Estrogen-induced interaction between KLF5 and estrogen receptor (ER) suppresses the function of ER in ER-positive breast cancer cells
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Mammalian Krüppel-like factors in health and diseasesThe double life of KLF5: Opposing roles in regulation of gene-expression, cellular function, and transformationThe induction of KLF5 transcription factor by progesterone contributes to progesterone-induced breast cancer cell proliferation and dedifferentiation.New cathepsin D inhibitor library utilizing hydroxyethyl isosteres with cyclic tertiary amines.Interruption of KLF5 acetylation converts its function from tumor suppressor to tumor promoter in prostate cancer cells.Klf5 deletion promotes Pten deletion-initiated luminal-type mouse prostate tumors through multiple oncogenic signaling pathwaysOestrogen causes degradation of KLF5 by inducing the E3 ubiquitin ligase EFP in ER-positive breast cancer cells.A functional interplay between ZNF217 and estrogen receptor alpha exists in luminal breast cancers.Transposable elements generate regulatory novelty in a tissue-specific fashion.
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P2860
Estrogen-induced interaction between KLF5 and estrogen receptor (ER) suppresses the function of ER in ER-positive breast cancer cells
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on January 2010
@en
vedecký článok
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vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
Estrogen-induced interaction b ...... R-positive breast cancer cells
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Estrogen-induced interaction between KLF5 and estrogen receptor
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type
label
Estrogen-induced interaction b ...... R-positive breast cancer cells
@en
Estrogen-induced interaction between KLF5 and estrogen receptor
@nl
prefLabel
Estrogen-induced interaction b ...... R-positive breast cancer cells
@en
Estrogen-induced interaction between KLF5 and estrogen receptor
@nl
P2860
P50
P356
P1476
Estrogen-induced interaction b ...... R-positive breast cancer cells
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P2093
P2860
P356
10.1002/IJC.24696
P577
2010-01-01T00:00:00Z