Protecting against cerebrovascular injury: contributions of 12/15-lipoxygenase to edema formation after transient focal ischemia. - Wikidata - wikimedia - TriplyDB

Protecting against cerebrovascular injury: contributions of 12/15-lipoxygenase to edema formation after transient focal ischemia.

Protecting against cerebrovascular injury: contributions of 12/15-lipoxygenase to edema formation after transient focal ischemia.

http://www.wikidata.org/entity/Q37367617

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Neuroprotection and antioxidants Methylophiopogonanone A Protects against Cerebral Ischemia/Reperfusion Injury and Attenuates Blood-Brain Barrier Disruption In Vitro Mammalian lipoxygenases and their biological relevance 12- and 15-lipoxygenases in adipose tissue inflammation Functional and pathological roles of the 12- and 15-lipoxygenases Therapies targeting lipid peroxidation in traumatic brain injury Pathophysiology of glia in perinatal white matter injury.Potent and selective inhibitors of human reticulocyte 12/15-lipoxygenase as anti-stroke therapies Increased nuclear apoptosis-inducing factor after transient focal ischemia: a 12/15-lipoxygenase-dependent organelle damage pathway.The 12/15-lipoxygenase as an emerging therapeutic target for Alzheimer's disease Iron mediates endothelial cell damage and blood-brain barrier opening in the hippocampus after transient forebrain ischemia in rats.Loss of miR-29b following acute ischemic stroke contributes to neural cell death and infarct size.The potential of 12/15-lipoxygenase inhibitors in stroke therapy.Cell type-specific dependency on the PI3K/Akt signaling pathway for the endogenous Epo and VEGF induction by baicalein in neurons versus astrocytes.The developing oligodendrocyte: key cellular target in brain injury in the premature infant Polyunsaturated fatty acid metabolism signature in ischemia differs from reperfusion in mouse intestine.12/15-Lipoxygenase metabolites of arachidonic acid activate PPARγ: a possible neuroprotective effect in ischemic brain Dietary flavonoids are neuroprotective through Nrf2-coordinated induction of endogenous cytoprotective proteins.DOR activation inhibits anoxic/ischemic Na+ influx through Na+ channels via PKC mechanisms in the cortex STAT-dependent upregulation of 12/15-lipoxygenase contributes to neuronal injury after stroke.Following experimental stroke, the recovering brain is vulnerable to lipoxygenase-dependent semaphorin signaling Inhibition of 12/15-lipoxygenase as therapeutic strategy to treat stroke Lipoxygenase: an emerging target for stroke therapy CD47 gene knockout protects against transient focal cerebral ischemia in mice Design of a novel thiophene inhibitor of 15-lipoxygenase-1 with both anti-inflammatory and neuroprotective properties Effect of human 15-lipoxygenase-1 metabolites on vascular function in mouse mesenteric arteries and hearts.12/15-lipoxygenase expression is increased in oligodendrocytes and microglia of periventricular leukomalacia Antioxidant enzyme gene transfer for ischemic diseases.12/15-Lipoxygenase targets neuronal mitochondria under oxidative stress.Mechanisms of ascorbyl radical formation in human platelet-rich plasma.Significance of brain tissue oxygenation and the arachidonic acid cascade in stroke.Novel insights into the pharmacology of flavonoids.Differential contribution of lipoxygenase isozymes to nigrostriatal vulnerability.Ischemia/Reperfusion.The role of Ca2+ in cell death caused by oxidative glutamate toxicity and ferroptosis.Cardiac 12/15 lipoxygenase-induced inflammation is involved in heart failure.Baicalin Attenuates Blood-Brain Barrier Disruption and Hemorrhagic Transformation and Improves Neurological Outcome in Ischemic Stroke Rats with Delayed t-PA Treatment: Involvement of ONOO--MMP-9 Pathway.Baicalein Promotes Neuronal and Behavioral Recovery After Intracerebral Hemorrhage Via Suppressing Apoptosis, Oxidative Stress and Neuroinflammation.Neuroprotection of taurine through inhibition of 12/15 lipoxygenase pathway in cerebral ischemia of rats.12/15-Lipoxygenase Inhibition or Knockout Reduces Warfarin-Associated Hemorrhagic Transformation After Experimental Stroke.

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Protecting against cerebrovascular injury: contributions of 12/15-lipoxygenase to edema formation after transient focal ischemia.

http://www.wikidata.org/entity/Q37367617

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 17 July 2008

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Protecting against cerebrovasc ...... fter transient focal ischemia.

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Protecting against cerebrovasc ...... fter transient focal ischemia.

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Protecting against cerebrovasc ...... fter transient focal ischemia.

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Protecting against cerebrovasc ...... fter transient focal ischemia.

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Protecting against cerebrovasc ...... fter transient focal ischemia.

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Protecting against cerebrovasc ...... fter transient focal ischemia.

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STROKEAHA.108.514927

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Protecting against cerebrovasc ...... fter transient focal ischemia.

@en

P2093

Eng H Lo

http://www.w3.org/2001/XMLSchema#string

Guang Jin

http://www.w3.org/2001/XMLSchema#string

Ken Arai

http://www.w3.org/2001/XMLSchema#string

Klaus van Leyen

http://www.w3.org/2001/XMLSchema#string

Monique F Stins

http://www.w3.org/2001/XMLSchema#string

Sophia Wang

http://www.w3.org/2001/XMLSchema#string

Yoshihiro Murata

http://www.w3.org/2001/XMLSchema#string

P2860

Novel docosanoids inhibit brain ischemia-reperfusion-mediated leukocyte infiltration and pro-inflammatory gene expression

Lipoxins and new lipid mediators in the resolution of inflammation

Resolvin E1 and protectin D1 activate inflammation-resolution programmes

Free radicals as triggers of brain edema formation after stroke.

Inhibition of 15-lipoxygenase leads to delayed organelle degradation in the reticulocyte.

Ischemic brain edema.

Neuroprotective effects of edaravone: a novel free radical scavenger in cerebrovascular injury.

Mammalian arachidonate 15-lipoxygenases structure, function, and biological implications.

Targets for vascular protection after acute ischemic stroke.

Exciting, radical, suicidal: how brain cells die after stroke.

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2538-2543

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10.1161/STROKEAHA.108.514927

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