cAMP stringently regulates human cathelicidin antimicrobial peptide expression in the mucosal epithelial cells by activating cAMP-response element-binding protein, AP-1, and inducible cAMP early repressor.

cAMP stringently regulates human cathelicidin antimicrobial peptide expression in the mucosal epithelial cells by activating cAMP-response element-binding protein, AP-1, and inducible cAMP early repressor.

Item
http://www.wikidata.org/entity/Q37372125
Ribavirin suppresses bacterial virulence by targeting LysR-type transcriptional regulatorsDNA methylation directly downregulates human cathelicidin antimicrobial peptide gene (CAMP) promoter activity.Evaluation of the ability of LL-37 to neutralise LPS in vitro and ex vivo.Higher plasma vitamin D is associated with reduced risk of Clostridium difficile infection in patients with inflammatory bowel diseases.Vitamin D deficiency is associated with community-acquired clostridium difficile infection: a case-control study.LL-37 in periodontal health and disease and its susceptibility to degradation by proteinases present in gingival crevicular fluid.An inducible and secreted eukaryote-like serine/threonine kinase of Salmonella enterica serovar Typhi promotes intracellular survival and pathogenesisSynergistic induction of human cathelicidin antimicrobial peptide gene expression by vitamin D and stilbenoids.PTH/PTHrP and vitamin D control antimicrobial peptide expression and susceptibility to bacterial skin infection.Physiological TLR5 expression in the intestine is regulated by differential DNA binding of Sp1/Sp3 through simultaneous Sp1 dephosphorylation and Sp3 phosphorylation by two different PKC isoformsAvian antimicrobial host defense peptides: from biology to therapeutic applications.Unique features of human cathelicidin LL-37.MUC2 Mucin and Butyrate Contribute to the Synthesis of the Antimicrobial Peptide Cathelicidin in Response to Entamoeba histolytica- and Dextran Sodium Sulfate-Induced Colitis.Cathelicidin signaling via the Toll-like receptor protects against colitis in mice.An AIL family protein promotes type three secretion system-1-independent invasion and pathogenesis of Salmonella enterica serovar Typhi.Bacterial subversion of cAMP signalling inhibits cathelicidin expression, which is required for innate resistance to Mycobacterium tuberculosis.Suppression of Spleen Tyrosine Kinase (Syk) by Histone Deacetylation Promotes, Whereas BAY61-3606, a Synthetic Syk Inhibitor Abrogates Colonocyte Apoptosis by ERK Activation.
P2860

cAMP stringently regulates human cathelicidin antimicrobial peptide expression in the mucosal epithelial cells by activating cAMP-response element-binding protein, AP-1, and inducible cAMP early repressor.

Item
http://www.wikidata.org/entity/Q37372125
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 16 June 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
cAMP stringently regulates hum ...... nducible cAMP early repressor.
@en
cAMP stringently regulates hum ...... nducible cAMP early repressor.
@nl
type
label
cAMP stringently regulates hum ...... nducible cAMP early repressor.
@en
cAMP stringently regulates hum ...... nducible cAMP early repressor.
@nl
prefLabel
cAMP stringently regulates hum ...... nducible cAMP early repressor.
@en
cAMP stringently regulates hum ...... nducible cAMP early repressor.
@nl
P1433
P1476
P2093
P2860
P304
P31
P356
P407
P433
P478
P577
P698
P932
P356
P1433
P1476
cAMP stringently regulates hum ...... nducible cAMP early repressor.
@en
P2093
P2860
P304
P31
P356
P407
P433
P478
P577
P698
P932