Bcl-xL is an oncogenic driver in colorectal cancer. - Wikidata - wikimedia - TriplyDB

Bcl-xL is an oncogenic driver in colorectal cancer.

Bcl-xL is an oncogenic driver in colorectal cancer.

http://www.wikidata.org/entity/Q37412492

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Enhancing venetoclax activity in acute myeloid leukemia by co-targeting MCL1.Interplay between apoptosis and autophagy in colorectal cancer.LOC285629 regulates cell proliferation and motility in colorectal cancer cells.Post-Transcriptional Regulation of Anti-Apoptotic BCL2 Family Members.

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Bcl-xL is an oncogenic driver in colorectal cancer.

http://www.wikidata.org/entity/Q37412492

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 18 August 2016

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Bcl-xL is an oncogenic driver in colorectal cancer.

@en

Bcl-xL is an oncogenic driver in colorectal cancer.

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type

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Bcl-xL is an oncogenic driver in colorectal cancer.

@en

Bcl-xL is an oncogenic driver in colorectal cancer.

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prefLabel

Bcl-xL is an oncogenic driver in colorectal cancer.

@en

Bcl-xL is an oncogenic driver in colorectal cancer.

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Cell Death and Disease

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Bcl-xL is an oncogenic driver in colorectal cancer.

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Achim Weber

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Adam Jassowicz

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Anette Heller

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Anna-Lena Scherr

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Bruno Christian Koehler

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Christin Elssner

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Dirk Jaeger

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Georg Gdynia

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Henning Schulze-Bergkamen

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Katarina Duglova

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P2860

Hallmarks of Cancer: The Next Generation

ABT-199, a potent and selective BCL-2 inhibitor, achieves antitumor activity while sparing platelets

Global cancer statistics, 2012

Regulation of apoptosis and cell cycle progression by MCL1. Differential role of proliferating cell nuclear antigen

An inhibitor of Bcl-2 family proteins induces regression of solid tumours

The Bcl2 family: regulators of the cellular life-or-death switch

TNF-α-induced intestinal epithelial cell shedding: implications for intestinal barrier function.

Mcl-1 overexpression in hepatocellular carcinoma: a potential target for antisense therapy.

MCL-1 localizes to sites of DNA damage and regulates DNA damage response

An inducible mouse model of colon carcinogenesis for the analysis of sporadic and inflammation-driven tumor progression.

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e2342

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scholarly article

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10.1038/CDDIS.2016.233

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8

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7

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Mathias Heikenwalder

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2016-08-18T00:00:00Z

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1003797796

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27537525

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colorectal cancer

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5108319

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