Mechanisms of STAT3 activation in the liver of FXR knockout mice. - Wikidata - wikimedia - TriplyDB

Mechanisms of STAT3 activation in the liver of FXR knockout mice.

Mechanisms of STAT3 activation in the liver of FXR knockout mice.

http://www.wikidata.org/entity/Q37440079

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Bile acid dysregulation, gut dysbiosis, and gastrointestinal cancer.FXR and liver carcinogenesis Activated FXR Inhibits Leptin Signaling and Counteracts Tumor-promoting Activities of Cancer-Associated Fibroblasts in Breast Malignancy.Mice with hepatocyte-specific FXR deficiency are resistant to spontaneous but susceptible to cholic acid-induced hepatocarcinogenesis.Bile acids promote diethylnitrosamine-induced hepatocellular carcinoma via increased inflammatory signaling.Role of farnesoid X receptor in cholestasis.Interactions Between Nuclear Receptor SHP and FOXA1 Maintain Oscillatory Homocysteine Homeostasis in Mice.Long-term Administration of Nuclear Bile Acid Receptor FXR Agonist Prevents Spontaneous Hepatocarcinogenesis in Abcb4-/- Mice.SOCS1 regulates hepatic regenerative response and provides prognostic makers for acute obstructive cholangitis.The FXR Agonist, Obeticholic Acid, Suppresses HCC Proliferation & Metastasis: Role of IL-6/STAT3 Signalling Pathway.Long noncoding RNA MEG3 induces cholestatic liver injury by interaction with PTBP1 to facilitate shp mRNA decay.Prolonged feeding with green tea polyphenols exacerbates cholesterol-induced fatty liver disease in mice.Prevention of spontaneous hepatocarcinogenesis in farnesoid X receptor-null mice by intestinal-specific farnesoid X receptor reactivation

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Mechanisms of STAT3 activation in the liver of FXR knockout mice.

http://www.wikidata.org/entity/Q37440079

description

article científic

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article scientifique

@fr

articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 03 October 2013

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Mechanisms of STAT3 activation in the liver of FXR knockout mice.

@en

Mechanisms of STAT3 activation in the liver of FXR knockout mice.

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type

label

Mechanisms of STAT3 activation in the liver of FXR knockout mice.

@en

Mechanisms of STAT3 activation in the liver of FXR knockout mice.

@nl

prefLabel

Mechanisms of STAT3 activation in the liver of FXR knockout mice.

@en

Mechanisms of STAT3 activation in the liver of FXR knockout mice.

@nl

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AJPGI.00155.2013

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American Journal of Physiology - Gastrointestinal and Liver Physiology

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Mechanisms of STAT3 activation in the liver of FXR knockout mice.

@en

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Grace L Guo

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Guodong Li

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James P Luyendyk

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Jessica A Williams

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Karen M Kassel

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Le Zhan

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Li Wang

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Ossama Tawfik

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Yan Zhu

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P2860

Identification of a nuclear receptor for bile acids

Bile acids: natural ligands for an orphan nuclear receptor

A family of cytokine-inducible inhibitors of signalling

Endogenous bile acids are ligands for the nuclear receptor FXR/BAR

Isolation of proteins that interact specifically with the retinoid X receptor: two novel orphan receptors

Bile acids regulate the ontogenic expression of ileal bile acid binding protein in the rat via the farnesoid X receptor

Farnesoid X receptor antagonizes nuclear factor kappaB in hepatic inflammatory response

STATs in cancer inflammation and immunity: a leading role for STAT3

Farnesoid X receptor protects liver cells from apoptosis induced by serum deprivation in vitro and fasting in vivo.

Upregulation of early growth response factor-1 by bile acids requires mitogen-activated protein kinase signaling.

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G829-37

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10.1152/AJPGI.00155.2013

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