What have worm models told us about the mechanisms of neuronal dysfunction in human neurodegenerative diseases?
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Integrated control of protein degradation in C. elegans muscleAlpha-synuclein and tau: teammates in neurodegeneration?Neuroprotective effects and mechanism of cognitive-enhancing choline analogs JWB 1-84-1 and JAY 2-22-33 in neuronal culture and Caenorhabditis elegans.Drosophila melanogaster in the study of human neurodegeneration.Identification of common genetic modifiers of neurodegenerative diseases from an integrative analysis of diverse genetic screens in model organismsDrosophila melanogaster as a model organism for Alzheimer's disease.A cocoa peptide protects Caenorhabditis elegans from oxidative stress and β-amyloid peptide toxicity.Human disease models in Drosophila melanogaster and the role of the fly in therapeutic drug discovery.Deciphering the role of natural variation in age-related protein homeostasis.Pharmacological maintenance of protein homeostasis could postpone age-related diseaseUtility of an improved model of amyloid-beta (Aβ₁₋₄₂) toxicity in Caenorhabditis elegans for drug screening for Alzheimer's diseaseStudying polyglutamine aggregation in Caenorhabditis elegans using an analytical ultracentrifuge equipped with fluorescence detection.Modeling Alzheimer's disease: from past to futureO-GlcNAc cycling shows neuroprotective potential in C. elegans models of neurodegenerative diseaseAlzheimer's disease and tauopathy studies in flies and worms.Neurodegenerative disorders: insights from the nematode Caenorhabditis elegansModeling human neurodegenerative diseases in transgenic systems.Role of the ubiquitin-proteasome system in nervous system function and disease: using C. elegans as a dissecting tool.Behind the curtain of tauopathy: a show of multiple players orchestrating tau toxicity.Optofluidic holographic microscopy with custom field of view (FoV) using a linear array detector.Chronic exposure to a glyphosate-containing pesticide leads to mitochondrial dysfunction and increased reactive oxygen species production in Caenorhabditis elegans.High-Content Microfluidic Screening Platform Used To Identify σ2R/Tmem97 Binding Ligands that Reduce Age-Dependent Neurodegeneration in C. elegans SC_APP Model.Transactivation Domain of Human c-Myc Is Essential to Alleviate Poly(Q)-Mediated Neurotoxicity in Drosophila Disease Models.Rapid Integration of Multi-copy Transgenes Using Optogenetic Mutagenesis in Caenorhabditis elegans.Protein Co-Aggregation Related to Amyloids: Methods of Investigation, Diversity, and Classification
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What have worm models told us about the mechanisms of neuronal dysfunction in human neurodegenerative diseases?
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 28 September 2009
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vedecký článok
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vetenskaplig artikel
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name
What have worm models told us ...... an neurodegenerative diseases?
@en
What have worm models told us ...... an neurodegenerative diseases?
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type
label
What have worm models told us ...... an neurodegenerative diseases?
@en
What have worm models told us ...... an neurodegenerative diseases?
@nl
prefLabel
What have worm models told us ...... an neurodegenerative diseases?
@en
What have worm models told us ...... an neurodegenerative diseases?
@nl
P2860
P356
P1476
What have worm models told us ...... an neurodegenerative diseases?
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P2093
Christopher D Link
Dawn Teschendorf
P2860
P2888
P356
10.1186/1750-1326-4-38
P577
2009-09-28T00:00:00Z
P5875
P6179
1033893215