Chromatin structure and transposable elements in organismal aging. - Wikidata - wikimedia - TriplyDB

Chromatin structure and transposable elements in organismal aging.

Chromatin structure and transposable elements in organismal aging.

http://www.wikidata.org/entity/Q37361621

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Epigenetics and aging Restricting retrotransposons: a review Preferential retrotransposition in aging yeast mother cells is correlated with increased genome instability.Predicting the three-dimensional folding of cis-regulatory regions in mammalian genomes using bioinformatic data and polymer models Small RNA Pathways That Protect the Somatic Genome Extension of Saccharomyces paradoxus chronological lifespan by retrotransposons in certain media conditions is associated with changes in reactive oxygen species.Trimethylation of Lys36 on H3 restricts gene expression change during aging and impacts life span.From inflammaging to healthy aging by dietary lifestyle choices: is epigenetics the key to personalized nutrition?Oocyte ageing and epigenetics.H3K36 methylation promotes longevity by enhancing transcriptional fidelity.Retrotransposon activation contributes to neurodegeneration in a Drosophila TDP-43 model of ALS Unique transposon landscapes are pervasive across Drosophila melanogaster genomes.Somatizing the transposons action.Epigenetic regulation of ageing: linking environmental inputs to genomic stability.Chromatin-modifying genetic interventions suppress age-associated transposable element activation and extend life span in Drosophila.Tightening the connection between transposable element mobilization and aging.What might retrotransposons teach us about aging?Indy gene variation in natural populations confers fitness advantage and life span extension through transposon insertion The fine line between lifespan extension and shortening in response to caloric restriction.The redundancy of the mammalian heterochromatic compartment.ADAR RNA editing below the backbone.Age-associated de-repression of retrotransposons in the Drosophila fat body, its potential cause and consequence.Epigenetic Basis of Cellular Senescence and Its Implications in Aging.Preferential Ty1 retromobility in mother cells and nonquiescent stationary phase cells is associated with increased concentrations of total Gag or processed Gag and is inhibited by exposure to a high concentration of calcium.The Retrotransposon storm and the dangers of a Collyer's genome.Unique patterns of trimethylation of histone H3 lysine 4 are prone to changes during aging in Caenorhabditis elegans somatic cells.

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Chromatin structure and transposable elements in organismal aging.

http://www.wikidata.org/entity/Q37361621

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 04 December 2013

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Chromatin structure and transposable elements in organismal aging.

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Chromatin structure and transposable elements in organismal aging.

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type

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Chromatin structure and transposable elements in organismal aging.

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Chromatin structure and transposable elements in organismal aging.

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prefLabel

Chromatin structure and transposable elements in organismal aging.

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Chromatin structure and transposable elements in organismal aging.

@nl

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FGENE.2013.00274

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Frontiers in Genetics

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Chromatin structure and transposable elements in organismal aging.

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Jason G Wood

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Stephen L Helfand

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The mutant form of lamin A that causes Hutchinson-Gilford progeria is a biomarker of cellular aging in human skin

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Accumulation of mutant lamin A causes progressive changes in nuclear architecture in Hutchinson-Gilford progeria syndrome

SIRT1 redistribution on chromatin promotes genomic stability but alters gene expression during aging

The SIR2/3/4 complex and SIR2 alone promote longevity in Saccharomyces cerevisiae by two different mechanisms

DICER1 loss and Alu RNA induce age-related macular degeneration via the NLRP3 inflammasome and MyD88

RNAi-mediated targeting of heterochromatin by the RITS complex

Polycomb Repressive Complex 2 and Trithorax modulate Drosophila longevity and stress resistance

The RNA-induced silencing complex: a versatile gene-silencing machine

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274

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scholarly article

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10.3389/FGENE.2013.00274

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