Role of MCP-1 in tumor necrosis factor-alpha-induced endothelial dysfunction in type 2 diabetic mice.
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Why is coronary collateral growth impaired in type II diabetes and the metabolic syndrome?Anti-Inflammatory Activity of Haskap Cultivars is Polyphenols-DependentProgressive histone alterations and proinflammatory gene activation: consequences of heme protein/iron-mediated proximal tubule injuryThe reciprocal relationship between adiponectin and LOX-1 in the regulation of endothelial dysfunction in ApoE knockout miceAdvanced glycation end products induce chemokine/cytokine production via activation of p38 pathway and inhibit proliferation and migration of bone marrow mesenchymal stem cells.Cardiovascular physiology at the bench for application in the clinic.miR-21-mediated decreased neutrophil apoptosis is a determinant of impaired coronary collateral growth in metabolic syndrome.Translational control of PML contributes to TNFα-induced apoptosis of MCF7 breast cancer cells and decreased angiogenesis in HUVECs.The link between metabolic abnormalities and endothelial dysfunction in type 2 diabetes: an update.Glucose and acute exercise influence factors secreted by circulating angiogenic cells in vitro.Diabetes and ageing-induced vascular inflammation.Association Between Tumor Necrosis Factor-α and Diabetic Peripheral Neuropathy in Patients with Type 2 Diabetes: a Meta-Analysis.Early Secreted Antigenic Target of 6 kDa of Mycobacterium tuberculosis Stimulates Macrophage Chemoattractant Protein-1 Production by Macrophages and Its Regulation by p38 Mitogen-Activated Protein Kinases and Interleukin-4.An overview of the inflammatory signalling mechanisms in the myocardium underlying the development of diabetic cardiomyopathy.The Role of Endoplasmic Reticulum Stress in Cardiovascular Disease and Exercise.Exercise Training Prevents Coronary Endothelial Dysfunction in Type 2 Diabetic MiceInteraction of IL-6 and TNF-α contributes to endothelial dysfunction in type 2 diabetic mouse hearts.Reduced monocyte adhesion to aortae of diabetic plasminogen activator inhibitor-1 knockout mice.Elevated 20-HETE impairs coronary collateral growth in metabolic syndrome via endothelial dysfunction.Anti-Inflammatory Activity of Marine Ovothiol A in an In Vitro Model of Endothelial Dysfunction Induced by Hyperglycemia.
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P2860
Role of MCP-1 in tumor necrosis factor-alpha-induced endothelial dysfunction in type 2 diabetic mice.
description
article científic
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article scientifique
@fr
articolo scientifico
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artigo científico
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bilimsel makale
@tr
scientific article published on 07 August 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Role of MCP-1 in tumor necrosi ...... ction in type 2 diabetic mice.
@en
Role of MCP-1 in tumor necrosi ...... ction in type 2 diabetic mice.
@nl
type
label
Role of MCP-1 in tumor necrosi ...... ction in type 2 diabetic mice.
@en
Role of MCP-1 in tumor necrosi ...... ction in type 2 diabetic mice.
@nl
prefLabel
Role of MCP-1 in tumor necrosi ...... ction in type 2 diabetic mice.
@en
Role of MCP-1 in tumor necrosi ...... ction in type 2 diabetic mice.
@nl
P2093
P2860
P1476
Role of MCP-1 in tumor necrosi ...... ction in type 2 diabetic mice.
@en
P2093
Cuihua Zhang
Emily Wilson
Jiyeon Yang
Louise Abbott
Yoonjung Park
P2860
P304
P356
10.1152/AJPHEART.00396.2009
P577
2009-08-07T00:00:00Z