA replication at the step of core protein expression.Strategies to eliminate HBV infection.Variability in the precore and core promoter regions of HBV strains in Morocco: characterization and impact on liver disease progressionHepatitis B virus genotype and basal core promoter/precore mutations are associated with hepatitis B-related acute-on-chronic liver failure without pre-existing liver cirrhosis.Identification of basic amino acids at the N-terminal end of the core protein that are crucial for hepatitis C virus infectivity.The hepatitis B virus x protein inhibits thymine DNA glycosylase initiated base excision repairControl or persistence of hepatitis B virus: the critical role of initial host-virus interactions.Pre-S deletion and complex mutations of hepatitis B virus related to young age hepatocellular carcinoma in Qidong, China.Hepatitis B virus genotypes and precore and core mutants in Brazilian patients.Genome replication, virion secretion, and e antigen expression of naturally occurring hepatitis B virus core promoter mutants.Hepatitis B virus core protein sensitizes hepatocytes to tumor necrosis factor-induced apoptosis by suppression of the phosphorylation of mitogen-activated protein kinase kinase 7Predominance of precore mutations and clinical significance of basal core promoter mutations in chronic hepatitis B virus infection in Indonesia.The infection efficiency and replication ability of circularized HBV DNA optimized the linear HBV DNA in vitro and in vivoClinical relevance of hepatitis B virus variantsAssociations between hepatitis B virus basal core promoter/pre-core region mutations and the risk of acute-on-chronic liver failure: a meta-analysis.Hepatitis B virus genetic variants: biological properties and clinical implicationsOverview of hepatitis B viral replication and genetic variabilityAssociations between hepatitis B virus genotype and mutants and the risk of hepatocellular carcinoma.Use of PCR in resolving diagnostic difficulties potentially caused by genetic variation of hepatitis B virus.Hepatitis B Virus Protein X Induces Degradation of Talin-1.Hepatitis B virus subgenotype A1 predominates in liver disease patients from Kerala, IndiaHepatitis B virus receptors and molecular drug targets.Identification of FDA-approved drugs that target hepatitis B virus transcription.Influence of the basal core promoter and precore mutation on replication of hepatitis B virus and antiviral susceptibility of different genotypes.Molecular epidemiology of an outbreak of fulminant hepatitis B.
P2860
Q24801344-5383FA72-7DC9-431D-9398-8FEACE0685A3Q24810141-39017E84-FE69-47EA-AEDB-2F2E39B180DEQ26772348-A397693A-CA1D-40F5-885A-4AEBCFCF1A3CQ26796488-C2AD9660-739F-4F5E-A5BE-5775FF25F1C9Q27469539-A554CAC0-E719-4510-84FE-1F527A4B8B77Q27472837-0F802B23-1F40-4E31-AAAE-6EAC921F2F46Q27473382-FFFB80BD-F5A0-4D11-8DB8-437A91FEF83DQ28068209-7B7F3C1A-92E4-4CC5-ABAA-BE4884CAEB92Q33640995-7DEC6498-E41A-44F3-BB2A-266EF5309421Q33783630-CDA4A617-BAA3-42F4-BBD6-1D0DEDDD2BD0Q33801311-7FC4BB0D-09FC-446F-A856-3BDD81F9DC16Q33801359-1757921F-0282-472D-B661-CCB1510D6B90Q33944550-912A75B8-48A9-4443-B57B-E362D9DCE2EEQ34057979-91A44A8C-AD0E-4ABD-8E99-8E97CCC05364Q34231638-B9935C36-AF1E-487C-807A-591ACA43E87FQ34301677-A39BAB4B-8244-4D15-8393-FBEDCEBB8FDDQ34308129-72E2D0DA-5C1A-4C1D-8932-956B56FD1736Q34384612-1D02D699-26E0-4660-9D46-E0D687009D80Q34389073-545C401C-4EFC-4BDE-B80A-BC4558E90794Q34416559-960FA3AC-2A2B-4159-B769-213B9A7EBD2AQ34473782-93693FED-E2B4-41B8-B26B-955F3E89E7F0Q34546460-0388002F-2040-4FA9-8125-613BDEE43369Q34651625-2B4D2E4F-7D98-486E-9C0A-76C34636CC3BQ34781061-9F88E5E2-6359-4569-B512-793BD75A9EE5Q35020504-D2E0112E-6763-4920-BD56-F2C5B7ED3017Q35115529-4A93B403-E405-45DA-9A58-51FAF1F6153EQ35125637-E07FFBB4-274F-42D8-9DB4-0A9BC7FCB8E8Q35380829-72ABF336-E4DE-4AC8-9249-0BD6934CFFDCQ35668730-BB05C870-DE4E-4265-B5DF-E971DD10AA42Q35799459-64C6B9B4-6E46-4D65-B503-153F1BA1DD57Q36795324-B49CB95A-E39E-4E99-9E75-5250079C12CDQ36807341-11D44186-48AB-438E-B857-B827EDC22FECQ36841560-61441C5E-6A5B-400E-BB18-6030FFFDAD7CQ37217802-45B6BA5B-F7FF-4E1B-9A00-30C3E41063E2Q37378812-8A9E5DDB-C87A-476B-9F6F-4FEEDD3A4703Q37439918-5154161F-9524-42B6-9D52-BC327DAE3BB2Q38775846-DB849D61-9D46-4162-8DEA-BE10BB5A3747Q38828885-7AACE9FE-3D55-45A1-AF7B-49B5E15D1629Q38917530-5898B726-2E45-4354-9CF9-72E212242CB9Q39460094-1D02D234-CED3-4D5A-B6C2-812893AB2FAF
P2860
Two core promotor mutations identified in a hepatitis B virus strain associated with fulminant hepatitis result in enhanced viral replication.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年学术文章
@wuu
1996年学术文章
@zh-cn
1996年学术文章
@zh-hans
1996年学术文章
@zh-my
1996年学术文章
@zh-sg
1996年學術文章
@yue
1996年學術文章
@zh
1996年學術文章
@zh-hant
name
Two core promotor mutations id ...... in enhanced viral replication.
@en
Two core promotor mutations id ...... in enhanced viral replication.
@nl
type
label
Two core promotor mutations id ...... in enhanced viral replication.
@en
Two core promotor mutations id ...... in enhanced viral replication.
@nl
prefLabel
Two core promotor mutations id ...... in enhanced viral replication.
@en
Two core promotor mutations id ...... in enhanced viral replication.
@nl
P2093
P2860
P356
P1476
Two core promotor mutations id ...... in enhanced viral replication.
@en
P2093
K Hasegawa
S A Rogers
T F Baumert
P2860
P304
P356
10.1172/JCI119037
P407
P577
1996-11-01T00:00:00Z