Oligodendrocytes contribute to motor neuron death in ALS via SOD1-dependent mechanism. - Wikidata - wikimedia - TriplyDB

Oligodendrocytes contribute to motor neuron death in ALS via SOD1-dependent mechanism.

Oligodendrocytes contribute to motor neuron death in ALS via SOD1-dependent mechanism.

http://www.wikidata.org/entity/Q37369313

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Transfer of pathogenic and nonpathogenic cytosolic proteins between spinal cord motor neurons in vivo in chimeric mice Modelling amyotrophic lateral sclerosis: progress and possibilities Modelling FUS Mislocalisation in an In Vitro Model of Innervated Human Muscle.Oligodendroglia: metabolic supporters of neurons.Modeling the C9ORF72 repeat expansion mutation using human induced pluripotent stem cells.High content analysis in amyotrophic lateral sclerosis.Axon degeneration: make the Schwann cell great again.Implications of white matter damage in amyotrophic lateral sclerosis (Review).Misfolded SOD1 is not a primary component of sporadic ALS.In vivo genome editing improves motor function and extends survival in a mouse model of ALS.Amyotrophic lateral sclerosis as a protein level, non-genomic disease: Therapy with S2RM exosome released molecules.Amyotrophic lateral sclerosis as a protein level, non-genomic disease: Therapy with S2RM exosome released molecules.Current Advances and Limitations in Modeling ALS/FTD in a Dish Using Induced Pluripotent Stem Cells.Can Astrocytes Be a Target for Precision Medicine?Representing Diversity in the Dish: Using Patient-Derived in Vitro Models to Recreate the Heterogeneity of Neurological Disease.Comprehensive analysis to explain reduced or increased SOD1 enzymatic activity in ALS patients and their relatives.Translating SOD1 Gene Silencing toward the Clinic: A Highly Efficacious, Off-Target-free, and Biomarker-Supported Strategy for fALS.A small-molecule inhibitor of SOD1-Derlin-1 interaction ameliorates pathology in an ALS mouse model.Amyotrophic lateral sclerosis

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Oligodendrocytes contribute to motor neuron death in ALS via SOD1-dependent mechanism.

http://www.wikidata.org/entity/Q37369313

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 29 September 2016

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

@cs

name

Oligodendrocytes contribute to motor neuron death in ALS via SOD1-dependent mechanism.

@en

Oligodendrocytes contribute to motor neuron death in ALS via SOD1-dependent mechanism.

@nl

type

label

Oligodendrocytes contribute to motor neuron death in ALS via SOD1-dependent mechanism.

@en

Oligodendrocytes contribute to motor neuron death in ALS via SOD1-dependent mechanism.

@nl

prefLabel

Oligodendrocytes contribute to motor neuron death in ALS via SOD1-dependent mechanism.

@en

Oligodendrocytes contribute to motor neuron death in ALS via SOD1-dependent mechanism.

@nl

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PNAS.1607496113

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Proceedings of the National Academy of Sciences of the United States of America

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Oligodendrocytes contribute to motor neuron death in ALS via SOD1-dependent mechanism.

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Ashley Frakes

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Brian K Kaspar

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Candice C Askwith

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Carlos J Miranda

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Christine E Beattie

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Dana McTigue

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Jonathan Vick

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Lyndsey Braun

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Paul R Heath

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P2860

Unconventional translation of C9ORF72 GGGGCC expansion generates insoluble polypeptides specific to c9FTD/ALS

Expanded GGGGCC hexanucleotide repeat in noncoding region of C9ORF72 causes chromosome 9p-linked FTD and ALS

A hexanucleotide repeat expansion in C9ORF72 is the cause of chromosome 9p21-linked ALS-FTD

Induction of pluripotent stem cells from adult human fibroblasts by defined factors

SOD1 integrates signals from oxygen and glucose to repress respiration.

Mutations in the FUS/TLS gene on chromosome 16 cause familial amyotrophic lateral sclerosis

Mutations in FUS, an RNA processing protein, cause familial amyotrophic lateral sclerosis type 6

TDP-43 mutations in familial and sporadic amyotrophic lateral sclerosis

Aberrant localization of FUS and TDP43 is associated with misfolding of SOD1 in amyotrophic lateral sclerosis

TARDBP mutations in individuals with sporadic and familial amyotrophic lateral sclerosis

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10.1073/PNAS.1607496113

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Laura Ferraiuolo

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2016-09-29T00:00:00Z

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