Acute lung injury induced by lipopolysaccharide is independent of complement activation. - Wikidata - wikimedia - TriplyDB

Acute lung injury induced by lipopolysaccharide is independent of complement activation.

Acute lung injury induced by lipopolysaccharide is independent of complement activation.

http://www.wikidata.org/entity/Q37365949

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Macrophage CD74 contributes to MIF-induced pulmonary inflammation Sepsis and ARDS: The Dark Side of Histones Bifunctional Lipocalin Ameliorates Murine Immune Complex-induced Acute Lung Injury A novel compound C12 inhibits inflammatory cytokine production and protects from inflammatory injury in vivo Inhibition of hypoxia inducible factor-1α ameliorates lung injury induced by trauma and hemorrhagic shock in rats.Complement inhibition alleviates paraquat-induced acute lung injury.Analysis of mechanical ventilation and lipopolysaccharide‑induced acute lung injury using DNA microarray analysis.Anti-inflammatory effects of β2 adrenergic receptor agonists in experimental acute lung injury.Role of C3, C5 and anaphylatoxin receptors in acute lung injury and in sepsis Role of Complement C5 in Experimental Blunt Chest Trauma-Induced Septic Acute Lung Injury (ALI).Protective Role of Proton-Sensing TDAG8 in Lipopolysaccharide-Induced Acute Lung Injury.Experimental design of complement component 5a-induced acute lung injury (C5a-ALI): a role of CC-chemokine receptor type 5 during immune activation by anaphylatoxin.DHA Suppresses Primary Macrophage Inflammatory Responses via Notch 1/ Jagged 1 Signaling.Bigelovii A Protects against Lipopolysaccharide-Induced Acute Lung Injury by Blocking NF-κB and CCAAT/Enhancer-Binding Protein δ Pathways Comparison of the therapeutic effects of human and mouse adipose-derived stem cells in a murine model of lipopolysaccharide-induced acute lung injury.Heat stress prevents lipopolysaccharide-induced apoptosis in pulmonary microvascular endothelial cells by blocking calpain/p38 MAPK signalling Attenuation of IgG immune complex-induced acute lung injury by silencing C5aR in lung epithelial cells.Inflammation in rat pregnancy inhibits spiral artery remodeling leading to fetal growth restriction and features of preeclampsia Opening the flood-gates: how neutrophil-endothelial interactions regulate permeability.Oxidative stress: acute and progressive lung injury.Growth factors and cytokines in acute lung injury.Protective role of cellular prion protein against TNFα-mediated inflammation through TACE α-secretase.Murine Models of Sepsis and Trauma: Can We Bridge the Gap?C5L2, the Second C5a Anaphylatoxin Receptor, Suppresses LPS-Induced Acute Lung Injury.Effects of CCK-8 and Cystathionine γ-Lyase/Hydrogen Sulfide System on Acute Lung Injury in Rats.Leukotriene B4 mediates gammadelta T lymphocyte migration in response to diverse stimuli.Cold stress aggravates inflammatory responses in an LPS-induced mouse model of acute lung injury.Ethyl pyruvate reduces mortality in an endotoxin-induced severe acute lung injury mouse model.ANTIOXIDANT AND ANTI-INFLAMMATORY EFFECTS OF RHAMNAZIN ON LIPOPOLYSACCHARIDE-INDUCED ACUTE LUNG INJURY AND INFLAMMATION IN RATS.Protective Effects of Dioscin against Lipopolysaccharide-Induced Acute Lung Injury through Inhibition of Oxidative Stress and Inflammation Targeting complement anaphylatoxin C5a receptor in hyperoxic lung injury in mice.The Interactive Roles of Lipopolysaccharides and dsRNA/Viruses on Respiratory Epithelial Cells and Dendritic Cells in Allergic Respiratory Disorders: The Hygiene Hypothesis.GHK Peptide Inhibits Bleomycin-Induced Pulmonary Fibrosis in Mice by Suppressing TGFβ1/Smad-Mediated Epithelial-to-Mesenchymal Transition.Interleukin-17A Plays the Same Role on Mice Acute Lung Injury Respectively Induced by Lipopolysaccharide and Paraquat.Protective mechanical ventilation does not exacerbate lung function impairment or lung inflammation following influenza A infection.The alpha-lipoic acid derivative DHLHZn: a new therapeutic agent for acute lung injury in vivo.

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P2860

Acute lung injury induced by lipopolysaccharide is independent of complement activation.

http://www.wikidata.org/entity/Q37365949

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

@tr

scientific article published on June 2008

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Acute lung injury induced by lipopolysaccharide is independent of complement activation.

@en

Acute lung injury induced by lipopolysaccharide is independent of complement activation.

@nl

type

label

Acute lung injury induced by lipopolysaccharide is independent of complement activation.

@en

Acute lung injury induced by lipopolysaccharide is independent of complement activation.

@nl

prefLabel

Acute lung injury induced by lipopolysaccharide is independent of complement activation.

@en

Acute lung injury induced by lipopolysaccharide is independent of complement activation.

@nl

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JIMMUNOL.180.11.7664

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Journal of Immunology

P1476

Acute lung injury induced by lipopolysaccharide is independent of complement activation.

@en

P2093

Brian A Nadeau

http://www.w3.org/2001/XMLSchema#string

Danielle E Day

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J Vidya Sarma

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Kyros Ipaktchi

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Laszlo M Hoesel

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Lin Leng

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Michael A Flierl

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Richard Bucala

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Stephanie R McGuire

http://www.w3.org/2001/XMLSchema#string

Thomas A Neff

http://www.w3.org/2001/XMLSchema#string

P2860

Innate immune recognition: mechanisms and pathways

Requirement and role of C5a in acute lung inflammatory injury in rats

Sequential recruitment of neutrophils into lung and bronchoalveolar lavage fluid in LPS-induced acute lung injury.

Critical role of endothelial CXCR2 in LPS-induced neutrophil migration into the lung.

Complement component C5 modulates the systemic tumor necrosis factor response in murine endotoxic shock

Macrophage migration inhibitory factor (MIF): its essential role in the immune system and cell growth.

Acute immunologic pulmonary alveolitis.

Complement levels and activity in the normal and LPS-injured lung.

Role of the LTB4/BLT1 pathway in allergen-induced airway hyperresponsiveness and inflammation.

Effects of leukotriene B4 in the human lung. Recruitment of neutrophils into the alveolar spaces without a change in protein permeability.

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7664-7672

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scholarly article

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10.4049/JIMMUNOL.180.11.7664

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11

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180

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Markus S Huber-Lang

Daniel Rittirsch

Firas S Zetoune

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2008-06-01T00:00:00Z

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5359180

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18490769

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lipopolysaccharide

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2753408

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