Reactivation of fetal splicing programs in diabetic hearts is mediated by protein kinase C signaling.
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Feedback Regulation of Kinase Signaling Pathways by AREs and GREsTranscriptome complexity in cardiac development and diseases--an expanding universe between genome and phenomeMolecular and Cellular Mechanisms of Cardiovascular Disorders in DiabetesGene Expression Analyses during Spontaneous Reversal of Cardiomyopathy in Mice with Repressed Nuclear CUG-BP, Elav-Like Family (CELF) Activity in Heart Muscle.Identification of Targets of CUG-BP, Elav-Like Family Member 1 (CELF1) Regulation in Embryonic Heart Muscle.Identification of transcripts regulated by CUG-BP, Elav-like family member 1 (CELF1) in primary embryonic cardiomyocytes by RNA-seqAltered CELF1 binding to target transcripts in malignant T cells.Dysregulation of RBFOX2 Is an Early Event in Cardiac Pathogenesis of DiabetesRbfox2 function in RNA metabolism is impaired in hypoplastic left heart syndrome patient hearts.Emerging roles for RNA-binding proteins as effectors and regulators of cardiovascular disease.Ancient antagonism between CELF and RBFOX families tunes mRNA splicing outcomes.Transcriptome-Wide Analysis Reveals Modulation of Human Macrophage Inflammatory Phenotype Through Alternative Splicing.Long Noncoding RNAs and RNA-Binding Proteins in Oxidative Stress, Cellular Senescence, and Age-Related Diseases.Developmentally regulated alternative splicing is perturbed in type 1 diabetic skeletal muscle.The Histamine H1 Receptor Participates in the Increased Dorsal Telencephalic Neurogenesis in Embryos from Diabetic Rats.Emerging roles of RNA-binding proteins in diabetes and their therapeutic potential in diabetic complications.RNA-binding protein CUGBP1 regulates insulin secretion via activation of phosphodiesterase 3B in mice.Post-Translational Modifications and RNA-Binding Proteins.P38 MAPK/miR-1 are involved in the protective effect of EGCG in high glucose-induced Cx43 downregulation in neonatal rat cardiomyocytes.Research on the Relationships between Endogenous Biomarkers and Exogenous Toxic Substances of Acute Toxicity in Radix Aconiti.Alternative mRNA Splicing in the Pathogenesis of Obesity.
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P2860
Reactivation of fetal splicing programs in diabetic hearts is mediated by protein kinase C signaling.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 22 October 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Reactivation of fetal splicing ...... by protein kinase C signaling.
@en
Reactivation of fetal splicing ...... by protein kinase C signaling.
@nl
type
label
Reactivation of fetal splicing ...... by protein kinase C signaling.
@en
Reactivation of fetal splicing ...... by protein kinase C signaling.
@nl
prefLabel
Reactivation of fetal splicing ...... by protein kinase C signaling.
@en
Reactivation of fetal splicing ...... by protein kinase C signaling.
@nl
P2093
P2860
P356
P1476
Reactivation of fetal splicing ...... by protein kinase C signaling
@en
P2093
Curtis A Nutter
Gene W Yeo
Ming-Lung Hung
Muge N Kuyumcu-Martinez
Patrick Liu
Rosario Espejo
Vaibhav Deshmukh
P2860
P304
35372-35386
P356
10.1074/JBC.M113.507426
P407
P577
2013-10-22T00:00:00Z