Molecular analysis of the role of the group A streptococcal cysteine protease, hyaluronic acid capsule, and M protein in a murine model of human invasive soft-tissue infection.
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Cytotoxic effects of streptolysin o and streptolysin s enhance the virulence of poorly encapsulated group a streptococciPerR confers phagocytic killing resistance and allows pharyngeal colonization by group A StreptococcusEmergence of a New Highly Successful Acapsular Group A Streptococcus Clade of Genotype emm89 in the United KingdomPathogenesis of group A streptococcal infectionsTracing the evolutionary history of the pandemic group A streptococcal M1T1 clone.IdeS, a novel streptococcal cysteine proteinase with unique specificity for immunoglobulin G.Interactions with fibronectin attenuate the virulence of Streptococcus pyogenesA naturally occurring mutation in ropB suppresses SpeB expression and reduces M1T1 group A streptococcal systemic virulence.SpeB of Streptococcus pyogenes differentially modulates antibacterial and receptor activating properties of human chemokines.Functional dissection of Streptococcus pyogenes M5 protein: the hypervariable region is essential for virulence.Absence of a cysteine protease effect on bacterial virulence in two murine models of human invasive group A streptococcal infection.NADase as a target molecule of in vivo suppression of the toxicity in the invasive M-1 group A Streptococcal isolates.Inhibition of dendritic cell maturation by group A StreptococcusInhibition of Inflammasome-Dependent Interleukin 1β Production by Streptococcal NAD+-Glycohydrolase: Evidence for Extracellular Activity.Cytolysin-dependent evasion of lysosomal killingVirulence gene regulation inside and outsideMutational analysis of the group A streptococcal operon encoding streptolysin S and its virulence role in invasive infection.A response regulator that represses transcription of several virulence operons in the group A streptococcus.The rgg gene of Streptococcus pyogenes NZ131 positively influences extracellular SPE B production.Absence of SpeB production in virulent large capsular forms of group A streptococcal strain 64Role of group A streptococcal virulence factors in adherence to keratinocytes.Characterization of an isogenic mutant of Streptococcus pyogenes Manfredo lacking the ability to make streptococcal acid glycoproteinInverse relation between disease severity and expression of the streptococcal cysteine protease, SpeB, among clonal M1T1 isolates recovered from invasive group A streptococcal infection casesRole of streptolysin O in a mouse model of invasive group A streptococcal disease.Identification and characterization of the scl gene encoding a group A Streptococcus extracellular protein virulence factor with similarity to human collagen.Restoration of Mga function to a Streptococcus pyogenes strain (M Type 50) that is virulent in miceImproved pattern for genome-based screening identifies novel cell wall-attached proteins in gram-positive bacteriaReciprocal, temporal expression of SpeA and SpeB by invasive M1T1 group a streptococcal isolates in vivo.Role of NADase in virulence in experimental invasive group A streptococcal infection.Signal transduction through CsrRS confers an invasive phenotype in group A Streptococcus.Two DNA-binding domains of Mga are required for virulence gene activation in the group A streptococcus.From transcription to activation: how group A streptococcus, the flesh-eating pathogen, regulates SpeB cysteine protease production.Cysteine proteinase SpeB from Streptococcus pyogenes - a potent modifier of immunologically important host and bacterial proteins.Streptolysin O and NAD-glycohydrolase prevent phagolysosome acidification and promote group A Streptococcus survival in macrophages.Platelet activation by Streptococcus pyogenes leads to entrapment in platelet aggregates, from which bacteria subsequently escape.Regulation of SpeB in Streptococcus pyogenes by pH and NaCl: a model for in vivo gene expressionDifferential recognition of surface proteins in Streptococcus pyogenes by two sortase gene homologs.Rheumatic fever in the 21st century.The contribution of group A streptococcal virulence determinants to the pathogenesis of sepsis.Antimicrobial Peptide Resistance Mechanisms of Gram-Positive Bacteria.
P2860
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P2860
Molecular analysis of the role of the group A streptococcal cysteine protease, hyaluronic acid capsule, and M protein in a murine model of human invasive soft-tissue infection.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on August 1998
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Molecular analysis of the role ...... nvasive soft-tissue infection.
@en
Molecular analysis of the role ...... nvasive soft-tissue infection.
@nl
type
label
Molecular analysis of the role ...... nvasive soft-tissue infection.
@en
Molecular analysis of the role ...... nvasive soft-tissue infection.
@nl
prefLabel
Molecular analysis of the role ...... nvasive soft-tissue infection.
@en
Molecular analysis of the role ...... nvasive soft-tissue infection.
@nl
P2093
P2860
P356
P1476
Molecular analysis of the role ...... nvasive soft-tissue infection.
@en
P2093
C D Ashbaugh
H B Warren
M R Wessels
P2860
P304
P356
10.1172/JCI3065
P407
P577
1998-08-01T00:00:00Z