A paradoxical protective role for the proinflammatory peptide substance P receptor (NK1R) in acute hyperoxic lung injury
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Lung endothelial HO-1 targeting in vivo using lentiviral miRNA regulates apoptosis and autophagy during oxidant injury.Ozone-induced airway epithelial cell death, the neurokinin-1 receptor pathway, and the postnatal developing lung.Cellular mechanism underlying formaldehyde-stimulated Cl- secretion in rat airway epithelium.Increased hyperoxia-induced lung injury in nitric oxide synthase 2 null mice is mediated via angiopoietin 2.Postnatal exposure history and airways: oxidant stress responses in airway explantsMarked Effects of Tachykinin in Myositis Both in the Experimental Side and Contralaterally: Studies on NK-1 Receptor Expressions in an Animal ModelC5L2, the Second C5a Anaphylatoxin Receptor, Suppresses LPS-Induced Acute Lung Injury.CD44 is protective during hyperoxia-induced lung injury.
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P2860
A paradoxical protective role for the proinflammatory peptide substance P receptor (NK1R) in acute hyperoxic lung injury
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 24 July 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
A paradoxical protective role ...... in acute hyperoxic lung injury
@en
A paradoxical protective role for the proinflammatory peptide substance P receptor
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type
label
A paradoxical protective role ...... in acute hyperoxic lung injury
@en
A paradoxical protective role for the proinflammatory peptide substance P receptor
@nl
prefLabel
A paradoxical protective role ...... in acute hyperoxic lung injury
@en
A paradoxical protective role for the proinflammatory peptide substance P receptor
@nl
P2093
P2860
P1476
A paradoxical protective role ...... in acute hyperoxic lung injury
@en
P2093
Alex Mitsialis
Craig Gerard
Marwan Dib
Norma P Gerard
Stewart Craig
P2860
P304
P356
10.1152/AJPLUNG.90509.2008
P577
2009-07-24T00:00:00Z