Proinflammatory cytokine tumor necrosis factor (TNF)-like weak inducer of apoptosis (TWEAK) suppresses satellite cell self-renewal through inversely modulating Notch and NF-κB signaling pathways.
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The TWEAK-Fn14 pathway: a potent regulator of skeletal muscle biology in health and diseaseTWEAK Regulates Muscle Functions in a Mouse Model of RNA Toxicity.TWEAK/Fn14, a pathway and novel therapeutic target in myotonic dystrophy.The PERK arm of the unfolded protein response regulates satellite cell-mediated skeletal muscle regenerationHeightened TWEAK-NF-κB signaling and inflammation-associated fibrosis in paralyzed muscles of men with chronic spinal cord injury.CD163 interacts with TWEAK to regulate tissue regeneration after ischaemic injuryTWEAK/Fn14 Signaling Axis Mediates Skeletal Muscle Atrophy and Metabolic Dysfunction.Role of the TWEAK-Fn14-cIAP1-NF-κB Signaling Axis in the Regulation of Myogenesis and Muscle HomeostasisThe role of mechanobiology in progression of rotator cuff muscle atrophy and degeneration.Early Growth Response 3 (Egr3) Contributes a Maintenance of C2C12 Myoblast Proliferation.Age-associated NF-κB signaling in myofibers alters the satellite cell niche and re-strains muscle stem cell functionDetection of satellite cells during skeletal muscle wound healing in rats: time-dependent expressions of Pax7 and MyoD in relation to wound age.Isolation, Culture, and Staining of Single Myofibers.Contributions of muscle-resident progenitor cells to homeostasis and disease.TRAF6 regulates satellite stem cell self-renewal and function during regenerative myogenesis.Tumor necrosis factor-like weak inducer of apoptosis regulates quadriceps muscle atrophy and fiber-type alteration in a rat model of chronic obstructive pulmonary disease.Involvement of Transient Receptor Potential Cation Channel Vanilloid 1 (TRPV1) in Myoblast Fusion.α7nAChR is expressed in satellite cells at different myogenic status during skeletal muscle wound healing in rats.
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Proinflammatory cytokine tumor necrosis factor (TNF)-like weak inducer of apoptosis (TWEAK) suppresses satellite cell self-renewal through inversely modulating Notch and NF-κB signaling pathways.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 22 October 2013
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Proinflammatory cytokine tumor ...... and NF-κB signaling pathways.
@en
Proinflammatory cytokine tumor necrosis factor
@nl
type
label
Proinflammatory cytokine tumor ...... and NF-κB signaling pathways.
@en
Proinflammatory cytokine tumor necrosis factor
@nl
prefLabel
Proinflammatory cytokine tumor ...... and NF-κB signaling pathways.
@en
Proinflammatory cytokine tumor necrosis factor
@nl
P2093
P2860
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P1476
Proinflammatory cytokine tumor ...... and NF-κB signaling pathways.
@en
P2093
Sajedah M Hindi
Vivek Mishra
Yuji Ogura
P2860
P304
35159-35169
P356
10.1074/JBC.M113.517300
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P577
2013-10-22T00:00:00Z