A new player in a deadly game: influenza viruses and the PI3K/Akt signalling pathway.
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Intracellular events and cell fate in filovirus infectionRotavirus nonstructural protein 1 suppresses virus-induced cellular apoptosis to facilitate viral growth by activating the cell survival pathways during early stages of infectionMacrophage Expression of Inflammatory Genes in Response to EMCV InfectionMicroRNAs in the Host Response to Viral Infections of Veterinary ImportanceMolecular mechanism behind rotavirus NSP1-mediated PI3 kinase activation: interaction between NSP1 and the p85 subunit of PI3 kinasePotent host-directed small-molecule inhibitors of myxovirus RNA-dependent RNA-polymerasesOncolytic effects of a novel influenza A virus expressing interleukin-15 from the NS reading frameThe human adenovirus E4-ORF1 protein subverts discs large 1 to mediate membrane recruitment and dysregulation of phosphatidylinositol 3-kinaseCharacterization of a novel mutation in NS1 protein of influenza A virus induced by a chemical substance for the attenuation of pathogenicityChemical Genomics Identifies the PERK-Mediated Unfolded Protein Stress Response as a Cellular Target for Influenza Virus InhibitionAvian influenza viruses inhibit the major cellular signalling integrator c-Abl.Heterologous SH3-p85beta inhibits influenza A virus replication.The Rac1 inhibitor NSC23766 exerts anti-influenza virus properties by affecting the viral polymerase complex activity.Networks of Host Factors that Interact with NS1 Protein of Influenza A VirusHighly pathogenic avian influenza H5N1 virus delays apoptotic responses via activation of STAT3.Innate immune response to influenza A virus in differentiated human alveolar type II cellsThe influenza virus NS1 protein as a therapeutic target.Surveillance in Eastern India (2007-2009) revealed reassortment event involving NS and PB1-F2 gene segments among co-circulating influenza A subtypes.Serum-free microcarrier based production of replication deficient influenza vaccine candidate virus lacking NS1 using Vero cells.Meta- and Orthogonal Integration of Influenza "OMICs" Data Defines a Role for UBR4 in Virus Budding.Role of AKT kinase in measles virus replication.Transient activation of the PI3K/Akt pathway promotes Newcastle disease virus replication and enhances anti-apoptotic signaling responsesThe Ras-PI3K signaling pathway is involved in clathrin-independent endocytosis and the internalization of influenza viruses.Cellular transcriptional profiling in human lung epithelial cells infected by different subtypes of influenza A viruses reveals an overall down-regulation of the host p53 pathway.Identification of BPR3P0128 as an inhibitor of cap-snatching activities of influenza virus.Activation of c-jun N-terminal kinase upon influenza A virus (IAV) infection is independent of pathogen-related receptors but dependent on amino acid sequence variations of IAV NS1.The PDZ-ligand and Src-homology type 3 domains of epidemic avian influenza virus NS1 protein modulate human Src kinase activity during viral infection.Antiviral potential of ERK/MAPK and PI3K/AKT/mTOR signaling modulation for Middle East respiratory syndrome coronavirus infection as identified by temporal kinome analysisToxoplasma gondii Proliferation Require Down-Regulation of Host Nox4 Expression via Activation of PI3 Kinase/Akt Signaling Pathway.Parasite-mediated upregulation of NK cell-derived gamma interferon protects against severe highly pathogenic H5N1 influenza virus infectionCellular networks involved in the influenza virus life cycle.Cell death regulation during influenza A virus infection by matrix (M1) protein: a model of viral control over the cellular survival pathwayA novel systems pharmacology model for herbal medicine injection: a case using Reduning injection.Antitumor action of the peroxisome proliferator-activated receptor-γ agonist rosiglitazone in hepatocellular carcinomaInability of NS1 protein from an H5N1 influenza virus to activate PI3K/Akt signaling pathway correlates to the enhanced virus replication upon PI3K inhibition.Influenza A viruses control expression of proviral human p53 isoforms p53β and Delta133p53α.Activation of the phosphatidylinositol 3-kinase/Akt signaling pathway during porcine circovirus type 2 infection facilitates cell survival and viral replicationSynergistic effect of the PDZ and p85β-binding domains of the NS1 protein on virulence of an avian H5N1 influenza A virus.Sprouty-Related Ena/Vasodilator-Stimulated Phosphoprotein Homology 1-Domain-Containing Protein-2 Critically Regulates Influenza A Virus-Induced PneumoniaKnockdown of specific host factors protects against influenza virus-induced cell death.
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A new player in a deadly game: influenza viruses and the PI3K/Akt signalling pathway.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 12 March 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
A new player in a deadly game: influenza viruses and the PI3K/Akt signalling pathway.
@en
A new player in a deadly game: influenza viruses and the PI3K/Akt signalling pathway.
@nl
type
label
A new player in a deadly game: influenza viruses and the PI3K/Akt signalling pathway.
@en
A new player in a deadly game: influenza viruses and the PI3K/Akt signalling pathway.
@nl
prefLabel
A new player in a deadly game: influenza viruses and the PI3K/Akt signalling pathway.
@en
A new player in a deadly game: influenza viruses and the PI3K/Akt signalling pathway.
@nl
P2860
P1476
A new player in a deadly game: influenza viruses and the PI3K/Akt signalling pathway.
@en
P2093
Christina Ehrhardt
P2860
P304
P356
10.1111/J.1462-5822.2009.01309.X
P577
2009-03-12T00:00:00Z