Monocyte activation in angiogenesis and collateral growth in the rabbit hindlimb.
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Etk/Bmx as a tumor necrosis factor receptor type 2-specific kinase: role in endothelial cell migration and angiogenesisHost microenvironment in breast cancer development: inflammatory and immune cells in tumour angiogenesis and arteriogenesisPeripheral monocytes from diabetic patients with coronary artery disease display increased bFGF and VEGF mRNA expressionSPECT and PET imaging of angiogenesis and arteriogenesis in pre-clinical models of myocardial ischemia and peripheral vascular diseaseMonocyte chemotactic protein (MCP)-1 promotes angiogenesis via a novel transcription factor, MCP-1-induced protein (MCPIP)Etk/Bmx transactivates vascular endothelial growth factor 2 and recruits phosphatidylinositol 3-kinase to mediate the tumor necrosis factor-induced angiogenic pathwayVEGFR1/CXCR4-positive progenitor cells modulate local inflammation and augment tissue perfusion by a SDF-1-dependent mechanismVascular growth responses to chronic arterial occlusion are unaffected by myeloid specific focal adhesion kinase (FAK) deletion.Macrophages: An Inflammatory Link Between Angiogenesis and Lymphangiogenesis.Contrast ultrasound targeted drug and gene delivery: an update on a new therapeutic modality.Macrophage Recruitment and Polarization During Collateral Vessel Remodeling in Murine Adipose Tissue.Monocytes are recruited from venules during arteriogenesis in the murine spinotrapezius ligation model.Proximal superficial femoral artery occlusion, collateral vessels, and walking performance in peripheral artery disease.Exogenous thrombin delivery promotes collateral capillary arterialization and tissue reperfusion in the murine spinotrapezius muscle ischemia model.Spatial and temporal coordination of bone marrow-derived cell activity during arteriogenesis: regulation of the endogenous response and therapeutic implications.Arteriolar remodeling following ischemic injury extends from capillary to large arteriole in the microcirculation.Bone marrow-derived cell-specific chemokine (C-C motif) receptor-2 expression is required for arteriolar remodeling.Capillary arterialization requires the bone-marrow-derived cell (BMC)-specific expression of chemokine (C-C motif) receptor-2, but BMCs do not transdifferentiate into microvascular smooth muscle.Molecular imaging of the paracrine proangiogenic effects of progenitor cell therapy in limb ischemia.Dysregulated selectin expression and monocyte recruitment during ischemia-related vascular remodeling in diabetes mellitusInhibition of delta-like-4-mediated signaling impairs reparative angiogenesis after ischemia.Macrophages in collateral arteriogenesis.Overexpression of catalase in myeloid cells causes impaired postischemic neovascularization.Impairment in ischemia-induced neovascularization in diabetes: bone marrow mononuclear cell dysfunction and therapeutic potential of placenta growth factor treatmentInhibitory effect of bucillamine on laser-induced choroidal neovascularization in rats.Effective transfection of a cis element "decoy" of the nuclear factor-kappaB binding site into the experimental choroidal neovascularization.Arteriogenesis in hypoperfused rat brain.Haptoglobin expression and activity during coronary collateralization.Beneficial effects of concurrent autologous bone marrow cell therapy and metabolic intervention in ischemia-induced angiogenesis in the mouse hindlimbIschemia is not required for arteriogenesis in zebrafish embryos.Tissue-engineered vascular grafts transform into mature blood vessels via an inflammation-mediated process of vascular remodeling.LKB1 deficiency in Tie2-Cre-expressing cells impairs ischemia-induced angiogenesis.Angiogenesis and vasculogenesis as therapeutic strategies for postnatal neovascularizationPleiotrophin (PTN) is expressed in vascularized human atherosclerotic plaques: IFN-{gamma}/JAK/STAT1 signaling is critical for the expression of PTN in macrophages.The CD200-CD200 receptor inhibitory axis controls arteriogenesis and local T lymphocyte influx.Arteriogenesis, the good and bad of it.Redox-dependent mechanisms in coronary collateral growth: the "redox window" hypothesisMonocyte activation state regulates monocyte-induced endothelial proliferation through Met signaling.Impact of genetic background and aging on mesenteric collateral growth capacity in Fischer 344, Brown Norway, and Fischer 344 x Brown Norway hybrid rats.Dll4-Notch signaling determines the formation of native arterial collateral networks and arterial function in mouse ischemia models.
P2860
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P2860
Monocyte activation in angiogenesis and collateral growth in the rabbit hindlimb.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on January 1998
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Monocyte activation in angiogenesis and collateral growth in the rabbit hindlimb.
@en
Monocyte activation in angiogenesis and collateral growth in the rabbit hindlimb.
@nl
type
label
Monocyte activation in angiogenesis and collateral growth in the rabbit hindlimb.
@en
Monocyte activation in angiogenesis and collateral growth in the rabbit hindlimb.
@nl
prefLabel
Monocyte activation in angiogenesis and collateral growth in the rabbit hindlimb.
@en
Monocyte activation in angiogenesis and collateral growth in the rabbit hindlimb.
@nl
P2093
P2860
P356
P1476
Monocyte activation in angiogenesis and collateral growth in the rabbit hindlimb.
@en
P2093
P2860
P356
10.1172/JCI119877
P407
P577
1998-01-01T00:00:00Z