Inhibition of histone deacetylase activity attenuates renal fibroblast activation and interstitial fibrosis in obstructive nephropathy. - Wikidata - wikimedia - TriplyDB

Inhibition of histone deacetylase activity attenuates renal fibroblast activation and interstitial fibrosis in obstructive nephropathy.

Inhibition of histone deacetylase activity attenuates renal fibroblast activation and interstitial fibrosis in obstructive nephropathy.

http://www.wikidata.org/entity/Q37417291

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Evidence for the involvement of epigenetics in the progression of renal fibrogenesis Histone deacetylases as targets for treatment of multiple diseases Epigenomics: the science of no-longer-junk DNA. Why study it in chronic kidney disease?Therapeutic efficacy of valproic acid in a combined monocrotaline and chronic hypoxia rat model of severe pulmonary hypertension Suramin alleviates glomerular injury and inflammation in the remnant kidney Inhibition of histone deacetylase impacts cancer stem cells and induces epithelial-mesenchyme transition of head and neck cancer Mefunidone attenuates tubulointerstitial fibrosis in a rat model of unilateral ureteral obstruction Piceatannol Attenuates Renal Fibrosis Induced by Unilateral Ureteral Obstruction via Downregulation of Histone Deacetylase 4/5 or p38-MAPK Signaling Histone deacetylase 1/2 mediates proliferation of renal interstitial fibroblasts and expression of cell cycle proteins Sphingosine Kinase 2 Deficiency Attenuates Kidney Fibrosis via IFN-γ.Systems Biology in Kidney Transplantation: The Application of Multi-Omics to a Complex Model.All-trans retinoic acid and sodium butyrate enhance natriuretic peptide receptor a gene transcription: role of histone modification A DDX5 S480A polymorphism is associated with increased transcription of fibrogenic genes in hepatic stellate cells Evaluation of Caspase 3 Enzyme and TNF-alpha as Biomarkers in Ureteropelvic Junction Obstruction in Children- a preliminary report.Recent Advances of Curcumin in the Prevention and Treatment of Renal Fibrosis.Pharmacological inhibition of Src kinase protects against acute kidney injury in a murine model of renal ischemia/reperfusion.Class-specific histone/protein deacetylase inhibition protects against renal ischemia reperfusion injury and fibrosis formation.The attenuation of renal fibrosis by histone deacetylase inhibitors is associated with the plasticity of FOXP3+IL-17+ T cells Blocking sirtuin 1 and 2 inhibits renal interstitial fibroblast activation and attenuates renal interstitial fibrosis in obstructive nephropathy.Class I HDAC activity is required for renal protection and regeneration after acute kidney injury Inhibition of STAT3 acetylation is associated with angiotesin renal fibrosis in the obstructed kidney.P300-dependent STAT3 acetylation is necessary for angiotensin II-induced pro-fibrotic responses in renal tubular epithelial cells.Histone deacetylase: a potential therapeutic target for fibrotic disorders Blocking the class I histone deacetylase ameliorates renal fibrosis and inhibits renal fibroblast activation via modulating TGF-beta and EGFR signaling Long-term administration of the histone deacetylase inhibitor vorinostat attenuates renal injury in experimental diabetes through an endothelial nitric oxide synthase-dependent mechanism Suramin inhibits renal fibrosis in chronic kidney disease.STAT1 signaling is not regulated by a phosphorylation-acetylation switch Delayed administration of suramin attenuates the progression of renal fibrosis in obstructive nephropathy Treatment of chronic kidney diseases with histone deacetylase inhibitors.Anti-Fibrotic Effects of Class I HDAC Inhibitor, Mocetinostat Is Associated with IL-6/Stat3 Signaling in Ischemic Heart Failure.Valproic acid attenuates proteinuria and kidney injury.Effect of erythropoietin on the expression of dynamin-related protein-1 in rat renal interstitial fibrosis.Qualitative and Quantitative Analysis of Histone Deacetylases in Kidney Tissue Sections.Activation of Sirtuin-1 Promotes Renal Fibroblast Activation and Aggravates Renal Fibrogenesis Genetic or pharmacologic blockade of EGFR inhibits renal fibrosis.Vorinostat protects against calcium oxalate-induced kidney injury in mice.Regulation of STAT signaling by acetylation.ITF2357 transactivates Id3 and regulate TGFβ/BMP7 signaling pathways to attenuate corneal fibrosis.Histone deacetylase inhibitors attenuate P-aIgA1-induced cell proliferation and extracellular matrix synthesis in human renal mesangial cells in vitro.HDAC inhibitors in experimental liver and kidney fibrosis.

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Inhibition of histone deacetylase activity attenuates renal fibroblast activation and interstitial fibrosis in obstructive nephropathy.

http://www.wikidata.org/entity/Q37417291

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 29 July 2009

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Inhibition of histone deacetyl ...... is in obstructive nephropathy.

@en

Inhibition of histone deacetyl ...... is in obstructive nephropathy.

@nl

type

label

Inhibition of histone deacetyl ...... is in obstructive nephropathy.

@en

Inhibition of histone deacetyl ...... is in obstructive nephropathy.

@nl

prefLabel

Inhibition of histone deacetyl ...... is in obstructive nephropathy.

@en

Inhibition of histone deacetyl ...... is in obstructive nephropathy.

@nl

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AJPRENAL.00282.2009

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American Journal of Physiology - Renal Physiology

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Inhibition of histone deacetyl ...... sis in obstructive nephropathy

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P2093

Evelyn Tolbert

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Haiping Mao

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Jagan Kothapally

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Maoyin Pang

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Shougang Zhuang

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Y Eugene Chin

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P2860

Acetylation-dependent signal transduction for type I interferon receptor

Histone deacetylases (HDACs): characterization of the classical HDAC family

Lysine acetylation: codified crosstalk with other posttranslational modifications

Cellular and molecular mechanisms of fibrosis

Trichostatin A, a histone deacetylase inhibitor, suppresses collagen synthesis and prevents TGF-beta(1)-induced fibrogenesis in skin fibroblasts

Phase 1 study of the histone deacetylase inhibitor vorinostat (suberoylanilide hydroxamic acid [SAHA]) in patients with advanced leukemias and myelodysplastic syndromes

Evidence that fibroblasts derive from epithelium during tissue fibrosis

Morphometry of interstitial fibrosis.

Sir2-dependent activation of acetyl-CoA synthetase by deacetylation of active lysine.

Histological, immunohistochemical and biological data in assessing interstitial fibrosis in patients with chronic glomerulonephritis.

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F996-F1005

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10.1152/AJPRENAL.00282.2009

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Murugavel Ponnusamy

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