Mycobacteria inhibit nitric oxide synthase recruitment to phagosomes during macrophage infection. - Wikidata - wikimedia - TriplyDB

Mycobacteria inhibit nitric oxide synthase recruitment to phagosomes during macrophage infection.

Mycobacteria inhibit nitric oxide synthase recruitment to phagosomes during macrophage infection.

http://www.wikidata.org/entity/Q37426403

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Pathogenesis, immunology, and diagnosis of latent Mycobacterium tuberculosis infection Antimicrobial Mechanisms of Macrophages and the Immune Evasion Strategies of Staphylococcus aureus Mechanism of inducible nitric oxide synthase exclusion from mycobacterial phagosomes Contrasting Lifestyles Within the Host Cell Control of parasitophorous vacuole expansion by LYST/Beige restricts the intracellular growth of Leishmania amazonensis Deletion of the Mycobacterium tuberculosis pknH gene confers a higher bacillary load during the chronic phase of infection in BALB/c mice RahU: an inducible and functionally pleiotropic protein in Pseudomonas aeruginosa modulates innate immunity and inflammation in host cells The Delta fbpA mutant derived from Mycobacterium tuberculosis H37Rv has an enhanced susceptibility to intracellular antimicrobial oxidative mechanisms, undergoes limited phagosome maturation and activates macrophages and dendritic cells.The role of the activated macrophage in clearing Listeria monocytogenes infection Cholesterol oxidase is indispensable in the pathogenesis of Mycobacterium tuberculosis Analysis of Toll-like receptors, iNOS and cytokine profiles in patients with pulmonary tuberculosis during anti-tuberculosis treatment.Interactions between naïve and infected macrophages reduce Mycobacterium tuberculosis viability A mycobacterial phosphoribosyltransferase promotes bacillary survival by inhibiting oxidative stress and autophagy pathways in macrophages and zebrafish.A small molecule deubiquitinase inhibitor increases localization of inducible nitric oxide synthase to the macrophage phagosome and enhances bacterial killing Discovery of a novel Toxoplasma gondii conoid-associated protein important for parasite resistance to reactive nitrogen intermediates.Novel Markers to Delineate Murine M1 and M2 Macrophages Antibodies against invasive phenotype-specific antigens increase Mycobacterium avium subspecies paratuberculosis translocation across a polarized epithelial cell model and enhance killing by bovine macrophages.Genetically Modified Live Attenuated Leishmania donovani Parasites Induce Innate Immunity through Classical Activation of Macrophages That Direct the Th1 Response in Mice.EBP50 induces apoptosis in macrophages by upregulating nitric oxide production to eliminate intracellular Mycobacterium tuberculosis Nitrosothiols in the immune system: signaling and protection.Methionine sulfoxide reductases and virulence of bacterial pathogens.Tactics of Mycobacterium avium subsp. paratuberculosis for intracellular survival in mononuclear phagocytes.Mechanisms and consequences of persistence of intracellular pathogens: leishmaniasis as an example.A Mycobacterium avium subsp. paratuberculosis Predicted Serine Protease Is Associated with Acid Stress and Intraphagosomal Survival.Mannosylated lipoarabinomannans from Mycobacterium avium subsp. paratuberculosis alters the inflammatory response by bovine macrophages and suppresses killing of Mycobacterium avium subsp. avium organisms.Intrapulmonary delivery of XCL1-targeting small interfering RNA in mice chronically infected with Mycobacterium tuberculosis Th1-Th2 polarisation and autophagy in the control of intracellular mycobacteria by macrophages.Adjusting to a new home: Mycobacterium tuberculosis gene expression in response to an intracellular lifestyle.Disruption of immune regulation by microbial pathogens and resulting chronic inflammation.The pathology of Mycobacterium tuberculosis infection.Outcome of Mycobacterium tuberculosis and Toll-like receptor interaction: immune response or immune evasion?Soluble human TLR2 ectodomain binds diacylglycerol from microbial lipopeptides and glycolipids.Macrophage takeover and the host-bacilli interplay during tuberculosis.Immune response to antituberculosis drug-loaded gelatin and polyisobutyl-cyanoacrylate nanoparticles in macrophages.Continual renewal and replication of persistent Leishmania major parasites in concomitantly immune hosts.Inhibitory effect of citrinin on lipopolisaccharide-induced nitric oxide production by mouse macrophage cells.Dynamic life and death interactions between Mycobacterium smegmatis and J774 macrophages.Naturally produced opsonizing antibodies restrict the survival of Mycobacterium tuberculosis in human macrophages by augmenting phagosome maturation Yersinia pestis versus Yersinia pseudotuberculosis: effects on host macrophages.CD23 mediates antimycobacterial activity of human macrophages.

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Mycobacteria inhibit nitric oxide synthase recruitment to phagosomes during macrophage infection.

http://www.wikidata.org/entity/Q37426403

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on May 2004

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Mycobacteria inhibit nitric ox ...... s during macrophage infection.

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Mycobacteria inhibit nitric ox ...... s during macrophage infection.

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type

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Mycobacteria inhibit nitric ox ...... s during macrophage infection.

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Mycobacteria inhibit nitric ox ...... s during macrophage infection.

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prefLabel

Mycobacteria inhibit nitric ox ...... s during macrophage infection.

@en

Mycobacteria inhibit nitric ox ...... s during macrophage infection.

@nl

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IAI.72.5.2872-2878.2004

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Infection and Immunity

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Mycobacteria inhibit nitric ox ...... s during macrophage infection.

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Barbara H Miller

http://www.w3.org/2001/XMLSchema#string

Graham S Timmins

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Jens F Poschet

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Marcos Burgos

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Michael A Marletta

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Rutilio A Fratti

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Sharon S Master

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Vojo Deretic

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P2860

Mechanism of nitric oxide-dependent killing of Mycobacterium bovis BCG in human alveolar macrophages

Mechanisms of NADPH oxidase activation: translocation of p40phox, Rac1 and Rac2 from the cytosol to the membranes in human neutrophils lacking p47phox or p67phox

A novel cytosolic component, p40phox, of respiratory burst oxidase associates with p67phox and is absent in patients with chronic granulomatous disease who lack p67phox

Strains of Mycobacterium tuberculosis differ in susceptibility to reactive nitrogen intermediates in vitro

Susceptibility of a panel of virulent strains of Mycobacterium tuberculosis to reactive nitrogen intermediates

Response to reactive nitrogen intermediates in Mycobacterium tuberculosis: induction of the 16-kilodalton alpha-crystallin homolog by exposure to nitric oxide donors

The involvement of cell-to-cell signals in the development of a bacterial biofilm

Nitric oxide and macrophage function

Lack of acidification in Mycobacterium phagosomes produced by exclusion of the vesicular proton-ATPase

Killing of virulent Mycobacterium tuberculosis by reactive nitrogen intermediates produced by activated murine macrophages

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2872-2878

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10.1128/IAI.72.5.2872-2878.2004

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2004-05-01T00:00:00Z

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