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Arrhythmia pharmacogenomics: methodological considerations.

Arrhythmia pharmacogenomics: methodological considerations.

http://www.wikidata.org/entity/Q37458988

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Implications for educating the next generation of nurses on genetics and genomics in the 21st century A case for pharmacogenomics in management of cardiac arrhythmias.

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Arrhythmia pharmacogenomics: methodological considerations.

http://www.wikidata.org/entity/Q37458988

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

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bilimsel makale

@tr

scientific article published on January 2009

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Arrhythmia pharmacogenomics: methodological considerations.

@en

Arrhythmia pharmacogenomics: methodological considerations.

@nl

type

label

Arrhythmia pharmacogenomics: methodological considerations.

@en

Arrhythmia pharmacogenomics: methodological considerations.

@nl

prefLabel

Arrhythmia pharmacogenomics: methodological considerations.

@en

Arrhythmia pharmacogenomics: methodological considerations.

@nl

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138161209789649529

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Current Pharmaceutical Design

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Arrhythmia pharmacogenomics: methodological considerations

@en

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Dan M Roden

http://www.w3.org/2001/XMLSchema#string

Prince J Kannankeri

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P2860

A novel form of short QT syndrome (SQT3) is caused by a mutation in the KCNJ2 gene

Mutation in glycerol-3-phosphate dehydrogenase 1 like gene (GPD1-L) decreases cardiac Na+ current and causes inherited arrhythmias

CAPON modulates cardiac repolarization via neuronal nitric oxide synthase signaling in the heart

Sodium channel β1 subunit mutations associated with Brugada syndrome and cardiac conduction disease in humans

Mutation in nuclear pore component NUP155 leads to atrial fibrillation and early sudden cardiac death

KCNQ1 gain-of-function mutation in familial atrial fibrillation

Identification of a KCNE2 gain-of-function mutation in patients with familial atrial fibrillation.

Loss-of-function mutations in the cardiac calcium channel underlie a new clinical entity characterized by ST-segment elevation, short QT intervals, and sudden cardiac death

Kv1.5 channelopathy due to KCNA5 loss-of-function mutation causes human atrial fibrillation

Prevalence of long-QT syndrome gene variants in sudden infant death syndrome

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3734-3741

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scholarly article

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10.2174/138161209789649529

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32

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15

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2009-01-01T00:00:00Z

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38112968

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19925424

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pharmacogenomics

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2788624

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