Endothelial injury induces vascular smooth muscle cell proliferation in highly localized regions of a direct contact co-culture system. - Wikidata - wikimedia - TriplyDB

Endothelial injury induces vascular smooth muscle cell proliferation in highly localized regions of a direct contact co-culture system.

Endothelial injury induces vascular smooth muscle cell proliferation in highly localized regions of a direct contact co-culture system.

http://www.wikidata.org/entity/Q37405943

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Amniotic fluid-derived stem cells demonstrated cardiogenic potential in indirect co-culture with human cardiac cells.Improved penile histology by phalloidin stain: circular and longitudinal cavernous smooth muscles, dual-endothelium arteries, and erectile dysfunction-associated changes.Formation of functional gap junctions in amniotic fluid-derived stem cells induced by transmembrane co-culture with neonatal rat cardiomyocytes Endothelial control of vasodilation: integration of myoendothelial microdomain signalling and modulation by epoxyeicosatrienoic acids.Smooth muscle cells orchestrate the endothelial cell response to flow and injury.A Cell Culture Model of Resistance Arteries.Myoendothelial gap junctional signaling induces differentiation of pulmonary arterial smooth muscle cells.Endothelial-Vascular Smooth Muscle Cells Interactions in Atherosclerosis

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P2860

Endothelial injury induces vascular smooth muscle cell proliferation in highly localized regions of a direct contact co-culture system.

http://www.wikidata.org/entity/Q37405943

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article científic

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artigo científico

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bilimsel makale

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scientific article published on 03 September 2008

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vedecký článok

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vetenskaplig artikel

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Endothelial injury induces vas ...... ect contact co-culture system.

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Cell Biochemistry and Biophysics

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Endothelial injury induces vas ...... rect contact co-culture system

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Joyce Y Wong

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P2860

Role of PDGF-B and PDGFR-beta in recruitment of vascular smooth muscle cells and pericytes during embryonic blood vessel formation in the mouse

A subpopulation of smooth muscle cells in injured rat arteries expresses platelet-derived growth factor-B chain mRNA

Effects of connexin-mimetic peptides on gap junction functionality and connexin expression in cultured vascular cells

Shear stress-induced upregulation of connexin 43 expression in endothelial cells on upstream surfaces of rat cardiac valves

Heterocellular contact at the myoendothelial junction influences gap junction organization.

Ca2+ and inositol 1,4,5-trisphosphate-mediated signaling across the myoendothelial junction.

Atherosclerosis-prone hemodynamics differentially regulates endothelial and smooth muscle cell phenotypes and promotes pro-inflammatory priming.

Dye tracers define differential endothelial and smooth muscle coupling patterns within the arteriolar wall.

Morphology favors an endothelial cell pathway for longitudinal conduction within arterioles.

5-Methyltetrahydrofolate and tetrahydrobiopterin can modulate electrotonically mediated endothelium-dependent vascular relaxation.

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scholarly article

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10.1007/S12013-008-9023-6

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Jeffrey G Jacot

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