CCAAT/enhancer-binding proteins and the pathogenesis of retrovirus infection. - Wikidata - wikimedia - TriplyDB

CCAAT/enhancer-binding proteins and the pathogenesis of retrovirus infection.

CCAAT/enhancer-binding proteins and the pathogenesis of retrovirus infection.

http://www.wikidata.org/entity/Q37425262

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HIV-1 transcriptional regulation in the central nervous system and implications for HIV cure research CCAAT enhancer binding protein and nuclear factor of activated T cells regulate HIV-1 LTR via a novel conserved downstream site in cells of the monocyte-macrophage lineage Impact of Tat Genetic Variation on HIV-1 Disease HTLV-1 bZIP factor supports proliferation of adult T cell leukemia cells through suppression of C/EBPα signaling Establishment of mesenchymal stem cell lines derived from the bone marrow of green fluorescent protein-transgenic mice exhibiting a diversity in intracellular transforming growth factor-β and bone morphogenetic protein signaling.Porcine epidemic diarrhea virus nucleoprotein contributes to HMGB1 transcription and release by interacting with C/EBP-β.CCAAT/enhancer binding protein β regulates the expression of tumor necrosis factor-α in the nucleus pulposus cells.Molecular Control of HIV and SIV Latency.Distinctive variation in the U3R region of the 5' Long Terminal Repeat from diverse HIV-1 strains.

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CCAAT/enhancer-binding proteins and the pathogenesis of retrovirus infection.

http://www.wikidata.org/entity/Q37425262

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

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bilimsel makale

@tr

scientific article published on April 2009

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

CCAAT/enhancer-binding proteins and the pathogenesis of retrovirus infection.

@en

CCAAT/enhancer-binding proteins and the pathogenesis of retrovirus infection.

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type

label

CCAAT/enhancer-binding proteins and the pathogenesis of retrovirus infection.

@en

CCAAT/enhancer-binding proteins and the pathogenesis of retrovirus infection.

@nl

prefLabel

CCAAT/enhancer-binding proteins and the pathogenesis of retrovirus infection.

@en

CCAAT/enhancer-binding proteins and the pathogenesis of retrovirus infection.

@nl

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Future Microbiology

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CCAAT/enhancer-binding proteins and the pathogenesis of retrovirus infection.

@en

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Brian Wigdahl

http://www.w3.org/2001/XMLSchema#string

Yujie Liu

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P2860

Glucocorticoid-stimulated preadipocyte differentiation is mediated through acetylation of C/EBPbeta by GCN5

Ig/EBP (C/EBP gamma) is a transdominant negative inhibitor of C/EBP family transcriptional activators

A nuclear factor for IL-6 expression (NF-IL6) is a member of a C/EBP family

A novel human CCAAT/enhancer binding protein gene, C/EBPepsilon, is expressed in cells of lymphoid and myeloid lineages and is localized on chromosome 14q11.2 close to the T-cell receptor alpha/delta locus

Binding of human immunodeficiency virus type 1 to CD4 and CXCR4 receptors differentially regulates expression of inflammatory genes and activates the MEK/ERK signaling pathway.

CCAAT/enhancer-binding proteins: structure, function and regulation

Absolute dependence on kappa B responsive elements for initiation and Tat-mediated amplification of HIV transcription in blood CD4 T lymphocytes

Interaction and functional collaboration of p300 and C/EBPbeta

CCAAT/enhancer binding protein epsilon is preferentially up-regulated during granulocytic differentiation and its functional versatility is determined by alternative use of promoters and differential splicing

Tat gets the "green" light on transcription initiation

P304

299-321

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scholarly article

P356

10.2217/FMB.09.4

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3

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4

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Michael Nonnemacher

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2009-04-01T00:00:00Z

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24239643

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19327116

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2710768

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