Excessive formation of hydroxyl radicals and aldehydic lipid peroxidation products in cultured skin fibroblasts from patients with complex I deficiency.
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Mitochondria, oxygen free radicals, and apoptosis.Novel germline variants identified in the inner mitochondrial membrane transporter TIMM44 and their role in predisposition to oncocytic thyroid carcinomasHuman NADH:ubiquinone oxidoreductase deficiency: radical changes in mitochondrial morphology?Chemistry and biochemistry of lipid peroxidation products.MnSOD overexpression prevents liver mitochondrial DNA depletion after an alcohol binge but worsens this effect after prolonged alcohol consumption in mice.Reactive oxygen species, heat stress and oxidative-induced mitochondrial damage. A review.Aldehyde adducts generated during lipid peroxidation modification of proteins.Effect of nitric oxide on naphthoquinone toxicity in endothelial cells: role of bioenergetic dysfunction and poly (ADP-ribose) polymerase activation.Multidrug resistance protein MRP1 protects against the toxicity of the major lipid peroxidation product 4-hydroxynonenalMenadione triggers cell death through ROS-dependent mechanisms involving PARP activation without requiring apoptosisElectron transport chain of Saccharomyces cerevisiae mitochondria is inhibited by H2O2 at succinate-cytochrome c oxidoreductase level without lipid peroxidation involvement.Effects of peroxisomal catalase inhibition on mitochondrial function.Complex I and complex III inhibition specifically increase cytosolic hydrogen peroxide levels without inducing oxidative stress in HEK293 cells.Alterations in mitochondrial function in a mouse model of hypertrophic cardiomyopathy.Oxidative Stress: Mechanistic Insights into Inherited Mitochondrial Disorders and Parkinson's Disease.Analysis of reactive oxygen species and antioxidant defenses in complex I deficient patients revealed a specific increase in superoxide dismutase activity.Mitochondrial network complexity and pathological decrease in complex I activity are tightly correlated in isolated human complex I deficiency.
P2860
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P2860
Excessive formation of hydroxyl radicals and aldehydic lipid peroxidation products in cultured skin fibroblasts from patients with complex I deficiency.
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article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on June 1997
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
Excessive formation of hydroxy ...... nts with complex I deficiency.
@en
Excessive formation of hydroxy ...... nts with complex I deficiency.
@nl
type
label
Excessive formation of hydroxy ...... nts with complex I deficiency.
@en
Excessive formation of hydroxy ...... nts with complex I deficiency.
@nl
prefLabel
Excessive formation of hydroxy ...... nts with complex I deficiency.
@en
Excessive formation of hydroxy ...... nts with complex I deficiency.
@nl
P2093
P2860
P356
P1476
Excessive formation of hydroxy ...... nts with complex I deficiency.
@en
P2093
B H Robinson
D C Lehotay
S Kassovska-Bratinova
S Pitkänen
P2860
P304
P356
10.1172/JCI119481
P407
P577
1997-06-01T00:00:00Z