IL-9-mediated survival of type 2 innate lymphoid cells promotes damage control in helminth-induced lung inflammation - Wikidata - wikimedia - TriplyDB

IL-9-mediated survival of type 2 innate lymphoid cells promotes damage control in helminth-induced lung inflammation

IL-9-mediated survival of type 2 innate lymphoid cells promotes damage control in helminth-induced lung inflammation

http://www.wikidata.org/entity/Q37397810

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The origin and role of innate lymphoid cells in the lung Type 2 innate lymphoid cells: at the cross-roads in allergic asthma Macrophage and Innate Lymphoid Cell Interplay in the Genesis of Fibrosis Deciphering Asthma Biomarkers with Protein Profiling Technology I-L-C-2 it: type 2 immunity and group 2 innate lymphoid cells in homeostasis The Differentiation of CD4(+) T-Helper Cell Subsets in the Context of Helminth Parasite Infection The importance of TSLP in allergic disease and its role as a potential therapeutic target Group 2 innate lymphoid cells in the lung Immune antibodies and helminth products drive CXCR2-dependent macrophage-myofibroblast crosstalk to promote intestinal repair Role of Macrophages in the Repair Process during the Tissue Migrating and Resident Helminth Infections Along the Axis between Type 1 and Type 2 Immunity; Principles Conserved in Evolution from Fish to Mammals The development and in vivo function of T helper 9 cells Characterization and Quantification of Innate Lymphoid Cell Subsets in Human Lung.Beyond NK cells: the expanding universe of innate lymphoid cells.Early local immune defences in the respiratory tract Importin-α7 is required for enhanced influenza A virus replication in the alveolar epithelium and severe lung damage in mice.MHCII-mediated dialog between group 2 innate lymphoid cells and CD4(+) T cells potentiates type 2 immunity and promotes parasitic helminth expulsion.Basophils promote innate lymphoid cell responses in inflamed skin.Cutting edge: maresin-1 engages regulatory T cells to limit type 2 innate lymphoid cell activation and promote resolution of lung inflammation Inhibition of adaptive immunity by IL9 can be disrupted to achieve rapid T-cell sensitization and rejection of progressive tumor challenges.Intranasal administration of Mycobacterium bovis BCG induces superior protection against aerosol infection with Mycobacterium tuberculosis in mice.Foxp3⁺ regulatory T cells delay expulsion of intestinal nematodes by suppression of IL-9-driven mast cell activation in BALB/c but not in C57BL/6 mice Tailored immune responses: novel effector helper T cell subsets in protective immunity.Chitinase-like proteins promote IL-17-mediated neutrophilia in a tradeoff between nematode killing and host damage.Group 2 innate lymphoid cell proportions are diminished in young helminth infected children and restored by curative anti-helminthic treatment.Transcriptional programs define molecular characteristics of innate lymphoid cell classes and subsets Interleukin-33 in Tissue Homeostasis, Injury, and Inflammation TH9 cells are required for tissue mast cell accumulation during allergic inflammation T helper 2 (Th2) cell differentiation, type 2 innate lymphoid cell (ILC2) development and regulation of interleukin-4 (IL-4) and IL-13 production.Cutting Edge: IL-25 Targets Dendritic Cells To Attract IL-9-Producing T Cells in Acute Allergic Lung Inflammation.The prostaglandin D₂ receptor CRTH2 regulates accumulation of group 2 innate lymphoid cells in the inflamed lung.Group 2 innate lymphoid cells utilize the IRF4-IL-9 module to coordinate epithelial cell maintenance of lung homeostasis Inflammatory group 2 innate lymphoid cells.Tissue residency of innate lymphoid cells in lymphoid and nonlymphoid organs.Intrinsic functional defects of type 2 innate lymphoid cells impair innate allergic inflammation in promyelocytic leukemia zinc finger (PLZF)-deficient mice.Emerging functions of amphiregulin in orchestrating immunity, inflammation, and tissue repair.The ETS1 transcription factor is required for the development and cytokine-induced expansion of ILC2.Innate lymphoid cells in the initiation, regulation and resolution of inflammation Arginase 1 is an innate lymphoid-cell-intrinsic metabolic checkpoint controlling type 2 inflammation Innate lymphoid cells in asthma: Will they take your breath away?

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IL-9-mediated survival of type 2 innate lymphoid cells promotes damage control in helminth-induced lung inflammation

http://www.wikidata.org/entity/Q37397810

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IL-9-mediated survival of type ...... inth-induced lung inflammation

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Jan-Eric Turner

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Mark Wilson

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Ulf Panzer

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P2860

Systemically dispersed innate IL-13-expressing cells in type 2 immunity

Transcription factor RORα is critical for nuocyte development

Nuocytes represent a new innate effector leukocyte that mediates type-2 immunity

IL25 elicits a multipotent progenitor cell population that promotes T(H)2 cytokine responses

The NF-kappaB regulator Bcl-3 and the BH3-only proteins Bim and Puma control the death of activated T cells

Identification of an interleukin (IL)-25-dependent cell population that provides IL-4, IL-5, and IL-13 at the onset of helminth expulsion

IL-25 induces IL-4, IL-5, and IL-13 and Th2-associated pathologies in vivo

The earliest T lineage-committed cells depend on IL-7 for Bcl-2 expression and normal cell cycle progression

IL-4 inhibits TGF-beta-induced Foxp3+ T cells and, together with TGF-beta, generates IL-9+ IL-10+ Foxp3(-) effector T cells

New IL-17 family members promote Th1 or Th2 responses in the lung: in vivo function of the novel cytokine IL-25

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210

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Peter J Morrison

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