Effects of conditional central expression of HIV-1 tat protein to potentiate cocaine-mediated psychostimulation and reward among male mice.
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Interaction between Tat and Drugs of Abuse during HIV-1 Infection and Central Nervous System DiseaseCortical consequences of HIV-1 Tat exposure in rats are enhanced by chronic cocaineExposure to HIV-1 Tat in brain impairs sensorimotor gating and activates microglia in limbic and extralimbic brain regions of male mice.Anxiety-like behavior of mice produced by conditional central expression of the HIV-1 regulatory protein, Tat.Conditional Tat protein brain expression in the GT-tg bigenic mouse induces cerebral fractional anisotropy abnormalities.Repeated cocaine treatment enhances HIV-1 Tat-induced cortical excitability via over-activation of L-type calcium channels.Progesterone protects normative anxiety-like responding among ovariectomized female mice that conditionally express the HIV-1 regulatory protein, Tat, in the CNS.Allosteric modulatory effects of SRI-20041 and SRI-30827 on cocaine and HIV-1 Tat protein binding to human dopamine transporter.Mutations at tyrosine 88, lysine 92 and tyrosine 470 of human dopamine transporter result in an attenuation of HIV-1 Tat-induced inhibition of dopamine transport.Sensitization of enteric neurons to morphine by HIV-1 Tat protein.Molecular mechanism of HIV-1 Tat interacting with human dopamine transporter.Didehydro-cortistatin A inhibits HIV-1 Tat mediated neuroinflammation and prevents potentiation of cocaine reward in Tat transgenic miceAbnormal context-reward associations in an immune-mediated neurodevelopmental mouse model with relevance to schizophrenia.HIV Tat Impairs Neurogenesis through Functioning As a Notch Ligand and Activation of Notch Signaling Pathway.Molecular mechanism: the human dopamine transporter histidine 547 regulates basal and HIV-1 Tat protein-inhibited dopamine transportHIV, Tat and dopamine transmission.HIV-1 TAT protein enhances sensitization to methamphetamine by affecting dopaminergic function.The effects of HIV-1 regulatory TAT protein expression on brain reward function, response to psychostimulants and delay-dependent memory in mice.The role of human dopamine transporter in NeuroAIDS.Central HIV-1 Tat exposure elevates anxiety and fear conditioned responses of male mice concurrent with altered mu-opioid receptor-mediated G-protein activation and β-arrestin 2 activity in the forebrain.CCR5 mediates HIV-1 Tat-induced neuroinflammation and influences morphine tolerance, dependence, and reward.HIV-1 and cocaine disrupt dopamine reuptake and medium spiny neurons in female rat striatum.Colon dysregulation in methamphetamine self-administering HIV-1 transgenic rats.Network Analysis of Hippocampal Neurons by Microelectrode Array in the Presence of HIV-1 Tat and Cocaine.Doxycycline-inducible and astrocyte-specific HIV-1 Tat transgenic mice (iTat) as an HIV/neuroAIDS model.HIV-1 proteins dysregulate motivational processes and dopamine circuitry.
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Effects of conditional central expression of HIV-1 tat protein to potentiate cocaine-mediated psychostimulation and reward among male mice.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 15 August 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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Effects of conditional central ...... on and reward among male mice.
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Effects of conditional central ...... on and reward among male mice.
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Effects of conditional central ...... on and reward among male mice.
@en
Effects of conditional central ...... on and reward among male mice.
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Effects of conditional central ...... on and reward among male mice.
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Effects of conditional central ...... on and reward among male mice.
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P2093
P2860
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Effects of conditional central ...... on and reward among male mice.
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Amanda N Carey
Christopher F Shay
Jason J Paris
Jay P McLaughlin
Johnny J He
Stacey M Gomes
P2860
P2888
P304
P356
10.1038/NPP.2013.201
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P577
2013-08-15T00:00:00Z