Dysfunctional expansion of hematopoietic stem cells and block of myeloid differentiation in lethal sepsis. - Wikidata - wikimedia - TriplyDB

Dysfunctional expansion of hematopoietic stem cells and block of myeloid differentiation in lethal sepsis.

Dysfunctional expansion of hematopoietic stem cells and block of myeloid differentiation in lethal sepsis.

http://www.wikidata.org/entity/Q37414700

about

Inflammatory modulation of HSCs: viewing the HSC as a foundation for the immune response Modulation of bleomycin-induced lung fibrosis by pegylated hyaluronidase and dopamine receptor antagonist in mice Stress and Non-Stress Roles of Inflammatory Signals during HSC Emergence and Maintenance The Role of Toll-Like Receptors in Hematopoietic Malignancies Systematic tracking of altered haematopoiesis during sporozoite-mediated malaria development reveals multiple response points Infection mobilizes hematopoietic stem cells through cooperative NOD-like receptor and Toll-like receptor signaling.Bactericidal/permeability-increasing protein (rBPI21) and fluoroquinolone mitigate radiation-induced bone marrow aplasia and death.Activated protein C targets CD8+ dendritic cells to reduce the mortality of endotoxemia in mice.Nice neighborhood: emerging concepts of the stem cell niche.Mammalian target of rapamycin activation underlies HSC defects in autoimmune disease and inflammation in mice.Prognostic significance of expression of a single microRNA, miR-181a, in cytogenetically normal acute myeloid leukemia: a Cancer and Leukemia Group B study.NLRP1 inflammasome activation induces pyroptosis of hematopoietic progenitor cells.The SKP2 E3 ligase regulates basal homeostasis and stress-induced regeneration of HSCs Heme-Mediated Induction of CXCL10 and Depletion of CD34+ Progenitor Cells Is Toll-Like Receptor 4 Dependent Excessive proliferation and impaired function of primitive hematopoietic cells in bone marrow due to senescence post chemotherapy in a T cell acute lymphoblastic leukemia model.Rosiglitazone Promotes Bone Marrow Adipogenesis to Impair Myelopoiesis under Stress.Early severe impairment of hematopoietic stem and progenitor cells from the bone marrow caused by CLP sepsis and endotoxemia in a humanized mice model Myeloid-derived suppressor cells evolve during sepsis and can enhance or attenuate the systemic inflammatory response.Odd couple: The unexpected partnership of glucocorticoid hormones and cysteinyl-leukotrienes in the extrinsic regulation of murine bone-marrow eosinopoiesis.Early sensing of Yersinia pestis airway infection by bone marrow cells.Replenishing B lymphocytes in health and disease.The function of hematopoietic stem cells is altered by both genetic and inflammatory factors in lupus mice.Norepinephrine inhibits macrophage migration by decreasing CCR2 expression.Mobilisation and dysfunction of haematopoietic stem/progenitor cells after Listonella anguillarum infection in ayu, Plecoglossus altivelis.Pten Cell Autonomously Modulates the Hematopoietic Stem Cell Response to Inflammatory Cytokines.Sepsis Induces Hematopoietic Stem Cell Exhaustion and Myelosuppression through Distinct Contributions of TRIF and MYD88.Perturbed mononuclear phagocyte system in severely burned and septic patients.High bone mass in mice lacking Cx37 because of defective osteoclast differentiation.Inflammatory signals regulate hematopoietic stem cells Hematopoiesis sculpted by pathogens: Toll-like receptors and inflammatory mediators directly activate stem cells.Regulation of hematopoietic stem cell activity by inflammation.From proliferation to proliferation: monocyte lineage comes full circle.Systemic TLR2 agonist exposure regulates hematopoietic stem cells via cell-autonomous and cell-non-autonomous mechanisms.Inflammation: a key regulator of hematopoietic stem cell fate in health and disease.Single Cell Phenotyping Reveals Heterogeneity among Haematopoietic Stem Cells Following Infection.Sepsis-Induced Thymic Atrophy Is Associated with Defects in Early Lymphopoiesis.B cells modulate systemic responses to Pneumocystis murina lung infection and protect on-demand hematopoiesis via T cell-independent innate mechanisms when type I interferon signaling is absent.Differential expression of the transcription factor ARID3a in lupus patient hematopoietic progenitor cells.Acquired deficiency of A20 results in rapid apoptosis, systemic inflammation, and abnormal hematopoietic stem cell function.In vivo time-lapse imaging shows diverse niche engagement by quiescent and naturally activated hematopoietic stem cells.

