about
Exosomes: vehicles for the transfer of toxic proteins associated with neurodegenerative diseases?The neutral sphingomyelinase pathway regulates packaging of the prion protein into exosomes.Stimulating the Release of Exosomes Increases the Intercellular Transfer of PrionsPrion infection impairs cholesterol metabolism in neuronal cells.Pathogenic mutations within the hydrophobic domain of the prion protein lead to the formation of protease-sensitive prion species with increased lethality.The secret life of extracellular vesicles in metal homeostasis and neurodegeneration.Dysregulation of the intrinsic apoptotic pathway mediates megakaryocytic hyperplasia in myeloproliferative neoplasms.The C-terminal domain of Escherichia coli dihydrodipicolinate synthase (DHDPS) is essential for maintenance of quaternary structure and efficient catalysis.Megakaryocytes in Myeloproliferative Neoplasms Have Unique Somatic Mutations.Megakaryocytic hyperplasia in myeloproliferative neoplasms is driven by disordered proliferative, apoptotic and epigenetic mechanisms.Automated enumeration of lymphoid and plasma cells in bone marrow to establish normal reference rangesA mutation in PTPN11 may drive leukemic transformation in a case of essential thrombocythemiaPlatelets in myeloproliferative neoplasms have a distinct transcript signature in the presence of marrow fibrosis
P50
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P50
description
cancer researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Belinda B Guo
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Belinda B Guo
@nl
Belinda B Guo
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Belinda Guo
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Belinda Guo
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type
label
Belinda B Guo
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Belinda B Guo
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Belinda B Guo
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Belinda Guo
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Belinda Guo
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altLabel
Belinda B. Guo
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prefLabel
Belinda B Guo
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Belinda B Guo
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Belinda B Guo
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Belinda Guo
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Belinda Guo
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P1960
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P2038
Belinda_Guo
P21
P31
P3829
P496
0000-0003-2146-8561