Efficacy of (S)-Lacosamide in preclinical models of cephalic pain.

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CRMP2 Phosphorylation Drives Glioblastoma Cell Proliferation.CRMP2-Neurofibromin Interface Drives NF1-related Pain.

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Efficacy of (S)-Lacosamide in preclinical models of cephalic pain.

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http://www.wikidata.org/entity/Q37451570

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on June 2016

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videnskabelig artikel

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vědecký článek

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Efficacy of (S)-Lacosamide in preclinical models of cephalic pain.

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Efficacy of

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label

Efficacy of (S)-Lacosamide in preclinical models of cephalic pain.

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Efficacy of

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Efficacy of (S)-Lacosamide in preclinical models of cephalic pain.

@en

Efficacy of

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P1476

Efficacy of (S)-Lacosamide in preclinical models of cephalic pain.

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David W Dodick

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Edwin Telemi

http://www.w3.org/2001/XMLSchema#string

Frank Porreca

http://www.w3.org/2001/XMLSchema#string

Jennifer Y Xie

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Ki Duk Park

http://www.w3.org/2001/XMLSchema#string

Nathan Eyde

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CRMP-2 peptide mediated decrease of high and low voltage-activated calcium channels, attenuation of nociceptor excitability, and anti-nociception in a model of AIDS therapy-induced painful peripheral neuropathy

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scholarly article

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10.1097/PR9.0000000000000565

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Rajesh Khanna

A Moutal

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2016-06-01T00:00:00Z

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27917413

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Efficacy of (S)-Lacosamide in preclinical models of cephalic pain.

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P2860

P2860

Efficacy of (S)-Lacosamide in preclinical models of cephalic pain.

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P1433

P1476

P2093

P2860

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P356

P433

P478

P50

P577

P698

P932

P356

P1433

P1476

P2093

P2860

P31

P356

P433

P478

P50

P577

P698

P932