Prevention of In vitro neutrophil adhesion to endothelial cells through shedding of L-selectin by C-reactive protein and peptides derived from C-reactive protein.
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Methods for analysis of matrix metalloproteinase regulation of neutrophil-endothelial cell adhesionTargeting C-Reactive Protein in Inflammatory Disease by Preventing Conformational ChangesA Haptotaxis Assay for Neutrophils using Optical Patterning and a High-content Approach.Relationship between the plasma concentration of C-reactive protein and severity of peripheral arterial diseaseHepatic acute-phase proteins control innate immune responses during infection by promoting myeloid-derived suppressor cell function.Endothelin-1 enhances neutrophil adhesion to human coronary artery endothelial cells: role of ET(A) receptors and platelet-activating factor.Extracellular signal-regulated kinase plays an essential role in endothelin-1-induced homotypic adhesion of human neutrophil granulocytesRole of monocytes in atherogenesis.Decreased CXCR1 and CXCR2 expression on neutrophils in anti-neutrophil cytoplasmic autoantibody-associated vasculitides potentially increases neutrophil adhesion and impairs migration.The connection between C-reactive protein and atherosclerosisReduction of C-reactive protein and the use of anti-hypertensives.C-reactive protein and diabetic retinopathy in Chinese patients with type 2 diabetes mellitusAn Intrinsically Disordered Motif Mediates Diverse Actions of Monomeric C-reactive Protein.Immune opsonins modulate BLyS/BAFF release in a receptor-specific fashion.Pretreatment C-reactive protein is a predictor for outcomes after reduced-intensity allogeneic hematopoietic cell transplantationRole of C-reactive protein in cerebrovascular disease: a critical review.C-reactive protein and inflammation: conformational changes affect function.Management of patients with respiratory infections in primary care: procalcitonin, C-reactive protein or both?C-reactive protein: a physiological activator of interleukin 6 receptor shedding.Neutrophil-derived reactive oxygen species can modulate neutrophil adhesion to endothelial cells in preeclampsia.IgA/IgM responses to tryptophan and tryptophan catabolites (TRYCATs) are differently associated with prenatal depression, physio-somatic symptoms at the end of term and premenstrual syndrome.Possible potentiation by certain antioxidants of the anti-inflammatory effects of diclofenac in rats.
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Prevention of In vitro neutrophil adhesion to endothelial cells through shedding of L-selectin by C-reactive protein and peptides derived from C-reactive protein.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on August 1997
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Prevention of In vitro neutrop ...... rived from C-reactive protein.
@en
Prevention of In vitro neutrop ...... rived from C-reactive protein.
@nl
type
label
Prevention of In vitro neutrop ...... rived from C-reactive protein.
@en
Prevention of In vitro neutrop ...... rived from C-reactive protein.
@nl
prefLabel
Prevention of In vitro neutrop ...... rived from C-reactive protein.
@en
Prevention of In vitro neutrop ...... rived from C-reactive protein.
@nl
P2093
P2860
P356
P1476
Prevention of In vitro neutrop ...... rived from C-reactive protein.
@en
P2093
P2860
P304
P356
10.1172/JCI119561
P407
P577
1997-08-01T00:00:00Z