Exposure to bacterial products renders macrophages highly susceptible to T-tropic HIV-1.
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Contribution of immune activation to the pathogenesis and transmission of human immunodeficiency virus type 1 infectionHumoral immune responses to Streptococcus pneumoniae in the setting of HIV-1 infectionCyclin T1-dependent genes in activated CD4 T and macrophage cell lines appear enriched in HIV-1 co-factors.Cytokine responses to treponema pectinovorum and treponema denticola in human gingival fibroblasts.Human peritoneal macrophages from ascitic fluid can be infected by a broad range of HIV-1 isolates.CCR5 signal transduction in macrophages by human immunodeficiency virus and simian immunodeficiency virus envelopesCathepsin G, a neutrophil-derived serine protease, increases susceptibility of macrophages to acute human immunodeficiency virus type 1 infectionMutagenesis of CXCR4 identifies important domains for human immunodeficiency virus type 1 X4 isolate envelope-mediated membrane fusion and virus entry and reveals cryptic coreceptor activity for R5 isolatesHuman immunodeficiency virus type 1 (HIV-1)-induced GRO-alpha production stimulates HIV-1 replication in macrophages and T lymphocytesMacrophage defences against respiratory tract infections.Bacterial lipopolysaccharide enhances cardiac dysfunction but not retroviral replication in murine AIDS: roles of macrophage infiltration and toll-like receptor 4 expression.Nuclear receptor signaling inhibits HIV-1 replication in macrophages through multiple trans-repression mechanismsProline-proline-glutamic acid (PPE) protein Rv1168c of Mycobacterium tuberculosis augments transcription from HIV-1 long terminal repeat promoter.Neisseria gonorrhoeae enhances HIV-1 infection of primary resting CD4+ T cells through TLR2 activation.Coreceptor competition for association with CD4 may change the susceptibility of human cells to infection with T-tropic and macrophagetropic isolates of human immunodeficiency virus type 1Fcgamma receptor-mediated suppression of human immunodeficiency virus type 1 replication in primary human macrophages.Cell-type-dependent effect of transforming growth factor beta, a major cytokine in breast milk, on human immunodeficiency virus type 1 infection of mammary epithelial MCF-7 cells or macrophages.A CD14-independent LPS receptor cluster.Comparative analysis of the capacity of elite suppressor CD4+ and CD8+ T cells to inhibit HIV-1 replication in monocyte-derived macrophages.Human alveolar macrophages and granulocyte-macrophage colony-stimulating factor-induced monocyte-derived macrophages are resistant to H2O2 via their high basal and inducible levels of catalase activity.
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P2860
Exposure to bacterial products renders macrophages highly susceptible to T-tropic HIV-1.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
@tr
scientific article published on October 1998
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Exposure to bacterial products renders macrophages highly susceptible to T-tropic HIV-1.
@en
Exposure to bacterial products renders macrophages highly susceptible to T-tropic HIV-1.
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type
label
Exposure to bacterial products renders macrophages highly susceptible to T-tropic HIV-1.
@en
Exposure to bacterial products renders macrophages highly susceptible to T-tropic HIV-1.
@nl
prefLabel
Exposure to bacterial products renders macrophages highly susceptible to T-tropic HIV-1.
@en
Exposure to bacterial products renders macrophages highly susceptible to T-tropic HIV-1.
@nl
P2093
P2860
P356
P1476
Exposure to bacterial products renders macrophages highly susceptible to T-tropic HIV-1.
@en
P2093
P2860
P304
P356
10.1172/JCI4151
P407
P577
1998-10-01T00:00:00Z