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Impact of heparanase on renal fibrosisDiabetic nephropathy is resistant to oral L-arginine or L-citrulline supplementationDelayed Treatment with a Small Pigment Epithelium Derived Factor (PEDF) Peptide Prevents the Progression of Diabetic Renal InjuryFK506 reduces albuminuria through improving podocyte nephrin and podocin expression in diabetic rats.Nur77-deficiency in bone marrow-derived macrophages modulates inflammatory responses, extracellular matrix homeostasis, phagocytosis and toleranceCD163+ M2c-like macrophages predominate in renal biopsies from patients with lupus nephritis.Arginase inhibition: a new treatment for preventing progression of established diabetic nephropathyBeta 2-adrenergic receptor agonists are novel regulators of macrophage activation in diabetic renal and cardiovascular complications.Blockade of CCR2 reduces macrophage influx and development of chronic renal damage in murine renovascular hypertension.Early protective role of MST1 knockdown in response to experimental diabetic nephropathyModerate-intensity exercise alters markers of alternative activation in circulating monocytes in females: a putative role for PPARγ.Commentary: Indoleamine 2,3-Dioxygenase-Expressing Aortic Plasmacytoid Dendritic Cells Protect against Atherosclerosis by Induction of Regulatory T Cells.Inflammation in diabetic nephropathy: moving toward clinical biomarkers and targets for treatment.NLRP3 expression and urinary HSP72 in relation to biomarkers of inflammation and oxidative stress in diabetic nephropathy patients.Heparanase regulation of cancer, autophagy and inflammation: new mechanisms and targets for therapy.High glucose induced-macrophage activation through TGF-β-activated kinase 1 signaling pathway.Macrophage-derived tumor necrosis factor-α mediates diabetic renal injury.Infiltrating macrophages in diabetic nephropathy promote podocytes apoptosis via TNF-α-ROS-p38MAPK pathway.Murine glomerular transcriptome links endothelial cell-specific molecule-1 deficiency with susceptibility to diabetic nephropathy.A novel pan-Nox inhibitor, APX-115, protects kidney injury in streptozotocin-induced diabetic mice: possible role of peroxisomal and mitochondrial biogenesis.Physiological and molecular effects of interleukin-18 administration on the mouse kidney.Distinct roles of arginases 1 and 2 in diabetic nephropathy.Inhibition of macrophage migration inhibitory factor reduces diabetic nephropathy in type II diabetes mice.Podocyte-specific chemokine (C-C motif) receptor 2 overexpression mediates diabetic renal injury in mice.Macrophages in diabetic nephropathy in patients with type 2 diabetes.Macrophage Depletion Lowered Blood Pressure and Attenuated Hypertensive Renal Injury and Fibrosis.
P2860
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P2860
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on 30 October 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Macrophages directly mediate diabetic renal injury.
@en
Macrophages directly mediate diabetic renal injury.
@nl
type
label
Macrophages directly mediate diabetic renal injury.
@en
Macrophages directly mediate diabetic renal injury.
@nl
prefLabel
Macrophages directly mediate diabetic renal injury.
@en
Macrophages directly mediate diabetic renal injury.
@nl
P2093
P2860
P1476
Macrophages directly mediate diabetic renal injury.
@en
P2093
Alaa S Awad
Hanning You
Timothy K Cooper
W Brian Reeves
P2860
P304
P356
10.1152/AJPRENAL.00141.2013
P577
2013-10-30T00:00:00Z