P2860

Q27003372-B9AF85B8-C079-4C9F-8558-272336945666 Q27308781-5C96C0C3-EEA2-4E8E-B47C-D424FE3D7748 Q28072920-16177861-4175-4415-A914-A22A40BDD25C Q28077740-DC0AF8BD-B3FE-47D5-A7A3-B88BB6980A5B Q28596876-4A4C72F7-84CB-4942-8F50-2BB801ED77E7 Q33857117-8B43D696-84EA-4E4C-B12B-2636907BFDAE Q33896849-DB36CBCB-481B-4DC4-B84D-E8FB1F64BB2C Q34090836-F7B1CD5D-8B55-4649-840A-F182748A14C4 Q34165273-4ED0347B-5C16-4596-99B4-DC6A3C641444 Q34245461-C9DC211F-D3B2-41C5-9E51-67ADDE53089A Q34477821-A1FE07EE-C48E-472B-BEED-231FEA8A068C Q34764912-33091853-331B-4F54-A803-DFF40593C3EB Q35067612-027F7406-7515-4407-BD6D-D82019F2D741 Q35837528-08EC85D9-7826-40D7-B151-FDCBDC0BDDC7 Q35857507-7054703F-59FD-45E9-ABB1-B24CFEB07E69 Q35928798-25B764D8-72C6-4EAC-BE62-5CB396D2A3D0 Q35954071-2BAA3815-749F-4DC3-9844-EAD69BE31816 Q36018144-4F8B107F-1296-479D-85B7-9A3A97C886FB Q36297766-4969498A-69AD-4A66-A995-4F3768439408 Q36421830-25C70913-7F72-439B-8FA3-25D72A7CE13D Q36605634-50D2D4EE-EAEB-42E4-ACE8-7C32BB5D08EE Q36682837-F5BC44E3-0603-43DA-896D-1F2F7BE760C3 Q36976763-EA155ED0-D2AB-4B32-B4C5-56C6D13AD36B Q37010607-AB2C8615-5C4E-494F-9D6D-960F6EBF1DCA Q37014691-4C6D60B2-B453-43B8-8110-0883B26375D6 Q37014715-33AD1988-1C13-4C05-B6D4-DDE6F0C30713 Q37074014-0F8BB0D4-DB47-46DC-835E-BC3A832622E8 Q37653316-71755EFA-977D-4BE2-812A-87BEF8BECF4E Q37828295-D427C7CB-3B5E-4B12-B983-39363E362F07 Q37955518-86604D93-F55F-4DF9-A734-25D9DB68AD0C Q38123812-312595A8-A60B-4690-8B0B-DB48985CFC37 Q38179249-E5F00796-B573-4D7A-A0CD-9423040A57D5 Q39675197-35A57CF0-0EC0-43A9-A4AC-3C58AF704C77 Q40066012-AB7FB869-93B9-4234-B470-9E58CFE75808 Q40082656-390AF7CE-8D54-48FC-AB28-6DB45F6821BB Q40611797-F21EFD85-AD22-4456-BDB5-47E23E889AA7 Q41632240-AE9B69A6-4036-4CD7-9BDB-3F4658C2D43B Q41656076-34E2474F-96F9-4AA9-8A6B-B5077519FF83 Q41904331-AF1796A9-FB67-4389-870C-DCC6066E6BBE Q42126871-BB98A009-8936-45E6-9323-E009A04FBFC1

P2860

Dysfunctional expansion of hematopoietic stem cells and block of myeloid differentiation in lethal sepsis.

http://www.wikidata.org/entity/Q37414700

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 20 August 2009

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Dysfunctional expansion of hem ...... ferentiation in lethal sepsis.

@en

Dysfunctional expansion of hem ...... ferentiation in lethal sepsis.

@nl

type

label

Dysfunctional expansion of hem ...... ferentiation in lethal sepsis.

@en

Dysfunctional expansion of hem ...... ferentiation in lethal sepsis.

@nl

prefLabel

Dysfunctional expansion of hem ...... ferentiation in lethal sepsis.

@en

Dysfunctional expansion of hem ...... ferentiation in lethal sepsis.

