Circulating monocytes in HIV-1-infected viremic subjects exhibit an antiapoptosis gene signature and virus- and host-mediated apoptosis resistance. - Wikidata - wikimedia - TriplyDB

Circulating monocytes in HIV-1-infected viremic subjects exhibit an antiapoptosis gene signature and virus- and host-mediated apoptosis resistance.

Circulating monocytes in HIV-1-infected viremic subjects exhibit an antiapoptosis gene signature and virus- and host-mediated apoptosis resistance.

http://www.wikidata.org/entity/Q37418469

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Drug-induced reactivation of apoptosis abrogates HIV-1 infection Asymmetric HIV-1 co-receptor use and replication in CD4(+) T lymphocytes Long-term programming of antigen-specific immunity from gene expression signatures in the PBMC of rhesus macaques immunized with an SIV DNA vaccine The Role of Caveolin 1 in HIV Infection and Pathogenesis.Genome-wide mRNA expression correlates of viral control in CD4+ T-cells from HIV-1-infected individuals Host genetics and HIV-1: the final phase?HIV-1 neuroimmunity in the era of antiretroviral therapy.Proteomic analysis of HIV-infected macrophages Increased monocyte turnover from bone marrow correlates with severity of SIV encephalitis and CD163 levels in plasma.Molecular mechanisms of HIV-1 persistence in the monocyte-macrophage lineage Microbial translocation induces persistent macrophage activation unrelated to HIV-1 levels or T-cell activation following therapy.HIV/SIV infection primes monocytes and dendritic cells for apoptosis.HIV-1 host interactions: integration of large-scale datasets Constitutive gene expression in monocytes from chronic HIV-1 infection overlaps with acute Toll-like receptor induced monocyte activation profiles.The CD14(+/low)CD16(+) monocyte subset is more susceptible to spontaneous and oxidant-induced apoptosis than the CD14(+)CD16(-) subset.Induction of SerpinB2 and Th1/Th2 modulation by SerpinB2 during lentiviral infections in vivo Shift in monocyte apoptosis with increasing viral load and change in apoptosis-related ISG/Bcl2 family gene expression in chronically HIV-1-infected subjects.JAM-A and ALCAM are therapeutic targets to inhibit diapedesis across the BBB of CD14+CD16+ monocytes in HIV-infected individuals.Analysis of HIV-1 expression level and sense of transcription by high-throughput sequencing of the infected cell Monocytes mediate HIV neuropathogenesis: mechanisms that contribute to HIV associated neurocognitive disorders Isolation of a TRAIL antagonist from the serum of HIV-infected patients.Replication-independent expression of anti-apoptosis marker genes in human peripheral blood mononuclear cells infected with the wild-type HIV-1 and reverse transcriptase variants Curcumin inhibits the replication of enterovirus 71 in vitro HIV-1 induces telomerase activity in monocyte-derived macrophages, possibly safeguarding one of its reservoirs.Human immunodeficiency virus (HIV) latency: the major hurdle in HIV eradication Caveolin-1 reduces HIV-1 infectivity by restoration of HIV Nef mediated impairment of cholesterol efflux by apoA-I.The α7-nicotinic receptor is upregulated in immune cells from HIV-seropositive women: consequences to the cholinergic anti-inflammatory response Antiretroviral therapy in HIV-1-infected individuals with CD4 count below 100 cells/mm3 results in differential recovery of monocyte activation.Retinoblastoma protein induction by HIV viremia or CCR5 in monocytes exposed to HIV-1 mediates protection from activation-induced apoptosis: ex vivo and in vitro study.Transcriptome analysis of primary monocytes from HIV-positive patients with differential responses to antiretroviral therapy.HIV-1 regulation of latency in the monocyte-macrophage lineage and in CD4+ T lymphocytes.Anti-apoptotic genes in the survival of monocytic cells during infection Evidence for the innate immune response as a correlate of protection in human immunodeficiency virus (HIV)-1 highly exposed seronegative subjects (HESN).Gene array studies in HIV-1 infection Anti-apoptotic mechanisms of HIV: lessons and novel approaches to curing HIV HIV-1 latency in monocytes/macrophages MicroRNA miR-126-5p Enhances the Inflammatory Responses of Monocytes to Lipopolysaccharide Stimulation by Suppressing Cylindromatosis in Chronic HIV-1 Infection.Transcriptome analysis of monocyte-HIV interactions.HIV gp120 in Lungs of ART-treated Individuals Impairs Alveolar Macrophage Responses To Pneumococci.Monocyte subsets exhibit transcriptional plasticity and a shared response to interferon in SIV-infected rhesus macaques.

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Circulating monocytes in HIV-1-infected viremic subjects exhibit an antiapoptosis gene signature and virus- and host-mediated apoptosis resistance.

http://www.wikidata.org/entity/Q37418469

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on April 2009

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Circulating monocytes in HIV-1 ...... mediated apoptosis resistance.

@en

Circulating monocytes in HIV-1 ...... mediated apoptosis resistance.

@nl

type

label

Circulating monocytes in HIV-1 ...... mediated apoptosis resistance.

@en

Circulating monocytes in HIV-1 ...... mediated apoptosis resistance.

@nl

prefLabel

Circulating monocytes in HIV-1 ...... mediated apoptosis resistance.

@en

Circulating monocytes in HIV-1 ...... mediated apoptosis resistance.

@nl

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JIMMUNOL.0801450

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Journal of Immunology

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Circulating monocytes in HIV-1 ...... mediated apoptosis resistance.

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Aidan Hancock

http://www.w3.org/2001/XMLSchema#string

Andrea Raymond

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Andrea S Foulkes

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Bethsebah Gekonge

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Calen Nicols

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Guido Silvestri

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Jane Shull

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Jay Kostman

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Karam Mounzer

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Louise Showe

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P2860

Microbial translocation is a cause of systemic immune activation in chronic HIV infection

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Molecular cloning and functional characterization of a novel human CC chemokine receptor (CCR5) for RANTES, MIP-1beta, and MIP-1alpha

HIV envelope induces a cascade of cell signals in non-proliferating target cells that favor virus replication

Statistical significance for genomewide studies

Human immunodeficiency virus type 1 Vpr protein binds to the uracil DNA glycosylase DNA repair enzyme

Cadmium induces caspase-independent apoptosis in liver Hep3B cells: role for calcium in signaling oxidative stress-related impairment of mitochondria and relocation of endonuclease G and apoptosis-inducing factor

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4459-4470

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scholarly article

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10.4049/JIMMUNOL.0801450

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7

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182

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2009-04-01T00:00:00Z

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24213615

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19299747

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apoptotic process

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2776064

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