Dominant role of CD47-thrombospondin-1 interactions in myeloma-induced fusion of human dendritic cells: implications for bone disease. - Wikidata - wikimedia - TriplyDB

Dominant role of CD47-thrombospondin-1 interactions in myeloma-induced fusion of human dendritic cells: implications for bone disease.

Dominant role of CD47-thrombospondin-1 interactions in myeloma-induced fusion of human dendritic cells: implications for bone disease.

http://www.wikidata.org/entity/Q37396070

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Therapeutic opportunities for targeting the ubiquitous cell surface receptor CD47 The Dark Side of Cell Fusion Lack of CD47 impairs bone cell differentiation and results in an osteopenic phenotype in vivo due to impaired signal regulatory protein α (SIRPα) signaling.Advances in the understanding of myeloma bone disease and tumour growth.Multiple myeloma mesenchymal stromal cells: Contribution to myeloma bone disease and therapeutics Bone marrow microenvironment in myelomagenesis: its potential role in early diagnosis.Cytokine-mediated bone destruction in rheumatoid arthritis.Thrombospondin-1 derived from APCs regulates their capacity for allosensitization Revisiting the matricellular concept.Thrombospondin-1 regulates bone homeostasis through effects on bone matrix integrity and nitric oxide signaling in osteoclasts Thrombospondin-1 production is enhanced by Porphyromonas gingivalis lipopolysaccharide in THP-1 cells.Dendritic cells and malignant plasma cells: an alliance in multiple myeloma tumor progression?Dendritic cell-mediated activation-induced cytidine deaminase (AID)-dependent induction of genomic instability in human myeloma.Identification of TAX2 peptide as a new unpredicted anti-cancer agent.The Interplay between the bone and the immune system Mechanisms of multiple myeloma bone disease.New insights into the molecular pathogenesis of langerhans cell histiocytosis.Contributions of the host microenvironment to cancer-induced bone disease.Harnessing human dendritic cell subsets for medicine.Tumor-host cell interactions in the bone disease of myeloma.MGUS to myeloma: a mysterious gammopathy of underexplored significance.Inhibition of Transforming Growth Factor-β Activation Diminishes Tumor Progression and Osteolytic Bone Disease in Mouse Models of Multiple Myeloma.Vesicle transfer and cell fusion: Emerging concepts of cell-cell communication in the tumor microenvironment.CD24, CD27, CD36 and CD302 gene expression for outcome prediction in patients with multiple myeloma.Immature dendritic cells in multiple myeloma are prone to osteoclast-like differentiation through interleukin-17A stimulation.Myeloma bone disease: Pathophysiology and management.Checkpoint Inhibition in Myeloma: Opportunities and Challenges

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Dominant role of CD47-thrombospondin-1 interactions in myeloma-induced fusion of human dendritic cells: implications for bone disease.

http://www.wikidata.org/entity/Q37396070

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 06 August 2009

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Dominant role of CD47-thrombos ...... implications for bone disease.

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Dominant role of CD47-thrombos ...... implications for bone disease.

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type

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Dominant role of CD47-thrombos ...... implications for bone disease.

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Dominant role of CD47-thrombos ...... implications for bone disease.

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Dominant role of CD47-thrombos ...... implications for bone disease.

@en

Dominant role of CD47-thrombos ...... implications for bone disease.

@nl

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BLOOD-2009-03-211920

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Dominant role of CD47-thrombos ...... implications for bone disease.

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Anjli Kukreja

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Ben-Hua Sun

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Karl Insogna

http://www.w3.org/2001/XMLSchema#string

Madhav V Dhodapkar

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Soroosh Radfar

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P2860

Role of CD47 as a marker of self on red blood cells

Integrin-associated protein (CD47) and its ligands

RANKL-induced DC-STAMP is essential for osteoclastogenesis

Lack of CD47 on nonhematopoietic cells induces split macrophage tolerance to CD47null cells

Macrophage inflammatory protein-1alpha is an osteoclastogenic factor in myeloma that is independent of receptor activator of nuclear factor kappaB ligand.

Selective blockade of inhibitory Fcgamma receptor enables human dendritic cell maturation with IL-12p70 production and immunity to antibody-coated tumor cells.

Multiple myeloma disrupts the TRANCE/ osteoprotegerin cytokine axis to trigger bone destruction and promote tumor progression.

The cell biology of thrombospondin-1.

Global gene expression profiling of multiple myeloma, monoclonal gammopathy of undetermined significance, and normal bone marrow plasma cells.

Recruitment of new osteoblasts and osteoclasts is the earliest critical event in the pathogenesis of human multiple myeloma.

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scholarly article

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10.1182/BLOOD-2009-03-211920

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