Severe preeclampsia is characterized by increased placental expression of galectin-1 - Wikidata - wikimedia - TriplyDB

Severe preeclampsia is characterized by increased placental expression of galectin-1

Severe preeclampsia is characterized by increased placental expression of galectin-1

http://www.wikidata.org/entity/Q37416956

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A primate subfamily of galectins expressed at the maternal-fetal interface that promote immune cell death Galectins: guardians of eutherian pregnancy at the maternal-fetal interface Placental Protein 13 (PP13) - A Placental Immunoregulatory Galectin Protecting Pregnancy Galectins: Double-edged Swords in the Cross-roads of Pregnancy Complications and Female Reproductive Tract Inflammation and Neoplasia Trichomoniasis: evaluation to execution Human infectious diseases and risk of preeclampsia: an updated review of the literature.Galectin-1 overexpression in endometriosis and its regulation by neuropeptides (CRH, UCN) indicating its important role in reproduction and inflammation Microbial invasion of the amniotic cavity in preeclampsia as assessed by cultivation and sequence-based methods Late-onset preeclampsia is associated with an imbalance of angiogenic and anti-angiogenic factors in patients with and without placental lesions consistent with maternal underperfusion.Maternal and neonatal circulating visfatin concentrations in patients with pre-eclampsia and a small-for-gestational age neonate.Placental protein 13 (PP13/galectin-13) undergoes lipid raft-associated subcellular redistribution in the syncytiotrophoblast in preterm preeclampsia and HELLP syndrome.Could alterations in maternal plasma visfatin concentration participate in the phenotype definition of preeclampsia and SGA?Interleukin-33 in the human placenta Maternal serum adiponectin multimers in patients with a small-for-gestational-age newborn.Emergence of hormonal and redox regulation of galectin-1 in placental mammals: implication in maternal-fetal immune tolerance Prototype and Chimera-Type Galectins in Placentas with Spontaneous and Recurrent Miscarriages.Interfering with Gal-1-mediated angiogenesis contributes to the pathogenesis of preeclampsia.Impact of T. vaginalis infection on innate immune responses and reproductive outcome.Tolerance induction at the early maternal-placental interface through selective cell recruitment and targeting by immune polypeptides.'Sweetening' pregnancy: galectins at the fetomaternal interface.A potential pathophysiological role for galectins and the renin-angiotensin system in preeclampsia.Resolution of inflammation pathways in preeclampsia-a narrative review.Immunological Tolerance, Pregnancy, and Preeclampsia: The Roles of Semen Microbes and the Father.Galectin-1 as a novel risk factor for both gestational hypertension and preeclampsia, specifially its expression at a low level in the second trimester and a high level after onset.The Role of Galectins as Modulators of Metabolism and Inflammation.

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P2860

Severe preeclampsia is characterized by increased placental expression of galectin-1

http://www.wikidata.org/entity/Q37416956

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

@tr

scientific article published on July 2008

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Severe preeclampsia is characterized by increased placental expression of galectin-1

@en

Severe preeclampsia is characterized by increased placental expression of galectin-1.

@nl

type

label

Severe preeclampsia is characterized by increased placental expression of galectin-1

@en

Severe preeclampsia is characterized by increased placental expression of galectin-1.

@nl

prefLabel

Severe preeclampsia is characterized by increased placental expression of galectin-1

@en

Severe preeclampsia is characterized by increased placental expression of galectin-1.

@nl

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Journal of Maternal-Fetal and Neonatal Medicine

P1476

Severe preeclampsia is characterized by increased placental expression of galectin-1

@en

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Adi L Tarca

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Asad Abbas

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Derek E Wildman

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Francesca Gotsch

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Jimmy Espinoza

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John Hotra

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Juan Pedro Kusanovic

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Nandor Gabor Than

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Offer Erez

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Roberto Romero

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P2860

Prevention of allogeneic fetal rejection by tryptophan catabolism

Preeclampsia: a view through the danger model

The placental problem: linking abnormal cytotrophoblast differentiation to the maternal symptoms of preeclampsia

Crystal structure of human Charcot-Leyden crystal protein, an eosinophil lysophospholipase, identifies it as a new member of the carbohydrate-binding family of galectins

TRAIL (Apo-2L) and TRAIL receptors in human placentas: implications for immune privilege

The danger model: a renewed sense of self

Isolation and characterization of Hofbauer cells from human placental villi

Soluble endoglin contributes to the pathogenesis of preeclampsia

Mother's little helpers: mechanisms of maternal-fetal tolerance

Inflammation-induced modulation of cellular galectin-1 and -3 expression in a model of rat peritonitis

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429-442

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scholarly article

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10.1080/14767050802041961

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7

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