Complement protein C3 binding to Mycobacterium tuberculosis is initiated by the classical pathway in human bronchoalveolar lavage fluid.
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Genetic association and expression studies indicate a role of toll-like receptor 8 in pulmonary tuberculosisMycobacterium tuberculosis AtsG (Rv0296c), GlmU (Rv1018c) and SahH (Rv3248c) Proteins Function as the Human IL-8-Binding Effectors and Contribute to Pathogen Entry into Human NeutrophilsOral vaccination with heat inactivated Mycobacterium bovis activates the complement system to protect against tuberculosisMolecular composition of the alveolar lining fluid in the aging lung.Bacillus anthracis interacts with plasmin(ogen) to evade C3b-dependent innate immunity.Complement system in lung disease.A new role of the complement system: C3 provides protection in a mouse model of lung infection with intracellular Chlamydia psittaci.Comparative Genomics Reveal That Host-Innate Immune Responses Influence the Clinical Prevalence of Legionella pneumophila Serogroups.Collectin CL-LK Is a Novel Soluble Pattern Recognition Receptor for Mycobacterium tuberculosisAssociation of the FCN2 Gene Single Nucleotide Polymorphisms with Susceptibility to Pulmonary Tuberculosis.Lung Mucosa Lining Fluid Modification of Mycobacterium tuberculosis to Reprogram Human Neutrophil Killing MechanismsDecreased C3 Activation by the devR Gene-Disrupted Mycobacterium tuberculosis Strain in Comparison to the Wild-Type StrainReduced Mycobacterium tuberculosis association with monocytes from diabetes patients that have poor glucose controlAssociation of Human Antibodies to Arabinomannan With Enhanced Mycobacterial Opsonophagocytosis and Intracellular Growth Reduction.Macrophages in tuberculosis: friend or foe.Evasion of complement-mediated lysis and complement C3 deposition are regulated by Francisella tularensis lipopolysaccharide O antigen.Highly Multiplexed Proteomic Analysis of Quantiferon Supernatants To Identify Biomarkers of Latent Tuberculosis Infection.Mycobacterium Tuberculosis Infection and Inflammation: what is Beneficial for the Host and for the Bacterium?Progress in Oral Vaccination against Tuberculosis in Its Main Wildlife Reservoir in Iberia, the Eurasian Wild Boar.Proteomic profiling of eccrine sweat reveals its potential as a diagnostic biofluid for active tuberculosis.System for Efficacy and Cytotoxicity Screening of Inhibitors Targeting Intracellular Mycobacterium tuberculosis.C-type lectin receptors in tuberculosis: what we know.Distinct phases of blood gene expression pattern through tuberculosis treatment reflect modulation of the humoral immune response.Integrating Lung Physiology, Immunology, and Tuberculosis.The Goldilocks model of immune symbiosis with Mycobacteria and Candida colonizers.Activation of the classical complement pathway by Bacillus anthracis is the primary mechanism for spore phagocytosis and involves the spore surface protein BclA.Differences between Mycobacterium-Host Cell Relationships in Latent Tuberculous Infection of Mice Ex Vivo and Mycobacterial Infection of Mouse Cells In Vitro.Multiple components in serum contribute to hepatic transgene expression by lipoplex in mice.Pattern recognition receptors and cytokines in Mycobacterium tuberculosis infection--the double-edged sword?Correlates of Vaccine-Induced Protection against Mycobacterium tuberculosis Revealed in Comparative Analyses of Lymphocyte Populations.Combination of host susceptibility and Mycobacterium tuberculosis virulence define gene expression profile in the host.Effect of animal sera on Bacillus anthracis Sterne spore germination and vegetative cell growth.Mycobacteria exploit nitric oxide-induced transformation of macrophages into permissive giant cells.Genetic variants of complement genes ficolin-2, mannose-binding lectin and complement factor H are associated with leprosy in Han Chinese from Southwest China.Tuberculosis in the elderly: Why inflammation matters.Human Properdin Modulates Macrophage: Mycobacterium bovis BCG Interaction via Thrombospondin Repeats 4 and 5.25-Hydroxyvitamin D concentrations in dogs with naturally acquired blastomycosis
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P2860
Complement protein C3 binding to Mycobacterium tuberculosis is initiated by the classical pathway in human bronchoalveolar lavage fluid.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on May 2004
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Complement protein C3 binding ...... bronchoalveolar lavage fluid.
@en
Complement protein C3 binding ...... bronchoalveolar lavage fluid.
@nl
type
label
Complement protein C3 binding ...... bronchoalveolar lavage fluid.
@en
Complement protein C3 binding ...... bronchoalveolar lavage fluid.
@nl
prefLabel
Complement protein C3 binding ...... bronchoalveolar lavage fluid.
@en
Complement protein C3 binding ...... bronchoalveolar lavage fluid.
@nl
P2093
P2860
P1476
Complement protein C3 binding ...... bronchoalveolar lavage fluid.
@en
P2093
J Scott Ferguson
Jennifer L Martin
Jeremy J Weis
Larry S Schlesinger
P2860
P304
P356
10.1128/IAI.72.5.2564-2573.2004
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P577
2004-05-01T00:00:00Z