Anti-sense transforming growth factor alpha oligonucleotides inhibit autocrine stimulated proliferation of a colon carcinoma cell line
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Role of transforming growth factor-alpha in von Hippel--Lindau (VHL)(-/-) clear cell renal carcinoma cell proliferation: a possible mechanism coupling VHL tumor suppressor inactivation and tumorigenesisAberrant regulation of transforming growth factor-alpha during the establishment of growth arrest and quiescence of growth factor independent cells.Regulation of transforming growth factor alpha expression in a growth factor-independent cell lineMembrane-anchored growth factors, the epidermal growth factor family: beyond receptor ligands.Molecular mechanisms of resistance to the EGFR monoclonal antibody cetuximab.Autocrine stimulation of a human lung mesothelioma cell line is mediated through the transforming growth factor alpha/epidermal growth factor receptor mitogenic pathwayTheaflavin-3, 3'-digallate induces epidermal growth factor receptor downregulation.Lapatinib resistance in HCT116 cells is mediated by elevated MCL-1 expression and decreased BAK activation and not by ERBB receptor kinase mutationEGFR family: structure physiology signalling and therapeutic targets.Amphiregulin anti-sense oligodeoxynucleotides inhibit growth and transformation of a human colon carcinoma cell line.The EGF/TGFalpha response element within the TGFalpha promoter consists of a multi-complex regulatory element.Resistance to Therapy.Calcium, a Cell Cycle Commander, Drives Colon Cancer Cell Diffpoptosis.Fucoidan from Laminaria cichorioides inhibits AP-1 transactivation and cell transformation in the mouse epidermal JB6 cells.Inhibition of bone resorption in vitro by antisense RNA and DNA molecules targeted against carbonic anhydrase II or two subunits of vacuolar H(+)-ATPaseGrowth control mechanisms in normal and transformed intestinal cellsGastrointestinal hormone mRNA expression in human colonic adenocarcinomas, hepatic metastases and cell lines.Multiple autocrine factors including an extracellular matrix protein are required for the proliferation and spreading of human colon carcinoma cells in vitro.TGF-alpha antisense gene therapy inhibits head and neck squamous cell carcinoma growth in vivo.High-throughput screening identified inherited genetic variations in the EGFR pathway contributing to skin toxicity of EGFR inhibitors.Derivation of phenobarbital-responsive immortal rat hepatocytes.Regulation of autocrine gastrin expression by the TGF alpha autocrine loop.Infection with a transforming growth factor alpha anti-sense retroviral expression vector reduces the in vitro growth and transformation of a human colon cancer cell line.Nephritogenic T cells use granzyme C as a cytotoxic mediator.Simultaneous blockage of different EGF-like growth factors results in efficient growth inhibition of human colon carcinoma xenografts.
P2860
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P2860
Anti-sense transforming growth factor alpha oligonucleotides inhibit autocrine stimulated proliferation of a colon carcinoma cell line
description
1992 nî lūn-bûn
@nan
1992年の論文
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1992年学术文章
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1992年学术文章
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1992年学术文章
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1992年学术文章
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1992年学术文章
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1992年學術文章
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1992年學術文章
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name
Anti-sense transforming growth ...... of a colon carcinoma cell line
@en
Anti-sense transforming growth ...... f a colon carcinoma cell line.
@nl
type
label
Anti-sense transforming growth ...... of a colon carcinoma cell line
@en
Anti-sense transforming growth ...... f a colon carcinoma cell line.
@nl
prefLabel
Anti-sense transforming growth ...... of a colon carcinoma cell line
@en
Anti-sense transforming growth ...... f a colon carcinoma cell line.
@nl
P2860
P356
P1476
Anti-sense transforming growth ...... of a colon carcinoma cell line
@en
P2093
Burgess AW
Sizeland AM
P2860
P304
P356
10.1091/MBC.3.11.1235
P577
1992-11-01T00:00:00Z