CDK5 promotes renal tubulointerstitial fibrosis in diabetic nephropathy via ERK1/2/PPARγ pathway
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Uninephrectomized High-Fat-Fed Nicotinamide-Streptozotocin-Induced Diabetic Rats: A Model for the Investigation of Diabetic Nephropathy in Type 2 Diabetes.MicroRNA-27a promotes podocyte injury via PPARγ-mediated β-catenin activation in diabetic nephropathy.MicroRNA-27a promotes renal tubulointerstitial fibrosis via suppressing PPARγ pathway in diabetic nephropathy.The non-neuronal cyclin-dependent kinase 5 is a fibrotic mediator potentially implicated in systemic sclerosis and a novel therapeutic target.
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CDK5 promotes renal tubulointerstitial fibrosis in diabetic nephropathy via ERK1/2/PPARγ pathway
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 27 April 2016
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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CDK5 promotes renal tubulointe ...... pathy via ERK1/2/PPARγ pathway
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CDK5 promotes renal tubulointe ...... athy via ERK1/2/PPARγ pathway.
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label
CDK5 promotes renal tubulointe ...... pathy via ERK1/2/PPARγ pathway
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CDK5 promotes renal tubulointe ...... athy via ERK1/2/PPARγ pathway.
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prefLabel
CDK5 promotes renal tubulointe ...... pathy via ERK1/2/PPARγ pathway
@en
CDK5 promotes renal tubulointe ...... athy via ERK1/2/PPARγ pathway.
@nl
P2093
P2860
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CDK5 promotes renal tubulointe ...... pathy via ERK1/2/PPARγ pathway
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P2093
Jianwei Tian
Xiaoyan Bai
Xiaoyan Hou
P2860
P304
36510-36528
P356
10.18632/ONCOTARGET.9058
P407
P577
2016-04-27T00:00:00Z