The roles of chemokines in rabies virus infection: overexpression may not always be beneficial.
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Ambivalent role of the innate immune response in rabies virus pathogenesisIntracerebral administration of recombinant rabies virus expressing GM-CSF prevents the development of rabies after infection with street virus.Expression of MIP-1alpha (CCL3) by a recombinant rabies virus enhances its immunogenicity by inducing innate immunity and recruiting dendritic cells and B cells.Gene order rearrangement of the M gene in the rabies virus leads to slower replication.Rabies virus expressing dendritic cell-activating molecules enhances the innate and adaptive immune response to vaccination.Met-CCL5 represents an immunotherapy strategy to ameliorate rabies virus infection.The inability of wild-type rabies virus to activate dendritic cells is dependent on the glycoprotein and correlates with its low level of the de novo-synthesized leader RNA.HIV-1 coinfection and morphine coexposure severely dysregulate hepatitis C virus-induced hepatic proinflammatory cytokine release and free radical production: increased pathogenesis coincides with uncoordinated host defenses.Viral Infection of the Central Nervous System and Neuroinflammation Precede Blood-Brain Barrier Disruption during Japanese Encephalitis Virus Infection.Critical Role of K1685 and K1829 in the Large Protein of Rabies Virus in Viral Pathogenicity and Immune EvasionInfection with street strain rabies virus induces modulation of the microRNA profile of the mouse brain.Profile of Cytokines and Chemokines Triggered by Wild-Type Strains of Rabies Virus in MiceRabies virus glycoprotein is an important determinant for the induction of innate immune responses and the pathogenic mechanisms.Recombinant rabies virus expressing dog GM-CSF is an efficacious oral rabies vaccine for dogs.Genome-Wide Transcriptional Profiling Reveals Two Distinct Outcomes in Central Nervous System Infections of Rabies Virus.A Novel Rabies Vaccine Expressing CXCL13 Enhances Humoral Immunity by Recruiting both T Follicular Helper and Germinal Center B CellsPhenotypic Consequences In vivo and In vitro of Rearranging the P Gene of RABV HEP-Flury.Regional astrocyte IFN signaling restricts pathogenesis during neurotropic viral infectionEnhancement of blood-brain barrier permeability and reduction of tight junction protein expression are modulated by chemokines/cytokines induced by rabies virus infection.Experimental rabies vaccines for humans.Targeting CCL5 in inflammation.Viral sensing at the blood-brain barrier: new roles for innate immunity at the CNS vasculature.Reverse genetics of rabies virus: new strategies to attenuate virus virulence for vaccine development.Rabies - epidemiology, pathogenesis, public health concerns and advances in diagnosis and control: a comprehensive review.The type I interferon response bridles rabies virus infection and reduces pathogenicity.Using rabies virus vaccine strain SRV9 as viral vector to express exogenous gene.An optimized HMGB1 expressed by recombinant rabies virus enhances immunogenicity through activation of dendritic cells in mice.Lab-Attenuated Rabies Virus Causes Abortive Infection and Induces Cytokine Expression in Astrocytes by Activating Mitochondrial Antiviral-Signaling Protein Signaling Pathway.Reverse Genetics of Rhabdoviruses
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The roles of chemokines in rabies virus infection: overexpression may not always be beneficial.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 09 September 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
The roles of chemokines in rab ...... may not always be beneficial.
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The roles of chemokines in rab ...... may not always be beneficial.
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The roles of chemokines in rab ...... may not always be beneficial.
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The roles of chemokines in rab ...... may not always be beneficial.
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The roles of chemokines in rab ...... may not always be beneficial.
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The roles of chemokines in rab ...... may not always be beneficial.
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The roles of chemokines in rab ...... may not always be beneficial.
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Harufusa Toriumi
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11808-11818
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10.1128/JVI.01346-09
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2009-09-09T00:00:00Z