Studies on the contribution of c-fos/AP-1 to arthritic joint destruction.
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Structure of the human histamine H1 receptor geneFibroblast-like synoviocytes: key effector cells in rheumatoid arthritisc-Fos/activator protein-1 transactivates wee1 kinase at G(1)/S to inhibit premature mitosis in antigen-specific Th1 cells.Small molecule inhibitors targeting activator protein 1 (AP-1).Signal transduction and transcription factors in rheumatic disease.Inhibition of transcription factors by anti-inflammatory and anti-rheumatic drugs: can variability in response be overcome?IL-1 inhibitors: novel agents in the treatment of rheumatoid arthritis.Punica granatum L. extract inhibits IL-1beta-induced expression of matrix metalloproteinases by inhibiting the activation of MAP kinases and NF-kappaB in human chondrocytes in vitro.Matrix metalloproteinases as therapeutic targets in arthritic diseases: bull's-eye or missing the mark?A systems approach to rheumatoid arthritis.Gene therapy in autoimmune diseases.Spontaneous and cytokine regulated c-fos gene expression in rheumatoid synovial cells: resistance to cytokine stimulation when the c-fos gene is overexpressed.Inhibitory effect of ribbon-type NF-kappaB decoy oligodeoxynucleotides on osteoclast induction and activity in vitro and in vivoc-Jun N-Terminal Kinase in Inflammation and Rheumatic Diseases.Soft coral-derived lemnalol alleviates monosodium urate-induced gouty arthritis in rats by inhibiting leukocyte infiltration and iNOS, COX-2 and c-Fos protein expressionA wogonin-rich-fraction of Scutellaria baicalensis root extract exerts chondroprotective effects by suppressing IL-1β-induced activation of AP-1 in human OA chondrocytes.Pathogenesis of joint destruction in rheumatoid arthritis.Synergistic induction of matrix metalloproteinase 1 by interleukin-1alpha and oncostatin M in human chondrocytes involves signal transducer and activator of transcription and activator protein 1 transcription factors via a novel mechanism.Plant extracts from stinging nettle (Urtica dioica), an antirheumatic remedy, inhibit the proinflammatory transcription factor NF-kappaB.A triple altered collagen II peptide with consecutive substitutions of TCR contacting residues inhibits collagen-induced arthritis.Human wee1 kinase is directly transactivated by and increased in association with c-Fos/AP-1: rheumatoid synovial cells overexpressing these genes go into aberrant mitosis.Suppressed severity of collagen-induced arthritis by in vivo transfection of nuclear factor kappaB decoy oligodeoxynucleotides as a gene therapy.N-(2-hydroxy phenyl) acetamide produces profound inhibition of c-Fos protein and mRNA expression in the brain of adjuvant-induced arthritic rats.Harpagoside suppresses IL-6 expression in primary human osteoarthritis chondrocytes.Interactions between the genes that regulate the body clock and those that worsen rheumatoid arthritis.Increased c-Fos/activator protein-1 confers resistance against anergy induction on antigen-specific T cell.
P2860
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P2860
Studies on the contribution of c-fos/AP-1 to arthritic joint destruction.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on March 1997
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
Studies on the contribution of c-fos/AP-1 to arthritic joint destruction.
@en
Studies on the contribution of c-fos/AP-1 to arthritic joint destruction.
@nl
type
label
Studies on the contribution of c-fos/AP-1 to arthritic joint destruction.
@en
Studies on the contribution of c-fos/AP-1 to arthritic joint destruction.
@nl
prefLabel
Studies on the contribution of c-fos/AP-1 to arthritic joint destruction.
@en
Studies on the contribution of c-fos/AP-1 to arthritic joint destruction.
@nl
P2093
P2860
P356
P1476
Studies on the contribution of c-fos/AP-1 to arthritic joint destruction.
@en
P2093
P2860
P304
P356
10.1172/JCI119277
P407
P577
1997-03-01T00:00:00Z