Oxidant alterations in CD16 expression are cytoskeletal induced. - Wikidata - wikimedia - TriplyDB

Oxidant alterations in CD16 expression are cytoskeletal induced.

Oxidant alterations in CD16 expression are cytoskeletal induced.

http://www.wikidata.org/entity/Q37438694

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LPS-Induced Macrophage Activation and Plasma Membrane Fluidity Changes are Inhibited Under Oxidative Stress.A cytofluorometric study of membrane rafts in human monocyte subsets in atherosclerosis.

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P2860

Oxidant alterations in CD16 expression are cytoskeletal induced.

http://www.wikidata.org/entity/Q37438694

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on December 2009

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Oxidant alterations in CD16 expression are cytoskeletal induced.

@en

Oxidant alterations in CD16 expression are cytoskeletal induced.

@nl

type

label

Oxidant alterations in CD16 expression are cytoskeletal induced.

@en

Oxidant alterations in CD16 expression are cytoskeletal induced.

@nl

prefLabel

Oxidant alterations in CD16 expression are cytoskeletal induced.

@en

Oxidant alterations in CD16 expression are cytoskeletal induced.

@nl

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Oxidant alterations in CD16 expression are cytoskeletal induced.

@en

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Eileen Bulger

http://www.w3.org/2001/XMLSchema#string

Joseph Cuschieri

http://www.w3.org/2001/XMLSchema#string

Megan Knoll

http://www.w3.org/2001/XMLSchema#string

Ronald V Maier

http://www.w3.org/2001/XMLSchema#string

Saman Arbabi

http://www.w3.org/2001/XMLSchema#string

Sana Sakr

http://www.w3.org/2001/XMLSchema#string

P2860

Oxidative stress generated by hemorrhagic shock recruits Toll-like receptor 4 to the plasma membrane in macrophages

Pathophysiology of polytrauma.

The CD14+ CD16+ blood monocytes: their role in infection and inflammation.

Oxidative stress, lipid rafts, and macrophage reprogramming.

Severity of illness and organ failure assessment in adult intensive care units.

Two-hit hypothesis and multiple organ dysfunction syndrome.

Hydrogen peroxide induces the production of tumor necrosis factor-alpha in RAW 264.7 macrophage cells via activation of p38 and stress-activated protein kinase.

Oxidative-induced calcium mobilization is dependent on annexin VI release from lipid rafts.

Potentiation of hydrogen peroxide, nitric oxide, and cytokine production in RAW 264.7 macrophage cells exposed to human and commercial isolates of Bifidobacterium.

Possible role of increased oxidant stress in multiple organ failure after systemic inflammatory response syndrome.

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572-577

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scholarly article

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10.1097/SHK.0B013E3181A72530

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6

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32

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2009-12-01T00:00:00Z

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19333136

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