Candida albicans infection affords protection against reinfection via functional reprogramming of monocytes. - Wikidata - wikimedia - TriplyDB

Candida albicans infection affords protection against reinfection via functional reprogramming of monocytes.

Candida albicans infection affords protection against reinfection via functional reprogramming of monocytes.

http://www.wikidata.org/entity/Q37394444

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Immune Dysfunction as a Cause and Consequence of Malnutrition Cryptococcus and Phagocytes: Complex Interactions that Influence Disease Outcome The Unspecific Side of Acquired Immunity Against Infectious Disease: Causes and Consequences How Chemotherapy Increases the Risk of Systemic Candidiasis in Cancer Patients: Current Paradigm and Future Directions Epigenetic Modulators of Monocytic Function: Implication for Steady State and Disease in the CNS Innate Immune Memory: The Latest Frontier of Adjuvanticity New Insights Into Tissue Macrophages: From Their Origin to the Development of Memory IL-17-Mediated Immunity to the Opportunistic Fungal Pathogen Candida albicans From genetics of inflammatory bowel disease towards mechanistic insights Ontogeny of early life immunity Immunological memory within the innate immune system Immune mechanisms in cerebral ischemic tolerance Neutrophil Attack Triggers Extracellular Trap-Dependent Candida Cell Wall Remodeling and Altered Immune Recognition Is boosting the immune system in sepsis appropriate?Diabetes propels the risk for cardiovascular disease: sweet monocytes becoming aggressive?Cell biology of Candida albicans-host interactions 'Trained immunity': consequences for lymphoid malignancies The effects of orally administered Beta-glucan on innate immune responses in humans, a randomized open-label intervention pilot-study Role of Serum Amyloid A, Granulocyte-Macrophage Colony-Stimulating Factor, and Bone Marrow Granulocyte-Monocyte Precursor Expansion in Segmented Filamentous Bacterium-Mediated Protection from Entamoeba histolytica.GM-CSF and IL-3 Modulate Human Monocyte TNF-α Production and Renewal in In Vitro Models of Trained Immunity.Primed immune responses to gram-negative peptidoglycans confer infection resistance in silkworms.Immunity against fungi.Inflammatory and myeloid-associated gene expression before and one day after infant vaccination with MVA85A correlates with induction of a T cell response Different Regulation of Interleukin-1 Production and Activity in Monocytes and Macrophages: Innate Memory as an Endogenous Mechanism of IL-1 Inhibition.Innate Lung Defense during Invasive Aspergillosis: New Mechanisms.Better immunity in later life: a position paper.From Monocytes to M1/M2 Macrophages: Phenotypical vs. Functional Differentiation.mTOR- and HIF-1α-mediated aerobic glycolysis as metabolic basis for trained immunity.Candida and host determinants of susceptibility to invasive candidiasis Innate immune programing by endotoxin and its pathological consequences BTB-ZF transcriptional regulator PLZF modifies chromatin to restrain inflammatory signaling programs.Innate lymphoid cells: new players in IL-17-mediated antifungal immunity.Inflammatory monocytes orchestrate innate antifungal immunity in the lung.Candida albicans morphology and dendritic cell subsets determine T helper cell differentiation.Exposure-dependent control of malaria-induced inflammation in children.Syk signaling in dendritic cells orchestrates innate resistance to systemic fungal infection.Small-molecule control of cytokine function: new opportunities for treating immune disorders Autophagy controls BCG-induced trained immunity and the response to intravesical BCG therapy for bladder cancer.Innate Defense against Fungal Pathogens Monosodium Urate Crystals Promote Innate Anti-Mycobacterial Immunity and Improve BCG Efficacy as a Vaccine against Tuberculosis.

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Candida albicans infection affords protection against reinfection via functional reprogramming of monocytes.

http://www.wikidata.org/entity/Q37394444

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artigo científico

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bilimsel makale

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scientific article published on August 2012

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vedecký článok

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vetenskaplig artikel

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Candida albicans infection aff ...... al reprogramming of monocytes.

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Candida albicans infection aff ...... al reprogramming of monocytes.

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Candida albicans infection aff ...... al reprogramming of monocytes.

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Candida albicans infection aff ...... al reprogramming of monocytes.

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Candida albicans infection aff ...... al reprogramming of monocytes.

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Candida albicans infection aff ...... al reprogramming of monocytes.

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J.CHOM.2012.06.006

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Cell Host & Microbe

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Candida albicans infection aff ...... nal reprogramming of monocytes

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Bart-Jan Kullberg

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Daniela C Ifrim

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Hendrik G Stunnenberg

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Jessica Quintin

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Jos W M van der Meer

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Leo A B Joosten

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Liesbeth Jacobs

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Mihai G Netea

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Ramnik J Xavier

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Sadia Saeed

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P2860

Genome-wide maps of chromatin state in pluripotent and lineage-committed cells

Human dectin-1 deficiency and mucocutaneous fungal infections

Set9, a novel histone H3 methyltransferase that facilitates transcription by precluding histone tail modifications required for heterochromatin formation.

High-resolution profiling of histone methylations in the human genome

Alternative activation of macrophages

Regulating RNA polymerase pausing and transcription elongation in embryonic stem cells

The chemokine system in diverse forms of macrophage activation and polarization

Epidemiology of invasive candidiasis: a persistent public health problem

TNF/iNOS-producing dendritic cells are the necessary evil of lethal influenza virus infection.

A specific primed immune response in Drosophila is dependent on phagocytes.

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223-232

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scholarly article

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10.1016/J.CHOM.2012.06.006

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2

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12

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Evangelos J Giamarellos-Bourboulis

Joost H A Martens

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2012-08-01T00:00:00Z

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22901542

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3864037

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