A common functional promoter variant links CNR1 gene expression to HDL cholesterol level. - Wikidata - wikimedia - TriplyDB

A common functional promoter variant links CNR1 gene expression to HDL cholesterol level.

A common functional promoter variant links CNR1 gene expression to HDL cholesterol level.

http://www.wikidata.org/entity/Q37416464

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Massively parallel quantification of the regulatory effects of noncoding genetic variation in a human cohort.Genomic approaches for understanding the genetics of complex disease.CB1 and CB2 Receptor Pharmacology.The Endocannabinoid System and Heart Disease: The Role of Cannabinoid Receptor Type 2.

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P2860

A common functional promoter variant links CNR1 gene expression to HDL cholesterol level.

http://www.wikidata.org/entity/Q37416464

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

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bilimsel makale

@tr

scientific article published on January 2013

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

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vědecký článek

@cs

name

A common functional promoter variant links CNR1 gene expression to HDL cholesterol level.

@en

A common functional promoter variant links CNR1 gene expression to HDL cholesterol level.

@nl

type

label

A common functional promoter variant links CNR1 gene expression to HDL cholesterol level.

@en

A common functional promoter variant links CNR1 gene expression to HDL cholesterol level.

@nl

prefLabel

A common functional promoter variant links CNR1 gene expression to HDL cholesterol level.

@en

A common functional promoter variant links CNR1 gene expression to HDL cholesterol level.

@nl

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Nature Communications

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A common functional promoter variant links CNR1 gene expression to HDL cholesterol level.

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D G Harrison

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K C Vickers

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M G Levin

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M P Anderson

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Q Feng

http://www.w3.org/2001/XMLSchema#string

R A Wilke

http://www.w3.org/2001/XMLSchema#string

W Chen

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Endothelial lipase concentrations are increased in metabolic syndrome and associated with coronary atherosclerosis.

The thrifty lipids: endocannabinoids and the neural control of energy conservation

The cannabinoid CB1 receptor antagonist SR141716 increases Acrp30 mRNA expression in adipose tissue of obese fa/fa rats and in cultured adipocyte cells

MicroRNAs are transported in plasma and delivered to recipient cells by high-density lipoproteins

Knowledge-driven multi-locus analysis reveals gene-gene interactions influencing HDL cholesterol level in two independent EMR-linked biobanks.

Construction of atorvastatin dose-response relationships using data from a large population-based DNA biobank.

Human S-adenosylhomocysteine hydrolase: common gene sequence variation and functional genomic characterization.

Robust replication of genotype-phenotype associations across multiple diseases in an electronic medical record.

A common CNR1 (cannabinoid receptor 1) haplotype attenuates the decrease in HDL cholesterol that typically accompanies weight gain.

PheWAS: demonstrating the feasibility of a phenome-wide scan to discover gene-disease associations.

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1973

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10.1038/NCOMMS2973

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2013-01-01T00:00:00Z

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