@nl

P1433

Q37414700-84978EEF-0FAB-441A-BC31-262649F244BB

P1476

Q37414700-1E03014B-88B8-4F44-9DE9-B7D2A859E48E

P2093

Q37414700-081929E6-9852-4CF5-B4C5-8ED04E8FC68F

Q37414700-2E8B8E0A-12BC-43B8-9FC6-C40B4AE0C79D

Q37414700-2EB0514E-98B3-4893-9F3C-A070E8E3F164

Q37414700-43B6F1C7-0652-4DCA-A405-C91C6B07B5DA

Q37414700-4D7E7610-3858-4BA3-90A9-DED1489F2209

Q37414700-50C62D09-B72E-490E-B572-B55137B45963

Q37414700-78A938B7-2CD5-42D2-AEE0-953A6D42C431

Q37414700-8000C6CA-3422-41DA-B1D4-72522C5F03A3

Q37414700-A1AF4C85-33A8-4F00-95A3-B02E17B05FA9

Q37414700-B719E029-D876-4435-9C4F-8CD7979C7E76

P2860

Q37414700-0C910190-263B-467F-9CA1-5B541B7A80F7

Q37414700-12B846E8-E2D6-4457-AB7A-96D88FAB0589

Q37414700-13C2D8A2-159A-44CA-9CED-C341C6FC6C0C

Q37414700-15C2E5F3-0C61-4088-9DDD-F1C1EA7F8B0F

Q37414700-1F23C82F-C957-49D0-BF8F-44D5FF6BE419

Q37414700-39DF091D-D220-4803-98AD-22AC174DA66B

Q37414700-4461DCA6-39B5-48E2-BF06-24154CB4B363

Q37414700-4C1C0148-DAE4-451C-9D8E-7D935D760BD0

Q37414700-5252CEF4-1B88-4921-B730-354D1793A43E

Q37414700-52F88E69-0C34-4B19-B935-505F80CE4A53

P304

Q37414700-51D406C5-A02A-43DC-845E-5D0B4740D59F

P31

Q37414700-620A03B5-9D0B-4809-8078-8BFA28B230D4

P356

Q37414700-5411FA82-525E-4FCA-90C0-E1DC49E8B404

P407

Q37414700-3232A4E0-1AD7-443D-A4C6-17895B395780

P433

Q37414700-D545E31B-4D69-4518-8372-AAFD26E224EC

P478

Q37414700-C5E3DF53-63A7-4042-8697-46854FC6AAFA

P577

Q37414700-1858B290-D695-44C7-AAFE-4241088EDE4C

P5875

Q37414700-F3066935-4591-493A-BADE-5244F9F11E99

P698

Q37414700-DAD9243A-4B54-457C-A26C-50B9347532D3

P921

Q37414700-3426A530-AD8B-4BF9-B822-EBF54D1EB0CD

P932

Q37414700-B3A1FF79-5B53-4357-82B3-4A13BF03D325

P356

BLOOD-2009-04-214916

P1433

P1476

Dysfunctional expansion of hem ...... ferentiation in lethal sepsis.

@en

P2093

Angelo Chora

http://www.w3.org/2001/XMLSchema#string

Boyan Goumnerov

http://www.w3.org/2001/XMLSchema#string

Carol A Karlewicz

http://www.w3.org/2001/XMLSchema#string

Christen Mumaw

http://www.w3.org/2001/XMLSchema#string

David M Dombkowski

http://www.w3.org/2001/XMLSchema#string

Harm HogenEsch

http://www.w3.org/2001/XMLSchema#string

Hasan Baydoun

http://www.w3.org/2001/XMLSchema#string

Laurence G Rahme

http://www.w3.org/2001/XMLSchema#string

Luis Fernandez

http://www.w3.org/2001/XMLSchema#string

Nadia Carlesso

http://www.w3.org/2001/XMLSchema#string

P2860

Myeloid commitment shifts toward monocytopoiesis after thermal injury and sepsis

Use of model plant hosts to identify Pseudomonas aeruginosa virulence factors

The stem-cell niche as an entity of action

Common virulence factors for bacterial pathogenicity in plants and animals

Getting to the site of inflammation: the leukocyte adhesion cascade updated

A clonogenic common myeloid progenitor that gives rise to all myeloid lineages

Neutrophils and immunity: challenges and opportunities

The pathophysiology and treatment of sepsis

p53 regulates hematopoietic stem cell quiescence.

Essential role of gamma interferon in survival of colon ascendens stent peritonitis, a novel murine model of abdominal sepsis.

P304

4064-4076

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1182/BLOOD-2009-04-214916

http://www.w3.org/2001/XMLSchema#string

P407

P433

19

http://www.w3.org/2001/XMLSchema#string

P478

114

http://www.w3.org/2001/XMLSchema#string

P577

2009-08-20T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

26756674

http://www.w3.org/2001/XMLSchema#string

P698

19696201

http://www.w3.org/2001/XMLSchema#string

P921

P932

2774548

http://www.w3.org/2001/XMLSchema#string