MicroRNA expression profiling of Leishmania donovani-infected host cells uncovers the regulatory role of MIR30A-3p in host autophagy.
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Leishmania donovani resides in modified early endosomes by upregulating Rab5a expression via the downregulation of miR-494.Non-coding RNAs in Host-Pathogen Interactions: Subversion of Mammalian Cell Functions by Protozoan Parasites.Countervailing, time-dependent effects on host autophagy promotes intracellular survival of leishmania.Leishmania donovani Activates Hypoxia Inducible Factor-1α and miR-210 for Survival in Macrophages by Downregulation of NF-κB Mediated Pro-inflammatory Immune Response.Integrated Analysis Reveals That miR-193b, miR-671, and TREM-1 Correlate With a Good Response to Treatment of Human Localized Cutaneous Leishmaniasis Caused by Leishmania braziliensis.
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MicroRNA expression profiling of Leishmania donovani-infected host cells uncovers the regulatory role of MIR30A-3p in host autophagy.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 26 July 2016
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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MicroRNA expression profiling ...... f MIR30A-3p in host autophagy.
@en
MicroRNA expression profiling ...... f MIR30A-3p in host autophagy.
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MicroRNA expression profiling ...... f MIR30A-3p in host autophagy.
@en
MicroRNA expression profiling ...... f MIR30A-3p in host autophagy.
@nl
prefLabel
MicroRNA expression profiling ...... f MIR30A-3p in host autophagy.
@en
MicroRNA expression profiling ...... f MIR30A-3p in host autophagy.
@nl
P2860
P1433
P1476
MicroRNA expression profiling ...... of MIR30A-3p in host autophagy
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P2093
Alok Kumar Singh
Chandrima Shaha
P2860
P304
P356
10.1080/15548627.2016.1203500
P577
2016-07-26T00:00:00Z